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Medical Research Council (MRC) Human Nutrition Research, MRC Keneba, Elsie Widdowson Laboratories, Fulbourn Road, Cambridge CB1 9NL, UK
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Medical Research Council (MRC) Human Nutrition Research, MRC Keneba, Elsie Widdowson Laboratories, Fulbourn Road, Cambridge CB1 9NL, UK
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concentration and iron status (2, 13, 14, 15, 16, 17) . As well as being regulated by FGF23, 1,25(OH) 2 D production is controlled by parathyroid hormone (PTH) (18) . PTH is secreted by the parathyroid glands in response to low circulating ionised Ca
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some conflicting findings concerning the impact of obesity on the adipose iron status, as some studies have determined that obesity is associated with iron deficiency and hyperglycemia despite enhanced insulin sensitivity ( 13 ). Conversely, it is well
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cause of haematologic changes in GD, next to the thyroid hormone excess ( 8 ). Hepcidin is a liver-derived protein responsive to Fe plasma concentration and helps maintain proper iron status. It acts mainly through blockage of Fe transporters in the
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Department of Endocrinology, The First Hospital of Lanzhou University, Lanzhou, Gansu, China
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Department of Endocrinology, The First Hospital of Lanzhou University, Lanzhou, Gansu, China
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Department of Endocrinology, The First Hospital of Lanzhou University, Lanzhou, Gansu, China
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-talk among gut microbiota, iron status, and liver fat accumulation, and the mechanism was speculated to be microbiome- and iron-linked metabolomic and transcriptomic signatures involving imbalances in gluconeogenic metabolites, ketone bodies, and cellular
Department of Clinical Medicine, Aalborg University, Aalborg, Denmark
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Department of Geriatrics, Aalborg University Hospital, Aalborg, Denmark
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://doi.org/10.1159/000362597 ) 18 Dewey KG Oaks BM U-shaped curve for risk associated with maternal hemoglobin, iron status, or iron supplementation . American Journal of Clinical Nutrition 2017 106 1694S – 1702S . ( https://doi.org/10.3945/ajcn.117
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results confirmed that the level of hepcidin was decreased under conditions of high glucose stimulation and had a disrupted effect on insulin secretion ( 14 ). In addition, the iron status induced by decreased hepcidin expression and its possible toxic
