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M Langeveld University of Cambridge Metabolic Research Laboratories, Wellcome Trust-MRC, Institute of Metabolic Science, Addenbrookes Hospital, Cambridge, UK

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C Y Tan University of Cambridge Metabolic Research Laboratories, Wellcome Trust-MRC, Institute of Metabolic Science, Addenbrookes Hospital, Cambridge, UK

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M R Soeters University of Cambridge Metabolic Research Laboratories, Wellcome Trust-MRC, Institute of Metabolic Science, Addenbrookes Hospital, Cambridge, UK

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S Virtue University of Cambridge Metabolic Research Laboratories, Wellcome Trust-MRC, Institute of Metabolic Science, Addenbrookes Hospital, Cambridge, UK

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G K Ambler Cambridge Vascular Unit, Addenbrookes Hospital, Hills Road, Cambridge, UK

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L P E Watson University of Cambridge Metabolic Research Laboratories, Wellcome Trust-MRC, Institute of Metabolic Science, Addenbrookes Hospital, Cambridge, UK
NIHR/Wellcome Trust Clinical Research Facility, Addenbrookes Hospital, Cambridge, UK

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P R Murgatroyd University of Cambridge Metabolic Research Laboratories, Wellcome Trust-MRC, Institute of Metabolic Science, Addenbrookes Hospital, Cambridge, UK
NIHR/Wellcome Trust Clinical Research Facility, Addenbrookes Hospital, Cambridge, UK

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V K Chatterjee University of Cambridge Metabolic Research Laboratories, Wellcome Trust-MRC, Institute of Metabolic Science, Addenbrookes Hospital, Cambridge, UK

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A Vidal-Puig University of Cambridge Metabolic Research Laboratories, Wellcome Trust-MRC, Institute of Metabolic Science, Addenbrookes Hospital, Cambridge, UK

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weight management strategy. More importantly, we focussed on changes in energy expenditure, food intake including appetite and satiety, and dermal temperature. Subjects and methods Subjects Healthy volunteers were recruited through local

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-16-0004
Volume/Issue:
Volume 5: Issue 2
Open access
Benjamin D Maylor Institute for Sport and Physical Activity Research, School of Sport Science and Physical Activity, University of Bedfordshire, Bedford, UK
Leicester Diabetes Centre, University of Leicester, Leicester General Hospital, Leicester, UK

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Julia K Zakrzewski-Fruer Institute for Sport and Physical Activity Research, School of Sport Science and Physical Activity, University of Bedfordshire, Bedford, UK

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Charlie J Orton Institute for Sport and Physical Activity Research, School of Sport Science and Physical Activity, University of Bedfordshire, Bedford, UK

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Daniel P Bailey Institute for Sport and Physical Activity Research, School of Sport Science and Physical Activity, University of Bedfordshire, Bedford, UK
Centre for Physical Activity in Health and Disease, Brunel University London, Uxbridge, UK
Division of Sport, Health and Exercise Sciences, Department of Life Sciences, Brunel University London, Uxbridge, UK

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-intensity physical activity breaks increased satiety, but had little effect on PYY, is similar to a prior study investigating responses to moderate-intensity physical activity breaks ( 10 ). The higher intensity of the physical activity in the present study may be

Article Type:
Research Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-22-0259
Volume/Issue:
Volume 12: Issue 2
Open access
H Vlaardingerbroek Department of Pediatrics, Subdivision of Endocrinology, Erasmus University Medical Center-Sophia Children’s Hospital, Rotterdam, The Netherlands
Willem-Alexander Children’s Hospital, Department of Pediatrics, Division of Endocrinology, Leiden University Medical Center, Leiden, The Netherlands

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E L T van den Akker Department of Pediatrics, Subdivision of Endocrinology, Erasmus University Medical Center-Sophia Children’s Hospital, Rotterdam, The Netherlands

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A C S Hokken-Koelega Department of Pediatrics, Subdivision of Endocrinology, Erasmus University Medical Center-Sophia Children’s Hospital, Rotterdam, The Netherlands

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-gene mutations that cause obesity can be detected. Hence, more and more genetic causes of monogenic and syndromic obesity are discovered. Hunger, satiety and energy expenditure are tightly controlled homeostatic processes, in which neuroendocrine factors play

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-21-0111
Volume/Issue:
Volume 10: Issue 5
Open access
Anette Lundqvist Department of Public Health and Clinical Medicine, Family Medicine, Umeå University, Umeå, Sweden

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Herbert Sandström Department of Public Health and Clinical Medicine, Family Medicine, Umeå University, Umeå, Sweden

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Torbjörn Bäckström Department of Clinical Sciences, Obstetrics and Gynecology, Umeå University, Umeå, Sweden

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appetite ( 16 ). Increased appetite and lack of satiety are mechanisms that result in overeating and development of obesity over time ( 19 ). Allopregnanolone dose dependently induces increased food intake in rodents ( 20 ) and makes the rodents prefer

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0046
Volume/Issue:
Volume 6: Issue 4
Open access
Shin-ya Ueda Department of Acupuncture, Morinomiya University of Medical Sciences, Osaka, Japan

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Hidehiro Nakahara Department of Acupuncture, Morinomiya University of Medical Sciences, Osaka, Japan

