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Aaron Lerner, Patricia Jeremias, and Torsten Matthias

autoimmune diseases + + + Celiac disease CD is an autoimmune inflammatory disorder of the small intestine, triggered by the ingestion of prolamins contained in wheat, barley or rye, in genetically susceptible individuals. Its incidence

Open access

Małgorzata Fuksiewicz, Maria Kowalska, Agnieszka Kolasińska-Ćwikła, Jarosław B Ćwikła, Łukasz Sawicki, Katarzyna Roszkowska-Purska, Joanna Drygiel, and Beata Kotowicz

digestive tract, including 59 with tumours located in the pancreas and 72 with lesions in the small intestine, caecum and appendix (midgut – the tumours originating from the central part of the archenteron) and in the colon (hindgut – tumours of the

Open access

Anna Malczewska, Magdalena Witkowska, Karolina Makulik, Agnes Bocian, Agata Walter, Joanna Pilch-Kowalczyk, Wojciech Zajęcki, Lisa Bodei, Kjell Oberg, and Beata Kos-Kudła

Introduction Neuroendocrine tumors (NETs) have increased in incidence with small intestine NETs (SINETs) being the most common and pancreatic NETs (PNETs), the third most common among gastroenteropancreatic (GEP) NETs per the SEER database

Open access

Chunliang Yang, Junyi Li, Fei Sun, Haifeng Zhou, Jia Yang, and Chao Yang

acting on intestine and kidney Intestine and kidney, responsible for glucose absorption and reabsorption, respectively, are subjected to the regulation of SGK1. Glucose transporter 1 (GLUT1) and sodium-glucose cotransporter 1 (SGLT1) are two important

Open access

Merete Gedde-Dahl, Espen Thiis-Evensen, Andreas Myklebust Tjølsen, Kjerstin Skrede Mordal, Morten Vatn, and Deidi S Bergestuen

characteristics. Total group ( n =34) Gender  Men 18 (52.9%)  Women 16 (47.1%) Age, years (median, range) 62 (18–76) Primary tumor location  Small intestine 33 (97.1%)  Appendix 1 (2

Open access

Wafaa M Rashed, Anas Saad, Muneer Al-Husseini, Ahmed Mahmoud Galal, Assem Mohamed Ismael, Ahmed M Al-Tayep, Ayman El Shafie, Mahmoud Ahmed Ali, and Ahmad Samir Alfaar

endocrine tissue (including the thymus and excluding thyroid) (O/E = 38.3, 95% CI = 10.4–98.1); the small intestine (O/E = 8.9, 95% CI = 1.1–32); the liver (O/E = 8.7, 95% CI = 1.1–31.6); the stomach (O/E = 5, 95% CI = 1–14.5); nodal NHL (O/E = 3.8, 95% CI

Open access

Irvin M Modlin, Harry Aslanian, Lisa Bodei, Ignat Drozdov, and Mark Kidd

1 included 91 GEP-NETs (gastric, n =3; duodenum, n =1; pancreas, n =41; small intestine, n =40; appendix, n =3; and colorectum, n =3), 18 with an unknown primary, and 16 non-GEP-NETs. Histopathologically, 51% were G1, 27% G2, and 12% G3

Open access

Anna Malczewska, Kjell Oberg, and Beata Kos-Kudla

. Cohorts The study cohort comprised 258 NENs, including gastroenteropancreatic (GEP) NENs ( n = 215): pancreatic, PNENs, n = 67; small intestine (midgut), SINENs, n = 40; rectal, RNENs, n = 45; gastric, GNENs, n = 44; appendiceal, ANENs, n = 10

Open access

Jens F Rehfeld

duodenum ( 15 ). And in the same year, Fleig described how blood from an isolated loop of the small intestine, into which acid was injected, increased bile-flow when transfused into another dog ( 16 ). Thus, already 1 year after Bayliss’ and Starling

Open access

Elham Barazeghi, Per Hellman, Gunnar Westin, and Peter Stålberg

) that were diagnosed with SI-NET and operated upon in Uppsala University Hospital. Twenty-three primary tumors, 21 mesenteric, 6 liver, and 1 extramesenteric lymph node metastases and 3 ‘normal’ small intestine tissue specimens were analyzed. Informed