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Shane M Regnier, Andrew G Kirkley, Daniel Ruiz, Wakanene Kamau, Qian Wu, Kurunthachalam Kannan and Robert M Sargis

) signaling ( 18 , 20 ). In recent studies, male C57BL/6 mice exposed to TF via a standard laboratory chow diet for 12 weeks exhibited increased adiposity, glucose intolerance and insulin resistance with concomitant impairments in adipose insulin sensitivity

Open access

Neil R Chappell, Beth Zhou, Amy K Schutt, William E Gibbons and Chellakkan S Blesson

/mL vs lean PCOS 55.08 ± 7.8 ng/mL for AMH). Lean PCOS mice were glucose intolerance but normal hemoglobin A1c levels Glucose values were consistently higher at 30 min (control 13.5 ± 0.5 mmol/L vs lean PCOS 15.5 ± 0.5 mmol/L, P  < 0.01), 60 min

Open access

Mohammed S Albreiki, Benita Middleton and Shelagh M Hampton

hormonal responses in healthy young participants. Significantly higher glucose and insulin in the BL session suggests glucose intolerance and insulin insensitivity. Elevated NEFAs level in the DL session prior to the meal could either be due to the

Open access

Chenghao Piao, Xiaojie Wang, Shiqiao Peng, Xinyu Guo, Hui Zhao, Li He, Yan Zeng, Fan Zhang, Kewen Zhu and Yiwei Wang

Introduction Insulin sensitivity during pregnancy is reduced with the advancement of gestation. Thus, the demand for insulin is elevated to maintain the common blood sugar ( 1 ). Gestational diabetes mellitus (GDM) is characterized by glucose

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Hélène Lasolle, Amandine Ferriere, Alexandre Vasiljevic, Sandrine Eimer, Marie-Laure Nunes and Antoine Tabarin

, sagittal T1 and T2 sequences, with and without gadolinium); measurement of hemoglobin A1C (HbA1c) and fasting blood glucose (FBG). Patients were considered diabetic if they were being treated for diabetes and/or when FBG was ≥126 mg/dL and/or HbA1c ≥6

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Aline Stangherlin Martins, Ann Kristine Jansen, Luiz Oswaldo Carneiro Rodrigues, Camila Maria Matos, Marcio Leandro Ribeiro Souza, Juliana Ferreira de Souza, Maria de Fátima Haueisen Sander Diniz, Sandhi Maria Barreto, Leonardo Mauricio Diniz, Nilton Alves de Rezende and Vincent Michael Riccardi

and/or venous disorders in general, including developmental vascular dysplasia, ASCVD, and aneurysm. However, the specific type of VD in individuals with NF1 and its correlation with fasting blood glucose (FBG) level has not been investigated in

Open access

Lilit Egshatyan, Daria Kashtanova, Anna Popenko, Olga Tkacheva, Alexander Tyakht, Dmitry Alexeev, Natalia Karamnova, Elena Kostryukova, Vladislav Babenko, Maria Vakhitova and Sergey Boytsov

deviation degree Patients without glucose intolerance, who had passed the preventive outpatient examination. Patients with prediabetes (preD) (impaired fasting glucose or impaired glucose tolerance or the level of HbA1c from 5.7 to 6.5%. Patients with newly

Open access

A V Dreval, I V Trigolosova, I V Misnikova, Y A Kovalyova, R S Tishenina, I A Barsukov, A V Vinogradova and B H R Wolffenbuttel

Endocrinology 2000 52 549 – 555 . ( doi:10.1046/j.1365-2265.2000.00986.x ). 5 Kreze A Kreze-Spirova E Mikulecky M . Risk factors for glucose intolerance in active acromegaly . Brazilian Journal of Medical and Biological Research 2001 34 1429

Open access

L Ahlkvist, K Brown and B Ahrén

Introduction In fully compensated insulin resistance, there is a sufficient upregulation of insulin secretion whereas in glucose intolerance and type 2 diabetes this upregulation is inadequate (1) . Several mechanisms have been suggested to

Open access

Chao Xu, Xiang-Fei Li, Hong-Yan Tian, Hua-Juan Shi, Ding-Dong Zhang, Kenneth Prudence Abasubong and Wen-Bin Liu

between hepatic glucose uptake and production ( 1 , 2 , 4 , 5 , 6 ). However, the physiological basis for such apparent glucose intolerance in fish is still poorly understood. Hence, investigating the underlying mechanisms is of great significance, as