Tingting Shu
Tingting Shu
Department of Central Laboratory, Jiangsu Province Official Hospital, Nanjing, Jiangsu, China
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Zhigang Lv
Zhigang Lv
Department of Central Laboratory, Jiangsu Province Official Hospital, Nanjing, Jiangsu, China
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Yuchun Xie
Yuchun Xie
Department of Central Laboratory, Jiangsu Province Official Hospital, Nanjing, Jiangsu, China
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Junming Tang
Junming Tang
Department of Clinical Laboratory, Yixing People Hospital, Affiliated Jiangsu University, Yixing, Wuxi, Jiangsu, China
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Xuhua Mao
Xuhua Mao
Department of Clinical Laboratory, Yixing People Hospital, Affiliated Jiangsu University, Yixing, Wuxi, Jiangsu, China
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inflammatory response, oxidative stress ( 12 ) and mitochondrial dysfunction pathways ( 13 ), which affect glucose metabolism in peripheral tissues ( 5 ). To date, there have been few reports describing the role of hepcidin in pancreatic β-cells. Our previous
Caio Jordão Teixeira
Caio Jordão Teixeira
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Junia Carolina Santos-Silva
Junia Carolina Santos-Silva
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Dailson Nogueira de Souza
Dailson Nogueira de Souza
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Alex Rafacho
Alex Rafacho
Department of Physiological Sciences, Center of Biological Sciences, Federal University of Santa Catarina, Florianópolis, Brazil
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Gabriel Forato Anhe
Gabriel Forato Anhe
Department of Pharmacology, Faculty of Medical Sciences, State University of Campinas, Campinas, Brazil
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Silvana Bordin
Silvana Bordin
Department of Physiology and Biophysics, Institute of Biomedical Sciences, University of Sao Paulo, Sao Paulo, Brazil
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). Undisturbed pregnancies in humans and rodents are also distinguished by morphological changes in the pancreatic islets. The most evident morphological adaptations described in the human endocrine pancreas are an increase in the pancreatic β-cell fractional
Chenmin Wei
Chenmin Wei
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
Department of Endocrinology, The Affiliated Jiangning Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Department of Endocrinology, The Affiliated Jiangning Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Zichen Zhang
Zichen Zhang
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Qi Fu
Qi Fu
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Yunqiang He
Yunqiang He
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Tao Yang
Tao Yang
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Min Sun
Min Sun
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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recovered after FFA deprivation, suggesting the presence of other pathways controlling the sensitivity of the islets’ glucose response. Cruz-Cruz et al. reported that the unitary conductance and ATP sensitivity of K ATP channels in pancreatic β cells in a
Zhandong Lei
Zhandong Lei
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
Department of Anatomy, Shanxi Medical University, Taiyuan, China
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Department of Anatomy, Shanxi Medical University, Taiyuan, China
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Yunfei Chen
Yunfei Chen
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Jin Wang
Jin Wang
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Yan Zhang
Yan Zhang
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Wenjuan Shi
Wenjuan Shi
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Xuejiao Wang
Xuejiao Wang
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Dehai Xing
Dehai Xing
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Dongxue Li
Dongxue Li
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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Xiangying Jiao
Xiangying Jiao
Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
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diabetes mellitus (T2DM), a deficiency in the functional pancreatic β-cell mass and decreased numbers of β cells cause insufficiency of insulin secretion, which constitutes the key mechanism of DM ( 4 ). Pancreatic β-cell replacement and regeneration
Xue-Lian Zhang
Xue-Lian Zhang
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Xinyi Zhao
Xinyi Zhao
Department of Physiology, School of Medicine, Jinan University, Guangzhou, China
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Yong Wu
Yong Wu
Department of Physiology, School of Medicine, Jinan University, Guangzhou, China
Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Wen-qing Huang
Wen-qing Huang
Department of Transfusion Medicine, Shenzhen Hospital, Southern Medical University, Shenzhen, Guangdong, China
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Jun-jiang Chen
Jun-jiang Chen
Department of Physiology, School of Medicine, Jinan University, Guangzhou, China
Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Peijie Hu
Peijie Hu
Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Wei Liu
Wei Liu
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Yi-Wen Chen
Yi-Wen Chen
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Jin Hao
Jin Hao
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Rong-Rong Xie
Rong-Rong Xie
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Hsiao Chang Chan
Hsiao Chang Chan
Epithelial Cell Biology Research Center, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong SAR, China
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Ye Chun Ruan
Ye Chun Ruan
Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Hui Chen
Hui Chen
Cell-Gene Therapy Translational Medicine Research Center, The Third Affiliated Hospital of Sun Yat-Sen University, Guangzhou, China
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Jinghui Guo
Jinghui Guo
Department of Physiology, School of Medicine, Jinan University, Guangzhou, China
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analyzed by two-way ANOVA. Pearson test was used for correlation analysis. A P value <0.05 was considered statistically significant. Results Ang(1–7) upregulates CFTR to potentiate insulin secretion in the pancreatic β-cell line Given the
Chenghao Piao
Chenghao Piao
