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A Chinoy Royal Manchester Children’s Hospital, Manchester, UK

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M Skae Royal Manchester Children’s Hospital, Manchester, UK

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A Babiker King Abdullah Specialized Children’s Hospital, Riyadh, Saudi Arabia

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D Kendall Royal Preston Hospital, Preston, UK

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M Z Mughal Royal Manchester Children’s Hospital, Manchester, UK

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R Padidela Royal Manchester Children’s Hospital, Manchester, UK

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therefore characterised by hypocalcaemia and hyperphosphatemia. The standard treatment for HPT is with active vitamin D (calcitriol (1,25(OH 2 )D)) or its analogue (alfacalcidol (ACD; 1-hydroxycholecalciferol)) and ensuring adequate oral Ca intake (through

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0234
Volume/Issue:
Volume 6: Issue 8
Open access
Jeremy Turner Norfolk and Norwich University Hospital, Colney Lane, Norwich, UK

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Neil Gittoes Centre for Endocrinology, Diabetes and Metabolism, University Hospitals Birmingham & University of Birmingham, Birmingham Health Partners, Birmingham, UK

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Peter Selby Department of Medicine, Manchester Royal Infirmary, Manchester, UK

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the Society for Endocrinology Clinical Committee The Society for Endocrinology, 22 Apex Court, Woodlands, Bradley Stoke, Bristol, UK

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Introduction Acute hypocalcaemia can be life threatening, necessitating urgent treatment. In severe cases, intravenous calcium forms the mainstay of initial therapy, but it is essential to ascertain the underlying cause and commence specific

Article Type:
Other
DOI:
https://doi.org/10.1530/EC-16-0056
Volume/Issue:
Volume 5: Issue 5
Open access
Jeremy Turner Norfolk and Norwich University Hospital, Colney Lane, Norwich, UK

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Neil Gittoes Centre for Endocrinology, Diabetes and Metabolism, University Hospitals Birmingham & University of Birmingham, Birmingham Health Partners, Birmingham, UK

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Peter Selby Department of Medicine, Manchester Royal Infirmary, Manchester, UK

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the Society for Endocrinology Clinical Committee4 The Society for Endocrinology, Woodlands, Bradley Stoke, Bristol, UK

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In this addendum to the above paper, the Society for Endocrinology Clinical Committee and the original authors provide additional advice on the dose equivalence of calcium gluconate and calcium chloride. The ‘Severe hypocalcaemia’ section

Article Type:
Addendum
DOI:
https://doi.org/10.1530/EC-16-0056a
Volume/Issue:
Volume 8: Issue 6
Open access
Katherine U Gaynor Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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Irina V Grigorieva Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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Samantha M Mirczuk Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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Sian E Piret Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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Kreepa G Kooblall Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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Mark Stevenson Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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Karine Rizzoti The Francis Crick Institute, London, UK

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Michael R Bowl Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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M Andrew Nesbit Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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Paul T Christie Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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William D Fraser Norwich Medical School, Faculty of Medicine and Health Sciences, University of East Anglia, Norwich, UK

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Tertius Hough MRC Mammalian Genetics Unit, MRC Harwell Institute, Harwell Science and Innovation Campus, Oxfordshire, UK

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Michael P Whyte Washington University in St Louis School of Medicine, Center for Metabolic Bone Disease and Molecular Research, St Louis, Missouri, USA

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Robin Lovell-Badge The Francis Crick Institute, London, UK

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Rajesh V Thakker Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism, Churchill Hospital, Oxford, UK

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Introduction Heritable hypoparathyroidism (HPT) is a genetically heterogeneous disease, characterized biochemically by hypocalcaemia, hyperphosphatemia, low plasma parathyroid hormone (PTH) concentrations and inappropriately normal or high

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-19-0478
Volume/Issue:
Volume 9: Issue 2
Open access
Marie Freel Queen Elizabeth University Hospital, Glasgow, UK

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-specialist clinicians and delay the appropriate treatment of this common and potentially life-threatening condition. Acute hypocalcaemia ( 5 ) and hypercalcaemia ( 6 ): disorders of calcium regulation are the second most common electrolyte disorder requiring endocrine

Article Type:
Editorial
DOI:
https://doi.org/10.1530/EC-16-0068
Volume/Issue:
Volume 5: Issue 5
Open access
Rasmus Reinke Department of Otorhinolaryngology, Head and Neck Surgery, Aarhus University Hospital, Aarhus, Denmark

