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Jia Liu, Min Liu, Zhe Chen, Yumei Jia and Guang Wang

Introduction Autoimmune thyroiditis (AIT) is the most common autoimmune thyroid disease ( 1 , 2 ). Because of varying degree of thyroid destruction, AIT patients manifest with different thyroid function including euthyroidism, subclinical or

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Eugenie S Lim, Shanty G Shah, Mona Waterhouse, Scott Akker, William Drake, Nick Plowman, Daniel M Berney, Polly Richards, Ashok Adams, Ewa Nowosinska, Carmel Brennan and Maralyn Druce

antibodies, and conversely, patients with positive TPO antibodies may never develop thyroid disease ( 20 ). Antibody reactions in autoimmune thyroiditis targeted towards thyroid-specific antigens may hasten thyroid destruction ( 21 ). It has also been

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Mírian Romitti, Vitor C Fabris, Patricia K Ziegelmann, Ana Luiza Maia and Poli Mara Spritzer

stronger and more aggressive inflammatory response in the thyroid, subsequently leading to thyroid destruction and hypothyroidism ( 26 ). Interestingly, a recent study has shown that serum CXCL10 concentrations are increased in women with PCOS, which

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Jaafar Jaafar, Eugenio Fernandez, Heba Alwan and Jacques Philippe

antibodies were not always found in three other studies, suggesting a bystander role of these antibodies ( 74 , 75 , 88 ). To better understand the mechanism of thyroid destruction by PD-1 antibodies, Delivanis et al ., performed a peripheral blood flow