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Marianne C Astor, Kristian Løvås, Anette S B Wolff, Bjørn Nedrebø, Eirik Bratland, Jon Steen-Johnsen and Eystein S Husebye

neonatal kidney can also be encountered in infancy. Hypomagnesemia blocks the release of PTH and probably also decreases the sensitivity to circulating PTH in target organs and thereby causes a biochemical picture identical to primary hypoparathyroidism (1

Open access

Maxime Duval, Kalyane Bach-Ngohou, Damien Masson, Camille Guimard, Philippe Le Conte and David Trewick

deficiency was defined as <50 nm/L (20 ng/mL), hypoparathyroidism by a PTH that was inappropriately low (<20 pg/mL) and hypomagnesemia as <0.6 mmol/L ( 6 , 11 , 18 ). Because of the retrospective nature of the study, approval by the local ethics committee

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Tao Yuan, Lanping Jiang, Chen Chen, Xiaoyan Peng, Min Nie, Xuemei Li, Xiaoping Xing, Xuewang Li and Limeng Chen

listed in Table 2 . Recurrent hypokalaemia and hyperkalaemia occurred in all GS patients. Hypomagnesemia was observed in 24 patients. A decreased urinary calcium/creatinine ratio (<0.1 mmol/mmol) was detected in 17 of the 28 patients, and the urinary

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Efstratios Kardalas, Stavroula A Paschou, Panagiotis Anagnostis, Giovanna Muscogiuri, Gerasimos Siasos and Andromachi Vryonidou

hypokalemia. Finally, hypomagnesemia is very important. More than 50% of clinically significant hypokalemia has concomitant magnesium deficiency and is clinically most frequently observed in individuals receiving loop or thiazide diuretic therapy. Concomitant