relationship between cognitive impairment due to SCH and the apoptosis in cerebral cortex remains unclear. The p75 neurotrophin receptor (p75 NTR ) is an important member of the death receptor family. Although the role of the p75 NTR -mediated signal
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Fan Zhang, Jian Chen, Xinyue Lin, Shiqiao Peng, Xiaohui Yu, Zhongyan Shan, and Weiping Teng
Thiago U Pantaleão, Andrea C F Ferreira, Maria C S Santos, Álvaro S P Figueiredo, Ruy A N Louzada, Doris Rosenthal, Denise P Carvalho, and Vânia M Corrêa da Costa
–1992 ( 14 ). Cerebellum, hypothalamus, cerebral cortex, hippocampus, pituitary, liver and kidney were excised after treatment, immediately frozen in liquid nitrogen, and stored at −80°C for mRNA extraction and deiodinase and H 2 O 2 generating activities
Patricia Iozzo and Maria Angela Guzzardi
( 24 , 25 ). Imaging of the glucose analog 18 F-labeled fluorodeoxyglucose ( 18 FDG) by positron emission tomography (PET) is the gold standard for the assessment of cerebral glucose metabolism in vivo . In this manuscript, we review evidence on
Jens F Rehfeld
system Cerebral cortex 400 Hippocampus 350 Hypothalamus 200 Cerebellum 2 Spinal cord 40 Peripheral nervous system Vagal nerve 25 Sciatic nerve 15 Nerveplexes in colonic wall
Fiona Broughton Pipkin, Hiten D Mistry, Chandrima Roy, Bernhard Dick, Jason Waugh, Rebecca Chikhi, Lesia O Kurlak, and Markus G Mohaupt
Introduction The fetal adrenal gland and hypothalamic–pituitary axis (HPA) play important roles during pregnancy. Between weeks 32 and 34 of gestation, there is a rapid maturation of the fetal adrenal cortex, allowing development of a variety of
Aldo Bonaventura, Fabrizio Montecucco, and Franco Dallegri
augmented blood flow to the frontal cortex during acute hypoglycemia in order to supply the most vulnerable part of the brain persists during normoglycemia, too, leading to autoregulation disruption (60, 61) . As a result, the risk of cerebral ischemia
Huifei Sophia Zheng, Jeffrey G Daniel, Julia M Salamat, Laci Mackay, Chad D Foradori, Robert J Kemppainen, Satyanarayana R Pondugula, Ya-Xiong Tao, and Chen-Che Jeff Huang
widely used for therapeutic and diagnostic purposes in clinical settings due to its potent GR activity and predominant effects. Treatment with Dex suppresses the HPA axis activity leading to decreased steroidogenesis in the adrenal cortex, eventually
Stine Linding Andersen and Stig Andersen
hyperthyroidism on the stability of thalamocortical synapses in the visual cortex of adult offspring . Cerebral Cortex 2017 27 3015 – 3027 . ( https://doi.org/10.1093/cercor/bhw096 ) 27 Laureano-Melo R Souza JS da Conceição RR Albuquerque JML
Janko Sattler, Jinwen Tu, Shihani Stoner, Jingbao Li, Frank Buttgereit, Markus J Seibel, Hong Zhou, and Mark S Cooper
pituitaries). The level of expression was unchanged in the hippocampus, hypothalamus, cerebral cortex or cerebellum. However, the level of expression within the pituitary was significantly increased by 29% ( P = 0.013) in the arthritis group ( Fig. 4A
Annelies van’t Westeinde, Leif Karlsson, Valeria Messina, Lena Wallensteen, Manuela Brösamle, Giorgio Dal Maso, Alessandro Lazzerini, Jette Kristensen, Diana Kwast, Lea Tschaidse, Matthias K Auer, Hanna F Nowotny, Luca Persani, Nicole Reisch, and Svetlana Lajic
negative feedback on the hypothalamus–pituitary–adrenal (HPA) axis, resulting in the over-production of adrenocorticotropic hormone (ACTH). The excess ACTH is shunted towards the androgen production pathways in the adrenal cortex and thus leads to an