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Stefano Christian Londero Department of Otorhinolaryngology, Head and Neck Surgery, Aarhus University Hospital, Aarhus, Denmark

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Martin Almquist Department of Surgery, Lund University Hospital, Lund, Sweden

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Lars Rejnmark Department of Endocrinology and Internal Medicine, Aarhus University Hospital, Aarhus, Denmark

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Lars Rolighed Department of Otorhinolaryngology, Head and Neck Surgery, Aarhus University Hospital, Aarhus, Denmark

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calcifications ( 7 ). Finally, hypoPT results in a longer hospital stay ( 8 ) after TT. The frequency of postoperative hypocalcaemia and/or hypoPT after TT varies significantly in the literature. This variation is thought to be at least partly due to

Article Type:
Research Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-23-0198
Volume/Issue:
Volume 12: Issue 9
Open access
Daniel Bell Department of Pharmacy, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Julia Hale Department of Endocrinology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Cara Go Department of Endocrinology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Ben G Challis Department of Endocrinology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Tilak Das Department of Radiology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK

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Brian Fish Department of Head and Neck Surgery, Cambridge University NHS Foundation Trust, Cambridge, UK

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Ruth T Casey Department of Endocrinology, Cambridge University Hospital NHS Foundation Trust, Cambridge, UK
Department of Medical Genetics, Cambridge University, Cambridge, UK

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.42 mmol/L ± 0.20 mmol/L and PO 1.22 mmol/L ± 0.25 mmol/L. There were two cases of post-operative hypocalcaemia amongst this subgroup; one patient taking cinacalcet 90 mg/day (highest dose in our cohort) required i.v. calcium and remained an inpatient for

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-21-0258
Volume/Issue:
Volume 10: Issue 11
Open access
Jan Calissendorff Department of Endocrinology, Metabolism and Diabetes, Karolinska University Hospital, Stockholm, Sweden
Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden

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Henrik Falhammar Department of Endocrinology, Metabolism and Diabetes, Karolinska University Hospital, Stockholm, Sweden
Department of Molecular Medicine and Surgery, Karolinska Institutet, Stockholm, Sweden

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years, respectively, and the LS dose ranged from 225 to 1708 mg. Thyroidectomy was uneventful in all 26 patients who underwent operation. However, 7 patients (27%) developed temporary post-operative hypocalcaemia, treated with alfacalcidol and calcium

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0025
Volume/Issue:
Volume 6: Issue 4
Open access
Jennifer Walsh The Mellanby Centre for Bone Research, The Medical School, The University of Sheffield, Sheffield, UK

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Neil Gittoes Centre for Endocrinology, Diabetes and Metabolism, University Hospitals Birmingham & University of Birmingham, Birmingham Health Partners, Birmingham, UK

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Peter Selby Department of Medicine, Manchester Royal Infirmary, Manchester, UK

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the Society for Endocrinology Clinical Committee The Society for Endocrinology, 22 Apex Court, Woodlands, Bradley Stoke, Bristol, UK

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Pamidronate 30–90 mg (depending on severity of hypercalcaemia) at 20 mg/h OR Ibandronic acid 2–4 mg Give more slowly and consider dose reduction in renal impairment Monitor serum calcium response: will reach nadir at 2–4 days Can cause hypocalcaemia

Article Type:
Other
DOI:
https://doi.org/10.1530/EC-16-0055
Volume/Issue:
Volume 5: Issue 5
Open access
Maxime Duval Department of Medicine, Clinique Jules Verne, Nantes, France

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Kalyane Bach-Ngohou Department of Biology, Laboratory of Clinical Biochemistry, CHU Nantes, Nantes, France

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Damien Masson Department of Biology, Laboratory of Clinical Biochemistry, CHU Nantes, Nantes, France

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Camille Guimard Department of Emergency Medicine, CHU Nantes, Nantes, France

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Philippe Le Conte Department of Emergency Medicine, CHU Nantes, Nantes, France

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David Trewick Department of Medicine, Clinique Jules Verne, Nantes, France
Department of Emergency Medicine, CHU Nantes, Nantes, France

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Turner J Gittoes N Selby P & Society for Endocrinology Clinical Committee . SOCIETY FOR ENDOCRINOLOGY ENDOCRINE EMERGENCY GUIDANCE: Emergency management of acute hypocalcaemia in adult patients . Endocrine Connections 2016 5 7 – 8 . ( https

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0267
Volume/Issue:
Volume 7: Issue 10
Open access