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(AMPK) has attracted considerable attention, due to its central role in governing glucose homeostasis. The activation of AMPK can lead to a series of physiological responses of the glycolipid metabolism by affecting intracellular signal transduction
School of Life Science and Engineering, Southwest Jiaotong University, Chengdu, Sichuan, China
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glycolipid metabolism (e.g. metformin) within 3 months before the study; (ii) pregnancy or luteal menstrual phase, which can also affect glycolipid metabolism and oxidative stress ( 29 , 30 ); (iii) smoking; and/or (iv) fasting glucose (Glu) ≥ 7.0 mmol/L and
Department of Clinical Medicine, Gansu University of Chinese Medicine, Lanzhou, Gansu, China
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). Chemerin, an 18-kDa protein secreted by adipose tissue, plays a key role in adipocyte differentiation and also helps to regulate APN expression and glycolipid metabolism ( 11 ). Several studies have demonstrated that chemerin is directly linked to human
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glucose changes. Therefore, it was inferred that these disorders of glycolipid metabolism were associated with elevated branched amino acids. The body weight of the rats in the HFTD group was significantly higher than that of the control group at the