Zhandong Lei
Zhandong Lei
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
Department of Anatomy, Shanxi Medical University, Taiyuan, China
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Department of Anatomy, Shanxi Medical University, Taiyuan, China
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Yunfei Chen
Yunfei Chen
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Jin Wang
Jin Wang
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Yan Zhang
Yan Zhang
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Wenjuan Shi
Wenjuan Shi
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Xuejiao Wang
Xuejiao Wang
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Dehai Xing
Dehai Xing
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Dongxue Li
Dongxue Li
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Xiangying Jiao
Xiangying Jiao
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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therapies provide promising approaches to the treatment of diabetes. Therefore, a key goal in the treatment of diabetes is to promote the proliferation of β cells and expand the functional β-cell mass. However, the mechanism driving β-cell proliferation as
Qi Che
Qi Che
Reproductive Medicine Center, Zhongshan Hospital, Fudan University, Shanghai, China
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Xirong Xiao
Xirong Xiao
Department of Obstetrics, Obstetrics and Gynecology Hospital, Fudan University, Shanghai, China
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Jun Xu
Jun Xu
Reproductive Medicine Center, Zhongshan Hospital, Fudan University, Shanghai, China
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Miao Liu
Miao Liu
Reproductive Medicine Center, Zhongshan Hospital, Fudan University, Shanghai, China
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Yongning Lu
Yongning Lu
Reproductive Medicine Center, Zhongshan Hospital, Fudan University, Shanghai, China
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Suying Liu
Suying Liu
Reproductive Medicine Center, Zhongshan Hospital, Fudan University, Shanghai, China
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Xi Dong
Xi Dong
Reproductive Medicine Center, Zhongshan Hospital, Fudan University, Shanghai, China
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mechanism of cancer proliferation and invasion bio-behavior will be of great significance for the clinical treatment of malignant tumor patients. As one of the most important hormone-dependent cancers, the tumorigenesis and progression of endometrial
Amarjit Saini
Amarjit Saini
Division of Clinical Physiology, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden
Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Linda Björkhem-Bergman
Linda Björkhem-Bergman
Division of Clinical Geriatrics, Departments of Neurobiology, Care Sciences and Neurobiology, Karolinska Institutet, Stockholm, Sweden
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Johan Boström
Johan Boström
Division of Clinical Physiology, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden
Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Mats Lilja
Mats Lilja
Division of Clinical Physiology, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden
Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Michael Melin
Michael Melin
Division of Clinical Physiology, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden
Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
Unit of Cardiology, Karolinska University Hospital, Stockholm, Sweden
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Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
Unit of Cardiology, Karolinska University Hospital, Stockholm, Sweden
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Karl Olsson
Karl Olsson
Division of Clinical Physiology, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden
Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Lena Ekström
Lena Ekström
Division of Clinical Pharmacology, Department of Laboratory Medicine, Karolinska Institutet, Stockholm, Sweden
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Peter Bergman
Peter Bergman
Division of Clinical Microbiology, Department of Laboratory Medicine, Karolinska Institutet, Stockholm, Sweden
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Mikael Altun
Mikael Altun
Division of Clinical Physiology, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden
Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Eric Rullman
Eric Rullman
Division of Clinical Physiology, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden
Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
Unit of Cardiology, Karolinska University Hospital, Stockholm, Sweden
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Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
Unit of Cardiology, Karolinska University Hospital, Stockholm, Sweden
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Thomas Gustafsson
Thomas Gustafsson
Division of Clinical Physiology, Department of Laboratory Medicine, Karolinska Institutet, Karolinska University Hospital, Stockholm, Sweden
Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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Unit of Clinical Physiology, Karolinska University Hospital, Stockholm, Sweden
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on myoblast proliferation and differentiation suggesting it is important in maintaining the SC pool ( 16 ). The VDR gene has multiple polymorphisms, most of which are identified by a bi-allelic variation in the restriction endonuclease site (e
Ping Gu
Ping Gu
Department of Neurology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China
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Yuege Lin
Yuege Lin
Department of Pathology, Nanjing Medical University, Nanjing, China
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Qi Wan
Qi Wan
Department of Neurology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China
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Dongming Su
Dongming Su
