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Satoshi Higuchi, Hideki Ota, Takuya Ueda, Yuta Tezuka, Kei Omata, Yoshikiyo Ono, Ryo Morimoto, Masataka Kudo, Fumitoshi Satoh and Kei Takase

, myocardial T1-mapping provides myocardial longitudinal (spin-lattice) relaxation times reflecting myocardial tissue properties, such as edema, fibrosis or fat infiltration, without the use of contrast medium. This technique is a sensitive tool for detecting

Open access

Marianne Aa Grytaas, Kjersti Sellevåg, Hrafnkell B Thordarson, Eystein S Husebye, Kristian Løvås and Terje H Larsen

a comparison between affected and unaffected myocardium and may not be sensitive to early, potentially reversible diffuse myocardial fibrosis affecting the whole myocardium ( 28 ). Recently, T1 mapping techniques assessing T1 relaxation times have

Open access

Jia Liu, Min Liu, Zhe Chen, Yumei Jia and Guang Wang

://doi.org/10.1136/heartjnl-2012-303052 ) 14 Gao X Liu M Qu A Chen Z Jia Y Yang N Feng X Liu J Xu Y Yang X , Native magnetic resonance T1-mapping identifies diffuse myocardial injury in hypothyroidism . PLoS ONE 2016 11 e0151266 . ( https

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Tsuneo Ogawa and Adolfo J de Bold

production is stimulated by hypoxia, angiotensin II, cytokines, growth factors and reduced by nitric oxide and ANF (174, 175, 176, 177, 178, 179, 180) . ET1 plasma levels are increased in congestive heart failure, myocardial infarction, pulmonary

Open access

Rossella Cannarella, Teresa Mattina, Rosita A Condorelli, Laura M Mongioì, Giuseppe Pandini, Sandro La Vignera and Aldo E Calogero

consistency. Basal serum luteotropin hormone (LH), follicle-stimulating hormone (FSH) and testosterone (T) levels were 5.61 IU/mL (normal values (n.v.): 1.5–9.3 IU/mL), 5.37 IU/mL (n.v.: 1.4–18.1 IU/mL) and 1.87 ng/mL (n.v.: 3–9 ng/mL), respectively. When he

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Yiqiong Ma, Zhaowei Chen, Yu Tao, Jili Zhu, Hongxia Yang, Wei Liang and Guohua Ding

the mitochondrial outer membrane Na + /Ca2 + transporter Ncx3, thereby stabilizing mitochondrial calcium flux and alleviating cellular damage caused by hypoxia ( 34 ). Deleting AKAP1 increases mitochondrial ROS production and aggravates myocardial