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T L C Wolters Division of Endocrinology, Department of Internal Medicine, Radboud University Medical Center, Nijmegen, The Netherlands

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C D C C van der Heijden Division of Experimental Internal Medicine, Department of Internal Medicine, Radboud University Medical Center, Nijmegen, The Netherlands
Division of Vascular Medicine, Department of Internal Medicine, Radboud University Medical Center, Nijmegen, The Netherlands

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N van Leeuwen Department of Physiology, Radboud Institute for Health Sciences, Radboud University Medical Center, Nijmegen, The Netherlands

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B T P Hijmans-Kersten Department of Physiology, Radboud Institute for Health Sciences, Radboud University Medical Center, Nijmegen, The Netherlands

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M G Netea Division of Experimental Internal Medicine, Department of Internal Medicine, Radboud University Medical Center, Nijmegen, The Netherlands

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J W A Smit Division of Endocrinology, Department of Internal Medicine, Radboud University Medical Center, Nijmegen, The Netherlands

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D H J Thijssen Department of Physiology, Radboud Institute for Health Sciences, Radboud University Medical Center, Nijmegen, The Netherlands
Research Institute for Sport and Exercise Sciences, Liverpool John Moores University, Liverpool, UK

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A R M M Hermus Division of Endocrinology, Department of Internal Medicine, Radboud University Medical Center, Nijmegen, The Netherlands

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N P Riksen Division of Vascular Medicine, Department of Internal Medicine, Radboud University Medical Center, Nijmegen, The Netherlands

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R T Netea-Maier Division of Endocrinology, Department of Internal Medicine, Radboud University Medical Center, Nijmegen, The Netherlands

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persistent endothelial dysfunction in controlled patients. These findings suggest that chronic inflammation could contribute to the high prevalence of CVD risk factors in acromegaly both during active disease as well as in adequately treated patients

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-19-0430
Volume/Issue:
Volume 8: Issue 12
Open access
Aasem Saif Internal Medicine Department, Cairo University, Cairo, Egypt

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Shrook Mousa Internal Medicine Department, Cairo University, Cairo, Egypt

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Maha Assem Internal Medicine Department, Cairo University, Cairo, Egypt

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Nashwa Tharwat National Nutrition Institute, Cairo, Egypt

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Alaa Abdelhamid Internal Medicine Department, Cairo University, Cairo, Egypt
Vascular Laboratory, Cairo University, Cairo, Egypt

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such as dyslipidemia, hypercoagulable state, increased arterial stiffness, obesity and endothelial dysfunction ( 1 , 2 , 3 , 4 ). Current controversy centers on whether subclinical hypothyroidism (SCH) is associated with atherosclerosis. Some studies

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0194
Volume/Issue:
Volume 7: Issue 10
Open access
Ningning Gong Department of Endocrinology, Gansu Provincial Hospital, Lanzhou, China
Department of Clinical Medicine, Gansu University of Chinese Medicine, Lanzhou, Gansu, China

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Cuixia Gao Department of Ultrasonic Diagnosis, Gansu Provincial Hospital, Lanzhou, China

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Xuedi Chen Department of Endocrinology, Gansu Provincial Hospital, Lanzhou, China

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Yu Wang Department of Endocrinology, Gansu Provincial Hospital, Lanzhou, China

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Limin Tian Department of Endocrinology, Gansu Provincial Hospital, Lanzhou, China

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atherosclerosis (AS) was reported in a large population-based study ( 2 , 3 ). Multiple lines of evidence have shown that adipokines ( 4 ), endothelial dysfunction ( 5 ) and dyslipidemia ( 6 ) play a central role in the development of AS, but the relationship

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0007
Volume/Issue:
Volume 7: Issue 2
Open access
Madalena von Hafe Department of Surgery and Physiology, Faculty of Medicine, University of Porto, Porto, Portugal

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João Sergio Neves Department of Surgery and Physiology, Faculty of Medicine, University of Porto, Porto, Portugal
Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar São João, Porto, Portugal

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Catarina Vale Department of Surgery and Physiology, Faculty of Medicine, University of Porto, Porto, Portugal

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Marta Borges-Canha Department of Surgery and Physiology, Faculty of Medicine, University of Porto, Porto, Portugal
Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar São João, Porto, Portugal

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Adelino Leite-Moreira Department of Surgery and Physiology, Faculty of Medicine, University of Porto, Porto, Portugal

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reserves – perhaps leading to cardiac pump dysfunction ( 12 ). Nevertheless, this phenotype seems to be reversible with the appropriate therapy ( 13 ). T3 is able to modify the activity of sodium, potassium and calcium channels, altering a variety of

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-19-0096
Volume/Issue:
Volume 8: Issue 5
Open access
Zeeshan Javed Department of Academic Diabetes, Endocrinology and Metabolism, Hull York Medical School, University of Hull, Hull, UK
Department of Endocrinology and Diabetes, Pakistan Kidney and Liver Institute and Research Centre, Knowledge City, Lahore, Pakistan

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Maria Papageorgiou Department of Academic Diabetes, Endocrinology and Metabolism, Hull York Medical School, University of Hull, Hull, UK
Division of Bone Diseases, Geneva University Hospitals and Faculty of Medicine, University of Geneva, Geneva, Switzerland

