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Małgorzata Fuksiewicz, Maria Kowalska, Agnieszka Kolasińska-Ćwikła, Jarosław B Ćwikła, Łukasz Sawicki, Katarzyna Roszkowska-Purska, Joanna Drygiel and Beata Kotowicz

digestive tract, including 59 with tumours located in the pancreas and 72 with lesions in the small intestine, caecum and appendix (midgut – the tumours originating from the central part of the archenteron) and in the colon (hindgut – tumours of the

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Gaëtan Prévost, Marie Picot, Marie-Anne Le Solliec, Arnaud Arabo, Hind Berrahmoune, Mouna El Mehdi, Saloua Cherifi, Alexandre Benani, Emmanuelle Nédélec, Françoise Gobet, Valéry Brunel, Jérôme Leprince, Hervé Lefebvre, Youssef Anouar and Nicolas Chartrel

Introduction Glucose homeostasis is a crucial parameter to maintain normal body function. Control of blood glucose levels is ensured by a highly sophisticated network of various hormones released by the pancreas, intestine, liver, adipose and

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Logan Mills, Panagiotis Drymousis, Yogesh Vashist, Christoph Burdelski, Andreas Prachalias, Parthi Srinivasan, Krishna Menon, Corina Cotoi, Saboor Khan, Judith Cave, Thomas Armstrong, Martin O Weickert, Jakob Izbicki, Joerg Schrader, Andreja Frilling, John K Ramage and Raj Srirajaskanthan

( Table 1 ). Larger tumours were significantly more likely to originate from the tail of the pancreas ( P  = 0.009) and to present with distant metastases ( P  = 0.001). Table 1 Demographics and pre-operative characteristics of patients

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Julia Modesto Vicente, Junia Carolina Santos-Silva, Caio Jordão Teixeira, Dailson Nogueira de Souza, Jean Franciesco Vettorazzi, Fabiola Sales Furtuoso, Isabel Gouveia Adabo, Fabio Takeo Sato, Marco Aurélio Ramirez Vinolo, Everardo Magalhães Carneiro, Silvana Bordin and Gabriel Forato Anhê

Fig. 3A. Fat pad weights were expressed as the percentage of body weight ( Table 1 ). Table 1 Adiposity and pancreas morphometric data of Virgin, L0 and L21 mice. Virgin L0 L21 Inguinal fat pad (% of body weight) 1

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Stavroula A Paschou, Nektaria Papadopoulou-Marketou, George P Chrousos and Christina Kanaka-Gantenbein

pancreas. A small percentage of affected patients (<10%) are classified as type 1B, with no evidence of autoimmunity and the pathogenesis in these cases is considered idiopathic ( 1 , 2 ). The aim of this comprehensive review is to present updated

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Jukka Koffert, Henri Honka, Jarmo Teuho, Saila Kauhanen, Saija Hurme, Riitta Parkkola, Vesa Oikonen, Andrea Mari, Andreas Lindqvist, Nils Wierup, Leif Groop and Pirjo Nuutila

Introduction The splanchnic region is a crucial regulator of metabolic homeostasis; and disturbances in the function of gut and pancreas predispose to impaired glucose regulation in the postprandial state ( 1 , 2 , 3 ). After a meal, blood

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Zi-Di Xu, Wei Zhang, Min Liu, Huan-Min Wang, Pei-Pei Hui, Xue-Jun Liang, Jie Yan, Yu-Jun Wu, Yan-Mei Sang, Cheng Zhu and Gui-Chen Ni

pancreas to determine the pancreatic histological type, while hypoglycemia can be controlled after the patients receive different degrees of pancreatectomy ( 13 ). There are few studies on CHI in Chinese children. In the present study, 60 CHI children

Open access

Taís S Assmann, Mariana Recamonde-Mendoza, Bianca M De Souza and Daisy Crispim

greater depth the functional involvement of miRNAs in T1DM, we selected those miRNAs that were consistently dysregulated in T1DM-related tissues (PBMCs, serum/plasma and pancreas) and performed bioinformatic analysis to retrieve their putative targets and

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Jairo Arturo Pinzón-Cortés, Angelina Perna-Chaux, Nicolás Steven Rojas-Villamizar, Angélica Díaz-Basabe, Diana Carolina Polanía-Villanueva, María Fernanda Jácome, Carlos Olimpo Mendivil, Helena Groot and Valeriano López-Segura

) Comparison of the levels of global methylation in the MFI (mean fluorescence intensity units) between patients with T2DM and controls in peripheral blood, pancreas, fat and muscle, respectively. The comparison was conducted using Student’s T test, Welch T

Open access

Irvin M Modlin, Harry Aslanian, Lisa Bodei, Ignat Drozdov and Mark Kidd

1 included 91 GEP-NETs (gastric, n =3; duodenum, n =1; pancreas, n =41; small intestine, n =40; appendix, n =3; and colorectum, n =3), 18 with an unknown primary, and 16 non-GEP-NETs. Histopathologically, 51% were G1, 27% G2, and 12% G3