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Adriana J van Ballegooijen, Marjolein Visser, Marieke B Snijder, Jacqueline M Dekker, Giel Nijpels, Coen D A Stehouwer, Michaela Diamant and Ingeborg A Brouwer

serum 25-hydroxyvitamin D (25(OH)D) levels, the principal circulating storage form, will result in proportionally higher parathyroid hormone (PTH) levels to maintain serum and total body calcium. Vitamin D and PTH receptors have been detected in

Open access

Rolf Jorde and Guri Grimnes

sufficient ( 6 ), whereas a level of 75 nmol/L has been recommended by the Endocrine Society ( 7 ). These recommendations are partly based on calcium absorption, presence of signs of osteomalacia, as well suppression of serum parathyroid hormone (PTH) by

Open access

Laura P B Elbers, Marije Wijnberge, Joost C M Meijers, Dennis C W Poland, Dees P M Brandjes, Eric Fliers and Victor E A Gerdes

hyperparathyroidism underlined the conflicting results of studies investigating the physiological effects of parathyroid hormone (PTH) and parathyroidectomy on, amongst others, endothelial dysfunction ( 4 ). In our current study, the objective was to study the direct

Open access

Marc Blondon, Emmanuel Biver, Olivia Braillard, Marc Righini, Pierre Fontana and Alessandro Casini

Hoofnagle A Sarnak MJ Shlipak MG Jenny NS Siscovick DS . Vitamin D, parathyroid hormone, and cardiovascular events among older adults . Journal of the American College of Cardiology 2011 1433 – 1441 . ( https://doi.org/10.1016/j.jacc.2011

Open access

Stan Ursem, Vito Francic, Martin Keppel, Verena Schwetz, Christian Trummer, Marlene Pandis, Felix Aberer, Martin R Grübler, Nicolas D Verheyen, Winfried März, Andreas Tomaschitz, Stefan Pilz, Barbara Obermayer-Pietsch and Annemieke C Heijboer

Introduction Parathyroid hormone (PTH) plays a critical role in maintaining adequate serum calcium homeostasis. It increases serum calcium by stimulating bone resorption, promoting phosphate excretion, converting vitamin D to its active form

Open access

Göran Oleröd, Lillemor Mattsson Hultén, Ola Hammarsten and Eva Klingberg

be used as LoQ. Serum intact parathyroid hormone (iPTH) was analyzed with CLIA on an Abbott ARCHITECT instrument (Abbott Diagnostics Division) and serum 1,25(OH) 2 D was analyzed on an IDS-iSYS instrument (Immunodiagnostic Systems Holdings, Boldon

Open access

Laura J Reid, Bala Muthukrishnan, Dilip Patel, Mike S Crane, Murat Akyol, Andrew Thomson, Jonathan R Seckl and Fraser W Gibb

undiagnosed as suggested by a recent study which identified measurement of parathyroid hormone (PTH) in only 31% of hypercalcaemic patients ( 4 ). In recent years, the normocalcaemic variant of PHPT has gained increasing recognition, although this is beyond

Open access

Martine Cohen-Solal, Thomas Funck-Brentano and Pablo Ureña Torres

decision in patients with CKD and fracture. Treatment decisions are dependent of the levels of parathyroid hormone (PTH) and bone specific alkaline phosphatase (BSAP) levels. The different conditions are shown, to guide treatment decisions appropriately

Open access

E Vignali, F Cetani, S Chiavistelli, A Meola, F Saponaro, R Centoni, L Cianferotti and C Marcocci

Introduction During the past 10–20 years several investigators have noticed that there are patients with elevated serum parathyroid hormone (PTH) levels associated with consistently normal serum calcium concentration and no other causes of secondary

Open access

Ranganathan R Rao, Harpal S Randeva, Sailesh Sankaranarayanan, Murthy Narashima, Matthias Möhlig, Hisham Mehanna and Martin O Weickert

Introduction Primary hyperparathyroidism (pHPT) in postmenopausal women is frequently associated with vitamin D deficiency (1, 2) , which in turn is an additional stimulus for further increases in circulating parathyroid hormone (PTH) levels (3, 4