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Katica Bajuk Studen and Marija Pfeifer

aim of this article is to explore and review current evidence that associates PCOS with cardiometabolic abnormalities and possibly an increased risk of cardiovascular disease. Insulin resistance, metabolic syndrome, obesity and sleep disturbances

Open access

Angelo Maria Patti, Kalliopi Pafili, Nikolaos Papanas and Manfredi Rizzo

( 19 , 20 , 21 ), and family history of CV risk is closely related to future CVD ( 22 , 23 ). Is there a synergistic effect of GDM and HDP on postpartum cardiometabolic risk? It has been reported that there is a significant link between

Open access

Ling-Jun Li, Izzuddin M Aris, Lin Lin Su, Yap Seng Chong, Tien Yin Wong, Kok Hian Tan and Jie Jin Wang

, HDP or co-existence of both pregnancy complications, on the risk of postpartum cardio-metabolic diseases, including metabolic syndrome (MetS), abnormal glucose metabolism and hypertension (HTN). Methods Study population and design This was

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Line K Johnson, Kirsten B Holven, Njord Nordstrand, Jan R Mellembakken, Tom Tanbo and Jøran Hjelmesæth

a lower prevalence of cardiometabolic risk factors including MS, cardiovascular inflammation and obesity (15) . Additionally, fiber intake improves gastrointestinal function and may prevent development of colorectal cancer (16) . Fiber intake from

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Nafiye Helvaci, Erdem Karabulut, Ahmet Ugur Demir and Bulent Okan Yildiz

is also associated with a substantial increase in risk for long-term cardiometabolic disorders including obesity, impaired glucose tolerance, type 2 diabetes, dyslipidemia, hypertension, coronary and other vascular disorders ( 4 ). There is also a

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Dorte Glintborg, Katrine Hass Rubin, Mads Nybo, Bo Abrahamsen and Marianne Andersen

requested studies regarding long-term morbidity in women with PCOS and subclinical hypothyroidism ( 14 ). It remains to be determined whether women with high-normal TSH levels represent a high-risk cardio-metabolic phenotype of women with PCOS. Our

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Shenglong Le, Leiting Xu, Moritz Schumann, Na Wu, Timo Törmäkangas, Markku Alén, Sulin Cheng and Petri Wiklund

might be an important regulator of puberty and a biomarker for cardiometabolic risk ( 3 ). However, confounding or reverse causation, may explain part of the association. Indeed, there may be a bidirectional relationship between SHBG, insulin resistance

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Andrea V Haas, Paul N Hopkins, Nancy J Brown, Luminita H Pojoga, Jonathan S Williams, Gail K Adler and Gordon H Williams

, individuals with incidentally discovered adrenal masses and biochemical work-up consistent with subclinical Cushing’s syndrome also have increased cardiometabolic risk ( 2 , 3 , 4 , 5 , 6 ). Even among individuals with biochemical work-up consistent with

Open access

Eliana Piantanida, Daniela Gallo, Giovanni Veronesi, Eugenia Dozio, Eugenia Trotti, Adriana Lai, Silvia Ippolito, Jessica Sabatino, Maria Laura Tanda, Antonio Toniolo, Marco Ferrario and Luigi Bartalena

cardiometabolic disease ( 45 , 46 ). A multivariate analysis involving analysis of demographic and anthropometric data and personal habits could better clarify this point. The Australian Diabetes, Obesity and Lifestyle Study reported that, among risk factors

Open access

Sanna Mustaniemi, Marja Vääräsmäki, Johan G Eriksson, Mika Gissler, Hannele Laivuori, Hilkka Ijäs, Aini Bloigu, Eero Kajantie and Laure Morin-Papunen

observations that low birth weight and preterm birth increase an individual’s risk of GDM ( 41 , 42 ). Preterm birth is also a well-established risk factor for T2D and related cardiometabolic risk factors ( 43 , 44 , 45 ). Moreover, the catch-up growth of