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effect of menopausal hormone therapy (MHT) on this risk is also discussed. Does transition to menopause predispose to higher CVD risk? Menopause, defined as the completion of 12 months since the final menstrual period (FMP) or at the time of
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estrogen treatment, several investigators examined immunoassay performance among adults taking oral exogenous estradiol treatment for either ovarian stimulation or menopausal hormone therapy ( 17 , 18 ). Dancoine et al. observed agreement between an
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Department of Obstetrics and Gynecology, Department of Research, University of Washington, Box 356460, 1959 NE Pacific Street, Seattle, Washington, USA
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CD002229 . ( doi:10.1002/14651858.CD002229.pub4 ). 88 Fournier A Mesrine S Boutron-Ruault MC Clavel-Chapelon F . Estrogen–progestagen menopausal hormone therapy and breast cancer: does delay from menopause onset to treatment initiation
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cohort study . BMJ 2014 349 g5992 . ( https://doi.org/10.1136/bmj.g5992 ) 23 Prabakaran S Schwartz A & Lundberg G . Cardiovascular risk in menopausal women and our evolving understanding of menopausal hormone therapy: risks, benefits, and
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development in a prospective study ( 7 ). There are currently no data with micronised progesterone. Micronised progesterone is often prescribed for endometrial protection with oestradiol as menopausal hormone therapy for cisgender women with an intact uterus
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). All the study participants underwent a physical examination; information on history of coronary heart disease (CHD) and menopausal hormone therapy (MHT) was available, as previously described ( 29 ). Study design CVD was defined as the self
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( 28 ), and in postmenopausal women on combined menopausal hormone therapy who performed better on visuospatial and working memory tasks compared to controls ( 29 , 30 ). Together these findings indicate that ADT may have suppressive effects on
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. Does menopausal hormone therapy (MHT), exercise or a combination of both, improve pain and function in post-menopausal women with greater trochanteric pain syndrome (GTPS)? A randomised controlled trial . BMC Women’s Health 2016 16 32 . ( https
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at a high dose level of 40 mg E4 (HDE4) per day ( 25 ), which is more than double the 15 mg daily dose developed for hormonal oral contraception and menopausal hormone therapy ( 63 , 64 , 65 ). Major differences between both methods are that tE2 is
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menopausal females . Medical Research Archives 2018 6 1663 . 49 Gosset A Pouillès JM & Trémollieres F . Menopausal hormone therapy for the management of osteoporosis . Best Practice and Research. Clinical Endocrinology and Metabolism 2021 35