Department of Pathology, Nanjing Medical University, Nanjing, China
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Qun Shu
Qun Shu
Department of Obstetrics, Shanghai First Maternity and Infant Health Hospital, School of Medicine, Tongji University, Shanghai, China
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to investigate the possible mechanism of oxytocin regulation of β-cell function and proliferation. Materials and methods Clinical characteristics During 2018–2019, pregnant women aged 18–40 years who were diagnosed with GDM at the Shanghai
Xu-Ting Song
Xu-Ting Song
Key Laboratory of Animal Genetics, Breeding and Reproduction, Education Department of Heilongjiang Province, College of Animal Science and Technology
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Jia-Nan Zhang
Jia-Nan Zhang
Key Laboratory of Animal Genetics, Breeding and Reproduction, Education Department of Heilongjiang Province, College of Animal Science and Technology
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Duo-Wei Zhao
Duo-Wei Zhao
Key Laboratory of Animal Genetics, Breeding and Reproduction, Education Department of Heilongjiang Province, College of Animal Science and Technology
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Yu-Fei Zhai
Yu-Fei Zhai
Key Laboratory of Animal Genetics, Breeding and Reproduction, Education Department of Heilongjiang Province, College of Animal Science and Technology
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Qi Lu
Qi Lu
Key Laboratory of Animal Genetics, Breeding and Reproduction, Education Department of Heilongjiang Province, College of Animal Science and Technology
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Mei-Yu Qi
Mei-Yu Qi
Institute of Animal Husbandry, Heilongjiang Academy of Agricultural Sciences, Harbin, China
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Ming-Hai Lu
Ming-Hai Lu
Department of Animal Science, Heilongjiang State Farms Science Technology Vocational College, Harbin, China
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Shou-Long Deng
Shou-Long Deng
CAS Key Laboratory of Genome Sciences and Information, Beijing Institute of Genomics, Chinese Academy of Sciences, Beijing, China
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Hong-Bing Han
Hong-Bing Han
Beijing Key Laboratory of Animal Genetic Improvement, China Agricultural University, Beijing, China
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Xiu-Qin Yang
Xiu-Qin Yang
Key Laboratory of Animal Genetics, Breeding and Reproduction, Education Department of Heilongjiang Province, College of Animal Science and Technology
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Yu-Chang Yao
Yu-Chang Yao
Key Laboratory of Animal Genetics, Breeding and Reproduction, Education Department of Heilongjiang Province, College of Animal Science and Technology
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myoblast proliferation and muscle repair. During skeletal muscle injury, IGF1 Ea is responsible for satellite cell differentiation, whereas IGF1 Ec is responsible for satellite cell activation and proliferation ( 16 , 17 ). Different splicing patterns of
Tian Zhou
Tian Zhou
School of Clinical Medicine, GuiZhou Medical University, Guiyang, Guizhou, China
Department of Breast Surgery, Affiliated Hospital of Guizhou Medical University, Guiyang, Guizhou, China
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Department of Breast Surgery, Affiliated Hospital of Guizhou Medical University, Guiyang, Guizhou, China
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Dai-wei Zhao
Dai-wei Zhao
School of Clinical Medicine, GuiZhou Medical University, Guiyang, Guizhou, China
Department of Surgery, Second People's Hospital of Guizhou Province, Guiyang, Guizhou, China
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Department of Surgery, Second People's Hospital of Guizhou Province, Guiyang, Guizhou, China
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Ning Ma
Ning Ma
School of Clinical Medicine, GuiZhou Medical University, Guiyang, Guizhou, China
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Xue-ying Zhu
Xue-ying Zhu
School of Clinical Medicine, GuiZhou Medical University, Guiyang, Guizhou, China
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Xing-hong Chen
Xing-hong Chen
Department of Surgery, Second People's Hospital of Guizhou Province, Guiyang, Guizhou, China
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Xue Luo
Xue Luo
Department of Breast Surgery, Affiliated Hospital of Guizhou Medical University, Guiyang, Guizhou, China
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Song Chen
Song Chen
School of Clinical Medicine, GuiZhou Medical University, Guiyang, Guizhou, China
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Qing-jun Gao
Qing-jun Gao
Department of Thyroid Surgery, Affiliated Hospital of Guizhou Medical University, Guiyang, Guizhou, China
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knockdown and overexpression. (F) Promotion of cancer cell proliferation by FOXP4 confirmed by cell-cycle experiments. Forkhead box P4 knockdown suppressed the proliferation and migration of thyroid cancer cell lines and arrested cell cycles in
Corina Verónica Sasso
Corina Verónica Sasso
Instituto de Medicina y Biología Experimental de Cuyo (IMBECU), CCT-Mendoza CONICET, Mendoza, Argentina
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Flavia Eliana Santiano
Flavia Eliana Santiano
Instituto de Medicina y Biología Experimental de Cuyo (IMBECU), CCT-Mendoza CONICET, Mendoza, Argentina
Universidad de Mendoza, Mendoza, Argentina
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Universidad de Mendoza, Mendoza, Argentina
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Fiorella Campo Verde Arboccó
Fiorella Campo Verde Arboccó
Instituto de Medicina y Biología Experimental de Cuyo (IMBECU), CCT-Mendoza CONICET, Mendoza, Argentina
Universidad de Mendoza, Mendoza, Argentina
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Universidad de Mendoza, Mendoza, Argentina
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Leila Ester Zyla
Leila Ester Zyla
