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failed to show benefit from prophylactic peri-operative glucocorticoids during cardiac surgery in children ( 8 ), the use of glucocorticoids in pediatric cardiac surgery is controversial and questionable ( 4 , 8 , 9 , 10 , 11 , 12 ). Conflict
Department of Child and Adolescent Medicine, Section of Pediatric Cardiology, University Hospital Jena, Am Klinikum, Jena, Germany
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Department of Women and Child Health, Hospital for Children and Adolescents and Center for Pediatric Research (CPL), University of Leipzig, Liebigstrasse, Leipzig, Germany
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Department of Women and Child Health, Hospital for Children and Adolescents and Center for Pediatric Research (CPL), University of Leipzig, Liebigstrasse, Leipzig, Germany
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childhood development remains not well understood. Recent studies have investigated influences of growth hormone factors on cardiac markers in different adult cohorts and small pediatric patient groups. For example, positive correlations of IGF-BP1 and IGF
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majority showed no evidence of disease; 8 had recurrent and 9 persistent disease. In 66 subjects, all of them long-term adult survivors of pediatric DTC with a median follow-up of 17 years, the prevalence of cardiac dysfunction was studied in relation to
Division of Epidemiology and Biometry, Carl von Ossietzky University, Oldenburg, Germany
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Faculty of Health Sciences and Medicine, University of Lucerne, Lucerne, Switzerland
Department of Pediatrics, McMaster Children’s Hospital and McMaster University, Hamilton, Ontario, Canada
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-0419 ) 17 Mong S Pomeroy SL Cecchin F Juraszek A & Alexander ME . Cardiac risk after craniopharyngioma therapy . Pediatric Neurology 2008 38 256 – 260 . ( https://doi.org/10.1016/j.pediatrneurol.2007.11.007 ) 18 Roth CL . Hypothalamic
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, few studies have been dedicated to characterizing the electrical alterations caused by excess vitamin D. On the cardiovascular system could be observed changes in ventricular repolarization and cardiac rhythm conduction resultant of the excess of
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.04) – – CT Mutation in GATA6 No mutation in IPF1. Yes Cardiac malformation, hydronephrosis hydroureter, absent gallbladder Taha et al. ( 22 ) M 35 Yes (11 months) 1700 (2.92) – – US and CT No mutation in PTF1A and PDX1, KCNJ11
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considerable proportion of the women with induced puberty no longer had any estrogen therapy ( 14 ). The reasons for insufficient medical care for women with TS in adulthood may be manifold. Transfer from pediatric to adult medical care is a challenge for
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chromosomes, and depending on the level of mosaicism (the proportion of affected cells to healthy ones), it can vary in severity ( 2 ). Short stature and primary ovarian failure (OF) are the hallmarks of TS, while it can also manifest with cardiac and renal
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Department of Neuroscience, Rehabilitation, Ophthalmology, Genetics, Maternal and Child Health, University of Genova, Genova, Italy
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safety of rhGH therapy (Norditropin® (somatropin); Novo Nordisk A/S) in routine clinical practice ( 23 ). Adult and pediatric patients with a variety of conditions affecting growth were eligible for inclusion, including patients with NS. All visits were
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surgery ( 2 , 7 , 8 , 9 , 10 ). However, due to the absence of a standard definition of hypocalcemia, the incidence of postsurgical hypoparathyroidism varies 0–20.2% ( 3 , 8 , 9 , 10 , 11 , 12 ). There are other less-frequent etiologies which may