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Department of Cardiology, Yanbian University Hospital, Yanji, China
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reperfusion in diabetic mice ( 50 , 51 , 52 ). However, the effect of liraglutide on cardiac endocrine and its mechanism are still unclear. Therefore, we studied the effects of liraglutide on cardiac endocrinology through in vivo and ex vivo experiments
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High Field MR Centre, Department of Biomedical Imaging and Image-guided Therapy, Medical University of Vienna, Vienna, Austria
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insulin, glucose and free fatty acids (FAs) results in increased lipid accumulation in the myocardium and cardiac glucolipotoxicity, which ends up in LV heart failure. Thyroid hormones Thyroid hormones exert effects virtually on every organ
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the impact of overt and subclinical hyperthyroidism on the cardiovascular system. We will thereafter present the latest insights on the cardiac effects of thyroid suppression therapy for the treatment of thyroid cancer. Finally, we will show new
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Department of Internal Medicine, Section of Metabolic Vascular Medicine, Division of Diabetes and Nutritional Sciences, Cardiovascular Endocrinology Laboratory, Faculty of Medicine, Hospital de Urgencias, National University of Córdoba, X5000 Córdoba, Argentina
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The cNP act as a basic functional link between cardiovascular system homeostasis, inflammation and certain metabolic functions ( Fig. 1 ). Figure 1 Main biological targets and effects of the cardiac natriuretic peptides ANP and BNP. Increased secretion
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Department of Medicine, University of Maryland School of Medicine, Baltimore, Maryland, USA
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plasma levels of EO, aldosterone and corticosterone extends beyond their effects on BP; these steroids and their receptors have been implicated in cardiac growth and fibrosis, podocyte damage, memory and mood ( 86 , 87 , 88 , 89 , 90 , 91 ). Thus
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, short-term or long-term mortality, cardiac surgery type, different intervention time, diabetic status of participants and compared to different intensity of liberal control. Finally, we pooled the effects of more health-related complications, which can
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–IGF axis has important metabolic effects on a variety of target tissues ( Fig. 1 ). Historically, treatment with recombinant human GH (rhGH) was discontinued at final height as defined by a growth velocity less than 2cm/year ( 1 ). However, extensive
Comprehensive Heart Failure Center, University & University Hospital Würzburg, Würzburg, Germany
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Department of Psychiatry, Psychosomatics and Psychotherapy, University Hospital, University of Würzburg, Würzburg, Germany
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Division of Cardiology, Department of Internal Medicine I, University Hospital, University of Würzburg, Würzburg, Germany
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Department of General, Visceral, Transplant, Vascular, and Pediatric Surgery, University Hospital, University of Würzburg, Würzburg, Germany
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Division of Cardiology, Department of Internal Medicine I, University Hospital, University of Würzburg, Würzburg, Germany
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Division of Cardiology, Department of Internal Medicine I, University Hospital, University of Würzburg, Würzburg, Germany
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Comprehensive Heart Failure Center, University & University Hospital Würzburg, Würzburg, Germany
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Heart Association (NYHA) classification, diastolic function, reductions in left ventricular (LV) mass index, and left atrial size ( 20 , 21 ). The beneficial effects of weight loss may further include improvements in metabolic and neurohormonal
Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, San Francisco, California, USA
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Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, San Francisco, California, USA
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Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, San Francisco, California, USA
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proliferative responses between mice and zebrafish imply that the effects of vitamin D signaling on cardiac regeneration are highly species and context-dependent ( 36 , 40 ). Han et al. demonstrated that supraphysiological vitamin D expression in neonatal
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mechanism of action, the combination of all the effects triggered by TH may activate several cellular pathways and determine different manifestations associated with cardiac dysfunction, for example, in the development of cardiac hypertrophy which precedes