therapy (HRT) has been associated with adverse effects such as an increased incidence of breast cancer and thromboembolic events ( 1 , 2 , 3 ). Venous thromboembolism events (VTEs), which encompasses deep venous thrombosis (DVT) and pulmonary embolism
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Emmanuelle Noirrit, Mélissa Buscato, Marion Dupuis, Bernard Payrastre, Coralie Fontaine, Jean-François Arnal, and Marie-Cécile Valera
Simon Chang, Christian Fynbo Christiansen, Anders Bojesen, Svend Juul, Anna-Marie B Münster, and Claus H Gravholt
thromboembolisms (VTE) ( 8 , 9 ), increased risk of arterial thrombosis (ATE) ( 8 ), and a more than two-fold increased rate of mortality related to thrombotic events ( 10 , 11 , 12 ). Thrombotic risk in men with KS could be affected directly by the presence of
Thera P Links, Trynke van der Boom, Wouter T Zandee, and Joop D Lefrandt
and IX, fibrinogen, von Willebrand factor, and plasminogen activator inhibitor-1 factor, whereas these factors are not increased in euthyroid patients. This may be linked to a probable tendency toward thrombosis in patients with hyperthyroidism
Simon Chang, Arkadiusz J Goszczak, Anne Skakkebæk, Jens Fedder, Anders Bojesen, M Vakur Bor, Moniek P M de Maat, Claus H Gravholt, and Anna-Marie B Münster
criteria included prior thrombosis, current anticoagulation therapy or use of platelet inhibitors, current use of narcotics, diabetes mellitus, and prior severe head trauma. Male controls with total testosterone levels below the assay reference were
Marc Blondon, Emmanuel Biver, Olivia Braillard, Marc Righini, Pierre Fontana, and Alessandro Casini
Introduction Thrombosis, collectively including ischemic heart disease, stroke and venous thromboembolism (VTE), causes more than one in four deaths worldwide ( 1 ). Venous thromboembolism itself is among the largest contributor of loss of
Mette Bøgehave, Dorte Glintborg, Jørgen Brodersen Gram, Else-Marie Bladbjerg, Marianne Skovsager Andersen, and Johannes Jakobsen Sidelmann
). CVD can be caused by atherosclerosis and thrombosis, and alterations in the coagulation system ( Fig. 1 ) contribute significantly to the pathogenesis of thrombosis. It remains unclear whether TRT induces a pro- or anticoagulant state. Reportedly
Jülide Durmuşoğlu, Henri J L M Timmers, Pepijn van Houten, Johan F Langenhuijsen, Ad R M M Hermus, and Annenienke C van de Ven
mitotane. The primary outcome of our study was the incidence of venous thromboembolic events after adrenalectomy. We considered a VTE within 6 months after surgery as a postoperative VTE ( 23 ). VTE comprises pulmonary embolism and deep venous thrombosis
Malin Nylander, Signe Frøssing, Caroline Kistorp, Jens Faber, and Sven O Skouby
, Switzerland) and plasma levels of hsCRP using a CardioPhase hsCRP kit on a BNII protein analyzer (Siemens Healthcare Diagnostics GmbH), with CV 4.6, 6.4 and 3.1%, respectively. All analyses were performed at Unit for Thrombosis Research, Department of Clinical
Laura P B Elbers, Marije Wijnberge, Joost C M Meijers, Dennis C W Poland, Dees P M Brandjes, Eric Fliers, and Victor E A Gerdes
suppletion on markers of coagulation with conflicting results hindering any definitive conclusions ( 13 , 14 , 15 , 16 ). However, the current evidence indicates that vitamin D deficiency does not lead to an increased risk of venous thrombosis ( 17 , 18
Hong Wang, Jie Cao, Jian-bin Su, Xue-qin Wang, Dong-mei Zhang, and Xiao-hua Wang
of thrombosis ( 13 ). Under the pathological condition of T2D, hyperglycemia promotes the product of AGEs; as a result, non-enzymatic glycation leads to structural modifications of AT3 and favors thrombosis ( 14 ). However, the incidence and
