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Anastasia P Athanasoulia-Kaspar, Kathrin H Popp and Gunter Karl Stalla

not required for this study as clinical study in human subjects was not conducted. The psychiatrist’s perspective: neuropsychiatric aspects of the dopaminergic treatment Apart from the common side effects of the dopaminergic treatment

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Chanyang Kim and Seungjoon Park

dopaminergic neurodegeneration in the substantia nigra pars compacta (SNpc) and the loss of projecting nerve fibers in the striatum ( 2 ), which results in dysfunction of extrapyramidal motor system. Although the precise molecular mechanisms underlying

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Sarah Byberg, Jesper Futtrup, Mikkel Andreassen and Jesper Krogh

treatment for most patients with prolactinoma. A link between high prolactin levels and cardiovascular mortality would have important clinical implications. According to current guidelines ( 22 ), asymptomatic patients with hyperprolactinemia are not

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L Bahler, H J Verberne, E Brakema, R Tepaske, J Booij, J B Hoekstra and F Holleman

Introduction In 30–50% of the patients with type 2 diabetes mellitus, current pharmaceutical options to reach an adequate glucose control are insufficient ( 1 ). Therefore, the search for new and better treatment options is still ongoing

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Irasema Mendieta, Rosa Elvira Nuñez-Anita, Gilberto Pérez-Sánchez, Lenin Pavón, Alfredo Rodríguez-Cruz, Guadalupe García-Alcocer and Laura Cristina Berumen

-methylxanthine (IBMX, 0.5 mM), forskolin (FSK, 0.5 mM) or IBMX + FSK (I + F) in fresh medium. Morphologic changes and proliferation rates were evaluated via light microscopy. The conservation of morphologic changes at 24 or 48 h after treatment withdrawal (WD

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Gavin P Vinson and Caroline H Brennan

adrenocortical hormones were even characterized that morphine toxicity was linked to the adrenal gland. Thus, Lewis (1) and Mackay & Mackay (2) showed that adrenalectomy increased morphine sensitivity in female rats, and chronic treatment with morphine in

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Stavroula Stavrou, Michael Gratz, Eileen Tremmel, Christina Kuhn, Simone Hofmann, Helene Heidegger, Mina Peryanova, Kerstin Hermelink, Stefan Hutter, Bettina Toth, Doris Mayr, Sven Mahner, Udo Jeschke and Aurelia Vattai

placenta, brain, spinal cord, stomach, pancreatic β-cells and immune cells like macrophages, leukocytes and dendritic cells ( 15 , 16 , 17 ). Different compounds, including dopaminergic, adrenergic and serotonergic classes and a wide spectrum of agonists

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Carolina Inda, Natalia G Armando, Paula A dos Santos Claro and Susana Silberstein

). In MN9D dopaminergic cells, UCN1 activates CRHR1 and modulates T-type calcium channels in a PKC-dependent manner ( 29 ), whereas in HEK293 cells, both UCNs and CRH acting via CRHR1 selectively inhibit CaV3.2 T-type calcium currents ( 30 ). As

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J Brossaud, V Pallet and J-B Corcuff

also show modifications of hypothalamo–pituitary–peripheral gland axes upon retinoid deprivation or treatment ( 31 , 32 , 33 , 34 , 35 , 36 ). Vitamin A and the hypothalamo–pituitary–thyroid axis RA does not seem to be involved in thyroid

Open access

Jean-Benoît Corcuff, Laurence Chardon, Ines El Hajji Ridah and Julie Brossaud

plastic containers and complete collection of urine during a 24-h period. With no treatment, 5HIAA tends to degrade during and after a 24-h urine collection. To avoid an underestimation of its concentration preservatives have to be added to urine. Ideally