Section of Pacing and Electrophysiology, Division of Cardiology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Dalian Medical University, Dalian, Liaoning, China
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Dalian Medical University, Dalian, Liaoning, China
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Introduction Thyroid hormone level changes with the development of acute myocardial infarction (AMI) in patients without prior thyroid disease ( 1 , 2 , 3 ). Low triiodothyronine (T3) syndrome (LT3S), manifested as serum T3 level and normal
Steno Diabetes Center Aarhus, Aarhus University Hospital, Aarhus, Denmark
Department of Clinical Epidemiology, Aarhus University Hospital and Aarhus University, Aarhus, Denmark
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Department of Clinical Medicine, Aarhus University, Aarhus, Denmark
Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark
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Steno Diabetes Center Aarhus, Aarhus University Hospital, Aarhus, Denmark
Department of Clinical Medicine, Aarhus University, Aarhus, Denmark
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Department of Clinical Medicine, Aarhus University, Aarhus, Denmark
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, employment, and the highest level of education achieved ( 24 ). Study outcomes Based on data from the DNPR, we identified all patients in the two cohorts with a first-time hospital diagnosis of CVD. We retrieved data on acute myocardial infarction (MI
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Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar São João, Porto, Portugal
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Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar São João, Porto, Portugal
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mechanism ( 31 ). T3 treatment (14 ng/g body weight, dose given daily) for 3 days after acute myocardial infarction in rats reduced myocyte apoptosis in the border area, possibly via Akt signaling ( 33 ). T3 administration in rats significantly increases
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Department of Internal Medicine, Section of Metabolic Vascular Medicine, Division of Diabetes and Nutritional Sciences, Cardiovascular Endocrinology Laboratory, Faculty of Medicine, Hospital de Urgencias, National University of Córdoba, X5000 Córdoba, Argentina
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per se creates a higher risk for developing systolic heart failure, especially if it is long-lasting and severe (BMI >40 kg/m 2 ). In contrast, the risk of acute myocardial infarction in ‘obese metabolically unhealthy’ subjects was significantly
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Division of Clinical Hypertension, Endocrinology and Metabolism, Tohoku University Graduate School of Medicine, Sendai, Miyagi, Japan
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criteria were a history of cardiac events (heart failure, myocardial infarction, valvular disease and arrhythmias) and severe renal dysfunction (estimated glomerular filtration rate <30 mL/min). Demographic characteristics of the patients were collected
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Department of Cardiology, Yanbian University Hospital, Yanji, China
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cardiovascular disease are assigned to the control group. The rest patients were divided into the acute non-ST-elevation myocardial infarction (NSTEMI) group, acute ST-elevation myocardial infarction (STEMI) group, and unstable angina (UA) group according to
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risk in patients with acute myocardial infarction: a prospective cohort study . BMC Cardiovascular Disorders 2019 19 50 . ( https://doi.org/10.1186/s12872-019-1027-1 ) 46 Ojamaa K Kenessey A Shenoy R Klein I . Thyroid hormone
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venous thromboembolism ( 6 , 7 , 8 , 9 ). Although reducing fasting plasma glucose levels to normal is seen as a way to prevent negative cardiovascular outcomes, such as myocardial infarction or stroke ( 10 ), the most recent study with insulin
Department of Endocrinology and Internal Medicine, Aarhus University Hospital, Aarhus N, Denmark
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Department of Molecular Medicine, Aarhus University Hospital, Aarhus N, Denmark
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205 – 213 . ( https://doi.org/10.7326/M17-2785 ) 29 Mehta LS Beckie TM DeVon HA Grines CL Krumholz HM Johnson MN Lindley KJ Vaccarino V Wang TY Watson KE , et al . Acute myocardial infarction in women: a scientific statement
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/72 Extra-adrenal 2/72 Tumour size in cm (mean ± s.d. ) 5.5 ± 2.9 Thirty-one patients had major complications including metastases, death and cardiovascular sequelae i.e. acute myocardial infarctions, acute cerebral events