Instituto de Medicina y Biología Experimental de Cuyo (IMBECU), CCT-Mendoza CONICET, Mendoza, Argentina
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Silvana Noemí Semino
Silvana Noemí Semino
Hospital Universitario, Universidad Nacional de Cuyo, Mendoza, Argentina
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Martin Eduardo Guerrero-Gimenez
Martin Eduardo Guerrero-Gimenez
Instituto de Medicina y Biología Experimental de Cuyo (IMBECU), CCT-Mendoza CONICET, Mendoza, Argentina
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Virginia Pistone Creydt
Virginia Pistone Creydt
Instituto de Medicina y Biología Experimental de Cuyo (IMBECU), CCT-Mendoza CONICET, Mendoza, Argentina
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Constanza Matilde López Fontana
Constanza Matilde López Fontana
Instituto de Medicina y Biología Experimental de Cuyo (IMBECU), CCT-Mendoza CONICET, Mendoza, Argentina
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Rubén Walter Carón
Rubén Walter Carón
Instituto de Medicina y Biología Experimental de Cuyo (IMBECU), CCT-Mendoza CONICET, Mendoza, Argentina
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models showed a lower risk of CRC in the presence of estrogens ( 3 , 4 ). Nevertheless, some studies indicate that, once the disease has developed, estrogens inhibit cell proliferation, while others suggest they induce mitogenic effects ( 5 ). The
Bushra Shahida
Bushra Shahida
Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden
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Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden
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Tereza Planck
Tereza Planck
Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden
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Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden
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Tania Singh
Tania Singh
Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
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Peter Åsman
Peter Åsman
Department of Clinical Sciences Malmö, Ophthalmology, Lund University, Malmö, Sweden
Department of Ophthalmology, Skåne University Hospital, Malmö, Sweden
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Department of Ophthalmology, Skåne University Hospital, Malmö, Sweden
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Mikael Lantz
Mikael Lantz
Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden
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Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden
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antigen-presenting cells including dendritic cells, monocytes, and B lymphocytes. Upon encountering antigen-presenting cells, T lymphocytes undergo proliferation, differentiating into distinct phenotypes. While some evolve into effector T lymphocytes
Jintao Hu
Jintao Hu
Department of Neurosurgery, Xinqiao Hospital, The Army Medical University, Chongqing, China
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Qingbo Chen
Qingbo Chen
Department of Neurosurgery, Xinqiao Hospital, The Army Medical University, Chongqing, China
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Xiao Ding
Xiao Ding
Department of Neurosurgery, Xinqiao Hospital, The Army Medical University, Chongqing, China
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Xin Zheng
Xin Zheng
Department of Neurosurgery, Xinqiao Hospital, The Army Medical University, Chongqing, China
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Xuefeng Tang
Xuefeng Tang
Department of Pathology, Xinqiao Hospital, The Army Medical University, Chongqing, China
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Song Li
Song Li
Department of Neurosurgery, Xinqiao Hospital, The Army Medical University, Chongqing, China
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Hui Yang
Hui Yang
Department of Neurosurgery, Xinqiao Hospital, The Army Medical University, Chongqing, China
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proliferation assay We used the WST-8 Cell Counting Kit-8 (CCK-8, Dojindo Laboratories, Mashiki-machi, Kumamoto, Japan) to measure cell proliferation based on the manufacturer’s instructions. The doubling time (DT) was obtained using the exponential growth
Xuechao Jiang
Xuechao Jiang
Scientific Research Center, Xinhua Hospital, Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China
Department of Pediatric Cardiology, Xinhua Hospital, Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Department of Pediatric Cardiology, Xinhua Hospital, Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Yonghui Wang
Yonghui Wang
Department of Endocrinology, Fifth People’s Hospital of Shanghai Fudan University, Shanghai, China
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Xiaoying Li
Xiaoying Li
Department of Endocrinology, Fifth People’s Hospital of Shanghai Fudan University, Shanghai, China
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Leqi He
Leqi He
Department of Clinical Laboratory Medicine, Fifth People’s Hospital of Shanghai Fudan University, Shanghai, China
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Qian Yang
Qian Yang
Department of Endocrinology, Fifth People’s Hospital of Shanghai Fudan University, Shanghai, China
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Wei Wang
Wei Wang
Department of Endocrinology, Fifth People’s Hospital of Shanghai Fudan University, Shanghai, China
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Jun Liu
Jun Liu
Department of Endocrinology, Fifth People’s Hospital of Shanghai Fudan University, Shanghai, China
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Bingbing Zha
Bingbing Zha
Department of Endocrinology, Fifth People’s Hospital of Shanghai Fudan University, Shanghai, China
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focused on lncRNA–mRNA pairs that were abnormally regulated in GD, especially those involving protein-coding genes that were related to cell proliferation. Among the identified genes, TCL1 family AKT coactivator A ( TCL1A ), and SH2 domain containing 1A