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Eriko Kawai Department of Environmental Physiology for Exercise, Osaka City University Graduate School of Medicine, Osaka, Japan

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Tatsuya Usui Department of Elementary and Preschool Education, Osaka Seikei College, Osaka, Japan

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Shintaro Tsuji Department of Elementary and Preschool Education, Osaka Seikei College, Osaka, Japan

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Tadayoshi Miyamoto Department of Acupuncture, Morinomiya University of Medical Sciences, Osaka, Japan

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, fullness, satiety and motivation to eat were reported on a 100-mm visual analog scale ( 19 ) and the feeling of dyspnea and leg fatigue were assessed according to Borg scales ( 20 ). Figure 1 Scheme of study 1. At 10:00 h ( t  = 60 min), a

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0323
Volume/Issue:
Volume 7: Issue 1
Open access
Bjarke R Medici Department of Medicine, Herlev Hospital, University of Copenhagen, Herlev, Denmark
Center for Clinical Metabolic Research, Gentofte Hospital, University of Copenhagen, Hellerup, Denmark

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Birte Nygaard Department of Medicine, Herlev Hospital, University of Copenhagen, Herlev, Denmark
Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark

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Jeppe L la Cour Department of Medicine, Herlev Hospital, University of Copenhagen, Herlev, Denmark

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Martin Krakauer Department of Clinical Physiology and Nuclear Medicine, Gentofte Hospital, University of Copenhagen, Hellerup, Denmark
Department of Clinical Physiology and Nuclear Medicine, Bispebjerg and Frederiksberg Hospital, University of Copenhagen, Copenhagen, Denmark

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Andreas Brønden Center for Clinical Metabolic Research, Gentofte Hospital, University of Copenhagen, Hellerup, Denmark
Department of Clinical Pharmacology, Bispebjerg and Frederiksberg Hospital, University of Copenhagen, Copenhagen, Denmark

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Mette P Sonne Department of Medicine, Herlev Hospital, University of Copenhagen, Herlev, Denmark

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Jens J Holst Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark

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Jens F Rehfeld Department of Clinical Biochemistry, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark

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Tina Vilsbøll Center for Clinical Metabolic Research, Gentofte Hospital, University of Copenhagen, Hellerup, Denmark
Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
Steno Diabetes Center Copenhagen, Herlev, Denmark

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Jens Faber Department of Medicine, Herlev Hospital, University of Copenhagen, Herlev, Denmark
Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark

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Filip K Knop Center for Clinical Metabolic Research, Gentofte Hospital, University of Copenhagen, Hellerup, Denmark
Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
Steno Diabetes Center Copenhagen, Herlev, Denmark

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even increasing body fat mass and that levothyroxine-induced increase in REE is accompanied by increased appetite and food intake, as mechanisms responsible for the lack of fat mass loss. Here, we evaluated appetite and satiety sensations as well as

Article Type:
Research Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-23-0314
Volume/Issue:
Volume 12: Issue 10
Open access
Alexis Sudlow Department of Upper GI Surgery, Southmead Hospital, Bristol, UK

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Carel W le Roux Department of Experimental Pathology, University College Dublin, Dublin, Ireland

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Dimitri J Pournaras Department of Upper GI Surgery, Southmead Hospital, Bristol, UK

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of restriction ( 29 ). The main effects resulting from AGB appear to be prolonged satiety, even during periods of fasting; however, it is unclear how this is mediated ( 29 ). There are, however, some mechanistic studies which have suggested that

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-19-0467
Volume/Issue:
Volume 9: Issue 2
Open access
Rajae Talbi Department of Medicine, Division of Endocrinology, Diabetes, and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA

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Victor M Navarro Department of Medicine, Division of Endocrinology, Diabetes, and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA

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neurons in the ARC that regulate hunger and satiety, suggesting the existence of a bidirectional metabolic-reproductive loop (discussed subsequently). The anorexigenic proopiomelanocortin (POMC) and orexigenic agouti-related peptide (AgRP)/neuropeptide Y

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-20-0068
Volume/Issue:
Volume 9: Issue 5
Open access
Metin Guclu Health Sciences University, Bursa Yuksek Ihtisas Education and Training Hospital, Department of Endocrinology and Metabolism, Bursa, Turkey

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Sinem Kiyici Health Sciences University, Bursa Yuksek Ihtisas Education and Training Hospital, Department of Endocrinology and Metabolism, Bursa, Turkey

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Zulfiye Gul Department of Pharmacology, Uludag University Medical Faculty, Bursa, Turkey

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Sinan Cavun Department of Pharmacology, Uludag University Medical Faculty, Bursa, Turkey

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liraglutide 3 mg/day has also been recently approved for the treatment of obesity. GLP-1 agonists are potential regulators of feeding behavior through their ability to inhibit gastric emptying, reduce food intake and induce satiety ( 4 , 5 ), but the exact

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0242
Volume/Issue:
Volume 7: Issue 1
Open access
Jens F Rehfeld Department of Clinical Biochemistry, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark

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secretion and growth, inhibition of gastric acid secretion via somatostatin cells and inhibition of gastric emptying ( 84 ), satiety via afferent vagal fibers ( 85 ), and gut motility. Only tyrosyl-sulfated CCK peptides are agonists for the CCK 1 receptor

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-21-0025
Volume/Issue:
Volume 10: Issue 3
Open access