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Leigh A Madden School of Life Sciences, University of Hull, Hull, UK

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Alan S Rigby Hull York Medical School, University of Hull, Hull, UK

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Eric S Kilpatrick Department of Pathology, Sidra Medical and Research Center, Doha, Qatar

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Stephen L Atkin Royal College of Surgeons in Ireland, Al Sayh, Bahrain

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Thozhukat Sathyapalan Department of Academic Diabetes, Endocrinology and Metabolism, Hull York Medical School, University of Hull, Hull, UK

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developing subclinical atherosclerosis even at a young age ( 3 , 4 ). Endothelial dysfunction is an early indicator of the atherosclerotic process, and several studies have demonstrated abnormal endothelial function assessed by flow-mediated dilation and

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-20-0173
Volume/Issue:
Volume 9: Issue 6
Open access
Daisuke Watanabe Department of Endocrinology and Hypertension, Tokyo Women’s Medical University, Shinjuku-ku, Tokyo, Japan

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Satoshi Morimoto Department of Endocrinology and Hypertension, Tokyo Women’s Medical University, Shinjuku-ku, Tokyo, Japan

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Noriko Morishima Department of Endocrinology and Hypertension, Tokyo Women’s Medical University, Shinjuku-ku, Tokyo, Japan

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Atsuhiro Ichihara Department of Endocrinology and Hypertension, Tokyo Women’s Medical University, Shinjuku-ku, Tokyo, Japan

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than among those with essential hypertension (EH) ( 3 ). In addition, severe arterial stiffness is associated with the absence of complete clinical success in lateralized PA after adrenalectomy (ADX) ( 4 ). Endothelial dysfunction is recognized as a

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-21-0057
Volume/Issue:
Volume 10: Issue 6
Open access
Tao Gao Department of General Practice, The Second Affiliated Hospital of Chongqing Medical University, Chongqing Medical University, Chongqing, China

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Rui Liu Department of Oncology. The Second Affiliated Hospital of Chongqing Medical University, Chongqing Medical University, Chongqing, China

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Chunli Li Institute of Life Sciences, Chongqing Medical University, Chongqing, China

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Xinglin Chu Department of General Practice, The Second Affiliated Hospital of Chongqing Medical University, Chongqing Medical University, Chongqing, China

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Qiao Guo Department of General Practice, The Second Affiliated Hospital of Chongqing Medical University, Chongqing Medical University, Chongqing, China

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Dazhi Ke Department of General Practice, The Second Affiliated Hospital of Chongqing Medical University, Chongqing Medical University, Chongqing, China

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). Fetuin-B plays an important role in regulating lipid metabolism and can result in lipid abnormalities that increase the risk of hypertension, cardiovascular disease, and endothelial dysfunction. Fetuin-B can cause macrophages to accumulate lipids in a

Article Type:
Research Article
Collections:
Cardiovascular
DOI:
https://doi.org/10.1530/EC-23-0172
Volume/Issue:
Volume 12: Issue 12
Open access
Wang Chengji College of Physical Education, Chaohu University, Anhui Province, China

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Fan Xianjin College of Physical Education, Chaohu University, Anhui Province, China

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release into the blood is increased and is now recognized as a marker of endothelial cell damage and dysfunction, is the most valuable regulatory factors produced by endothelial cells. It has been shown that diabetic patients are hypercoagulable and their

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0060
Volume/Issue:
Volume 7: Issue 4
Open access
Panisa Hantrakun Department of Obstetrics and Gynecology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Rattanaporn Sekararithi Department of Obstetrics and Gynecology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Thidarat Jaiwongkam Cardiac Electrophysiology Research and Training Center (CERT), Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Sirinart Kumfu Cardiac Electrophysiology Research and Training Center (CERT), Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Chatree Chai-adisaksopha Division of Hematology, Department of Internal Medicine, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Nipon Chattipakorn Cardiac Electrophysiology Research and Training Center (CERT), Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Theera Tongsong Department of Obstetrics and Gynecology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Phudit Jatavan Department of Obstetrics and Gynecology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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placental lactogen (HPL), the placental hormone of powerful insulin resistance. Theoretically, GDM is a temporary metabolic derangement of insulin resistance, not involving endothelial cell damages and platelet dysfunction. It usually completely disappears

Article Type:
Research Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-22-0110
Volume/Issue:
Volume 11: Issue 4
Open access
Antonia Ertelt Equine Clinic, Free University of Berlin, Berlin, Germany

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Ann-Kristin Barton Equine Clinic, Free University of Berlin, Berlin, Germany

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Robert R Schmitz Equine Clinic, Free University of Berlin, Berlin, Germany

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Heidrun Gehlen Equine Clinic, Free University of Berlin, Berlin, Germany

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, 49, 50, 51) . All these factors contribute to a generalized proinflammatory state of the organism. Symptoms of metabolic syndrome are used to identify humans with an increased risk of cardiovascular disease (52) . Owing to endothelial dysfunction

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-14-0038
Volume/Issue:
Volume 3: Issue 3
Open access