Search for other papers by Jia Liu in
Google Scholar
PubMed
Search for other papers by Min Liu in
Google Scholar
PubMed
Search for other papers by Zhe Chen in
Google Scholar
PubMed
Search for other papers by Yumei Jia in
Google Scholar
PubMed
Search for other papers by Guang Wang in
Google Scholar
PubMed
Introduction Autoimmune thyroiditis (AIT) is the most common autoimmune thyroid disease ( 1 , 2 ). Because of varying degree of thyroid destruction, AIT patients manifest with different thyroid function including euthyroidism, subclinical or
AESKU.KIPP Institute, Wendelsheim, Germany
Search for other papers by Aaron Lerner in
Google Scholar
PubMed
Search for other papers by Patricia Jeremias in
Google Scholar
PubMed
Search for other papers by Torsten Matthias in
Google Scholar
PubMed
Introduction Hashimoto's thyroiditis Hashimoto’s thyroiditis (HT) is one of the most common autoimmune endocrine diseases, characterized by an autoimmune-mediated destruction of the thyroid gland. Initially considered a rarity, HT has now
Search for other papers by A Rouland in
Google Scholar
PubMed
INSERM Unit, LNC-UMR 1231, University of Burgundy, Dijon, France
Search for other papers by J-C Chauvet-Gelinier in
Google Scholar
PubMed
INSERM Unit, LNC-UMR 1231, University of Burgundy, Dijon, France
Search for other papers by A-L Sberna in
Google Scholar
PubMed
Search for other papers by E Crevisy in
Google Scholar
PubMed
Search for other papers by P Buffier in
Google Scholar
PubMed
Search for other papers by T Mouillot in
Google Scholar
PubMed
INSERM Unit, LNC-UMR 1231, University of Burgundy, Dijon, France
Search for other papers by J-M Petit in
Google Scholar
PubMed
INSERM Unit, LNC-UMR 1231, University of Burgundy, Dijon, France
Search for other papers by B Vergès in
Google Scholar
PubMed
assessment and compare the Type A profiles of patients with T1D and patients presenting with other auto-immune disease (Graves’ disease or Hashimoto’s thyroiditis), in order to validate the hypothesis that increased Type A personality traits is related to the
Department of Nuclear Medicine, The Affiliated Hospital of Jiangsu University, Zhenjiang, China
Search for other papers by Fei Wu in
Google Scholar
PubMed
Search for other papers by Chaoming Mao in
Google Scholar
PubMed
Search for other papers by Xiao Mou in
Google Scholar
PubMed
Search for other papers by Chengcheng Xu in
Google Scholar
PubMed
Search for other papers by Tingting Zheng in
Google Scholar
PubMed
Search for other papers by Ling Bu in
Google Scholar
PubMed
Search for other papers by Xuan Luo in
Google Scholar
PubMed
Search for other papers by Qingyan Lu in
Google Scholar
PubMed
Search for other papers by Xuefeng Wang in
Google Scholar
PubMed
Introduction Hashimoto’s thyroiditis (HT) is a very common organ-specific autoimmune disease in which the Th1-mediated immune response contributes to the destruction of thyroid follicular cells (TFCs) ( 1 ). In recent years, many studies have
Search for other papers by Weiwei He in
Google Scholar
PubMed
Search for other papers by Bin Wang in
Google Scholar
PubMed
Search for other papers by Kaida Mu in
Google Scholar
PubMed
Search for other papers by Jing Zhang in
Google Scholar
PubMed
Search for other papers by Yanping Yang in
Google Scholar
PubMed
Search for other papers by Wei Yao in
Google Scholar
PubMed
Search for other papers by Sheli Li in
Google Scholar
PubMed
Search for other papers by Jin-an Zhang in
Google Scholar
PubMed
Introduction In autoimmune thyroid diseases (AITDs), one of the autoimmune diseases, cytokine-mediated immunity play a key role during the pathogenesis and development ( 1 ). AITDs are common endocrine autoimmune diseases with prevalence of
Search for other papers by John E M Midgley in
Google Scholar
PubMed
Search for other papers by Rolf Larisch in
Google Scholar
PubMed