Department of Radiology, The Second Affiliated Hospital of Shenyang Medical College, Shenyang City, Liaoning Province, People’s Republic of China
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Xiaojie Wang
Xiaojie Wang
Department of Human Anatomy, Shenyang Medical College, Shenyang City, Liaoning Province, People’s Republic of China
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Shiqiao Peng
Shiqiao Peng
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang City, Liaoning Province, People’s Republic of China
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Xinyu Guo
Xinyu Guo
Department of Obstetrics, The Second Affiliated Hospital of Shenyang Medical College, Shenyang City, Liaoning Province, People’s Republic of China
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Hui Zhao
Hui Zhao
Department of Laboratory Medicine, The Second Affiliated Hospital of Shenyang Medical College, Shenyang City, Liaoning Province, People’s Republic of China
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Li He
Li He
Department of Gastroenterology, The Second Affiliated Hospital of Shenyang Medical College, Shenyang City, Liaoning Province, People’s Republic of China
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Yan Zeng
Yan Zeng
Department of Obstetrics, The Second Affiliated Hospital of Shenyang Medical College, Shenyang City, Liaoning Province, People’s Republic of China
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Fan Zhang
Fan Zhang
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Affiliated Hospital of China Medical University, Shenyang City, Liaoning Province, People’s Republic of China
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Kewen Zhu
Kewen Zhu
Department of Human Anatomy, Shenyang Medical College, Shenyang City, Liaoning Province, People’s Republic of China
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Yiwei Wang
Yiwei Wang
Department of Human Anatomy, Shenyang Medical College, Shenyang City, Liaoning Province, People’s Republic of China
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foetus for development ( 5 ). The need for maternal insulin is an elevated result of gestational insulin resistance, which is impacted by attenuated food intake and placental hormone production. Consequently, the sizes of pancreatic β cells are enhanced
Ping Gu
Ping Gu
Department of Neurology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China
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Yuege Lin
Yuege Lin
Department of Pathology, Nanjing Medical University, Nanjing, China
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Qi Wan
Qi Wan
Department of Neurology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China
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Dongming Su
Dongming Su
Department of Pathology, Nanjing Medical University, Nanjing, China
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Qun Shu
Qun Shu
Department of Obstetrics, Shanghai First Maternity and Infant Health Hospital, School of Medicine, Tongji University, Shanghai, China
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Introduction During pregnancy, the increased energy demand for fetal growth leads to a higher glucose metabolic ratio ( 1 ). As a physiological adaptation, insulin secretion by pancreatic β-cells is increased, along with a compensatory
K E Lines
K E Lines
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R P Vas Nunes
R P Vas Nunes
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M Frost
M Frost
Academic Endocrine Unit, OCDEM, University of Oxford, Churchill Hospital, Oxford, UK
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C J Yates
C J Yates
Academic Endocrine Unit, OCDEM, University of Oxford, Churchill Hospital, Oxford, UK
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M Stevenson
M Stevenson
Academic Endocrine Unit, OCDEM, University of Oxford, Churchill Hospital, Oxford, UK
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R V Thakker
R V Thakker
Academic Endocrine Unit, OCDEM, University of Oxford, Churchill Hospital, Oxford, UK
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under the influence of a tissue-specific promoter ( 25 , 26 ). This has led to the development of mouse models in which pancreatic β-cell-specific rat insulin promoters are used to target Cre expression ( RIP-Cre ) and produce mice harbouring PNETs that
Thozhukat Sathyapalan
Thozhukat Sathyapalan
Department of Academic Diabetes, Endocrinology and Metabolism, Hull York Medical School, University of Hull, Hull, UK
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Anne-Marie Coady
Anne-Marie Coady
Department of Obstetric Ultrasound, Hull and East Yorkshire Women’s and Children’s Hospital, Hull, UK
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Eric S Kilpatrick
Eric S Kilpatrick
Department of Clinical Biochemistry, Sidra Medical and Research Center, Doha, Qatar
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Stephen L Atkin
Stephen L Atkin
Department of Medicine, Weill Cornell Medical College, Doha, Qatar
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reduction in insulin resistance especially with weight loss in patients with T2DM will result in normalisation of pancreatic β-cell requirement and even reversal of T2DM ( 9 ). Statins have shown to improve certain features of polycystic ovary syndrome
Hui-qing Yuan
Hui-qing Yuan
Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Jia-xi Miao
Jia-xi Miao
Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Jia-ping Xu
Jia-ping Xu
Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Su-xiang Zhu
Su-xiang Zhu
Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Feng Xu
Feng Xu
Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Xiao-hua Wang
Xiao-hua Wang
Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Chun-hua Wang
Chun-hua Wang
Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Chao Yu
Chao Yu
Department of Clinical Laboratory, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Xue-qin Wang
Xue-qin Wang
Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Jian-bin Su
Jian-bin Su
Department of Endocrinology, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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Dong-mei Zhang
Dong-mei Zhang
Medical Research Center, Affiliated Hospital 2 of Nantong University, and First People’s Hospital of Nantong City, Nantong, China
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due to inadequate compensatory insulin secretion from pancreatic β-cells when they counteract insulin resistance ( 1 , 2 ). In addition, numerous findings highlighted that abnormal α-cell secretion may take part in the progression and exacerbation of
