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Emmanuelle Noirrit, Mélissa Buscato, Marion Dupuis, Bernard Payrastre, Coralie Fontaine, Jean-François Arnal, and Marie-Cécile Valera

therapy (HRT) has been associated with adverse effects such as an increased incidence of breast cancer and thromboembolic events ( 1 , 2 , 3 ). Venous thromboembolism events (VTEs), which encompasses deep venous thrombosis (DVT) and pulmonary embolism

Open access

Simon Chang, Christian Fynbo Christiansen, Anders Bojesen, Svend Juul, Anna-Marie B Münster, and Claus H Gravholt

thromboembolisms (VTE) ( 8 , 9 ), increased risk of arterial thrombosis (ATE) ( 8 ), and a more than two-fold increased rate of mortality related to thrombotic events ( 10 , 11 , 12 ). Thrombotic risk in men with KS could be affected directly by the presence of

Open access

Thera P Links, Trynke van der Boom, Wouter T Zandee, and Joop D Lefrandt

and IX, fibrinogen, von Willebrand factor, and plasminogen activator inhibitor-1 factor, whereas these factors are not increased in euthyroid patients. This may be linked to a probable tendency toward thrombosis in patients with hyperthyroidism

Open access

Simon Chang, Arkadiusz J Goszczak, Anne Skakkebæk, Jens Fedder, Anders Bojesen, M Vakur Bor, Moniek P M de Maat, Claus H Gravholt, and Anna-Marie B Münster

criteria included prior thrombosis, current anticoagulation therapy or use of platelet inhibitors, current use of narcotics, diabetes mellitus, and prior severe head trauma. Male controls with total testosterone levels below the assay reference were

Open access

Marc Blondon, Emmanuel Biver, Olivia Braillard, Marc Righini, Pierre Fontana, and Alessandro Casini

Introduction Thrombosis, collectively including ischemic heart disease, stroke and venous thromboembolism (VTE), causes more than one in four deaths worldwide ( 1 ). Venous thromboembolism itself is among the largest contributor of loss of

Open access

Mette Bøgehave, Dorte Glintborg, Jørgen Brodersen Gram, Else-Marie Bladbjerg, Marianne Skovsager Andersen, and Johannes Jakobsen Sidelmann

). CVD can be caused by atherosclerosis and thrombosis, and alterations in the coagulation system ( Fig. 1 ) contribute significantly to the pathogenesis of thrombosis. It remains unclear whether TRT induces a pro- or anticoagulant state. Reportedly

Open access

Jülide Durmuşoğlu, Henri J L M Timmers, Pepijn van Houten, Johan F Langenhuijsen, Ad R M M Hermus, and Annenienke C van de Ven

mitotane. The primary outcome of our study was the incidence of venous thromboembolic events after adrenalectomy. We considered a VTE within 6 months after surgery as a postoperative VTE ( 23 ). VTE comprises pulmonary embolism and deep venous thrombosis

Open access

Malin Nylander, Signe Frøssing, Caroline Kistorp, Jens Faber, and Sven O Skouby

, Switzerland) and plasma levels of hsCRP using a CardioPhase hsCRP kit on a BNII protein analyzer (Siemens Healthcare Diagnostics GmbH), with CV 4.6, 6.4 and 3.1%, respectively. All analyses were performed at Unit for Thrombosis Research, Department of Clinical

Open access

Laura P B Elbers, Marije Wijnberge, Joost C M Meijers, Dennis C W Poland, Dees P M Brandjes, Eric Fliers, and Victor E A Gerdes

suppletion on markers of coagulation with conflicting results hindering any definitive conclusions ( 13 , 14 , 15 , 16 ). However, the current evidence indicates that vitamin D deficiency does not lead to an increased risk of venous thrombosis ( 17 , 18

Open access

Hong Wang, Jie Cao, Jian-bin Su, Xue-qin Wang, Dong-mei Zhang, and Xiao-hua Wang

of thrombosis ( 13 ). Under the pathological condition of T2D, hyperglycemia promotes the product of AGEs; as a result, non-enzymatic glycation leads to structural modifications of AT3 and favors thrombosis ( 14 ). However, the incidence and