North Lakes Clinical, Department of Nuclear Medicine, Medical Department I, Ruhr Center for Rare Diseases (CeSER), 20 Wheatley Avenue, Ilkley LS29 8PT, UK
Search for other papers by Johannes W Dietrich in
Google Scholar
PubMed
Search for other papers by Rudolf Hoermann in
Google Scholar
PubMed
, age, height, weight, smoking habits (75% answered), prior surgery or radioiodine treatment, thyroid medication (brand, dosage, duration, time of intake), other drugs, laboratory tests (FT 3 , FT 4 , TSH and, if autoimmune thyroiditis was suspected or
Search for other papers by Lia Ferreira in
Google Scholar
PubMed
Search for other papers by João Silva in
Google Scholar
PubMed
Search for other papers by Susana Garrido in
Google Scholar
PubMed
Search for other papers by Carlos Bello in
Google Scholar
PubMed
Search for other papers by Diana Oliveira in
Google Scholar
PubMed
Search for other papers by Hélder Simões in
Google Scholar
PubMed
Search for other papers by Isabel Paiva in
Google Scholar
PubMed
Search for other papers by Joana Guimarães in
Google Scholar
PubMed
Search for other papers by Marta Ferreira in
Google Scholar
PubMed
Search for other papers by Teresa Pereira in
Google Scholar
PubMed
Search for other papers by Rita Bettencourt-Silva in
Google Scholar
PubMed
Search for other papers by Ana Filipa Martins in
Google Scholar
PubMed
Search for other papers by Tiago Silva in
Google Scholar
PubMed
Search for other papers by Vera Fernandes in
Google Scholar
PubMed
Search for other papers by Maria Lopes Pereira in
Google Scholar
PubMed
Search for other papers by Adrenal Tumors Study Group of the Portuguese Society of Endocrinology in
Google Scholar
PubMed
diagnosed with one or more associated autoimmune endocrinopathies. The most common of these were autoimmune thyroiditis (60.7%), type 1 diabetes mellitus (17.3%) and pernicious anaemia (6.7%), with 103 (68.7%) of the patients meeting the criteria for APS-2
Search for other papers by Yun Cai in
Google Scholar
PubMed
Search for other papers by Jieni Yan in
Google Scholar
PubMed
Search for other papers by Yong Gu in
Google Scholar
PubMed
Search for other papers by Heng Chen in
Google Scholar
PubMed
Search for other papers by Yang Chen in
Google Scholar
PubMed
Search for other papers by Xinyu Xu in
Google Scholar
PubMed
Search for other papers by Mei Zhang in
Google Scholar
PubMed
Search for other papers by Liping Yu in
Google Scholar
PubMed
Search for other papers by Xuqin Zheng in
Google Scholar
PubMed
Search for other papers by Tao Yang in
Google Scholar
PubMed
Introduction Autoimmune thyroid disease (AITD) is a common organ-specific autoimmune disorder which includes two main clinical diseases: Graves’ disease (GD) and Hashimoto’s thyroiditis (HT). Both GD and HT share the immunologic manifestation
Search for other papers by Suvanjaa Sivalingam in
Google Scholar
PubMed
Search for other papers by Marianne Thvilum in
Google Scholar
PubMed
Search for other papers by Thomas Heiberg Brix in
Google Scholar
PubMed
Department of Clinical Research, University of Southern Denmark, Odense M, Denmark
Search for other papers by Laszlo Hegedüs in
Google Scholar
PubMed
Department of Regional Health Research, University of Southern Denmark, Odense M, Denmark
Search for other papers by Frans Brandt in
Google Scholar
PubMed
Introduction Hyperthyroidism is a common condition with a lifetime risk of 2–10% ( 1 , 2 ). Graves’ disease (GD) and toxic nodular goitre (TNG) are the most frequent causes of hyperthyroidism ( 2 ). GD is an organ specific autoimmune disease
Search for other papers by Maria Stelmachowska-Banaś in
Google Scholar
PubMed
Search for other papers by Izabella Czajka-Oraniec in
Google Scholar
PubMed
, 63 ). However, many patients with ICI-induced thyroiditis were negative for TPOAbs and TgAbs, which might suggest a pathogenesis that differs from the classical autoimmune spontaneous thyroiditis ( 64 ). It seems that antithyroid antibodies are not