Center for Clinical Metabolic Research, Gentofte Hospital, University of Copenhagen, Hellerup, Denmark
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Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
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Department of Clinical Physiology and Nuclear Medicine, Bispebjerg and Frederiksberg Hospital, University of Copenhagen, Copenhagen, Denmark
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Department of Clinical Pharmacology, Bispebjerg and Frederiksberg Hospital, University of Copenhagen, Copenhagen, Denmark
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Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
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Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
Steno Diabetes Center Copenhagen, Herlev, Denmark
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Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
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Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
Steno Diabetes Center Copenhagen, Herlev, Denmark
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knowledge, not been directly addressed. Notably, the abovementioned studies did not measure appetite and food intake, and thus, we hypothesized that levothyroxine substitution therapy – despite its REE-increasing effect – may induce mechanisms preserving or
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Medical Microbiology Department, College of Basic Medicine, Qingdao University, Qingdao, China
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Physiology Department, College of Basic Medicine, Qingdao University, Qingdao, China
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the latter has become an increasingly recognized region involved in the control of food intake and energy balance. Abundant direct nerve projections are present between LPBN and hypothalamus or brainstem. Moreover, many feeding relevant peptides
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NIHR/Wellcome Trust Clinical Research Facility, Addenbrookes Hospital, Cambridge, UK
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NIHR/Wellcome Trust Clinical Research Facility, Addenbrookes Hospital, Cambridge, UK
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whether increasing energy expenditure through mild cold exposure is accompanied by an increase in appetite and food intake. Cold exposure is known to increase food and energy intake in a wide range of animal species, e.g., piglets, rats and birds ( 7 , 8
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melanocortin receptors, MC3R and MC4R, with MC4R being the primary receptor mediating the metabolic action of melanocortins ( 28 , 29 ). On the other hand, the orexigenic AgRP and NPY peptides – both produced in AgRP neurons – increase food intake and decrease
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University Clinic of Medicine, Cantonal Hospital Baselland, Liestal, Switzerland
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that modulates this circadian cortisol rhythm. The cortisol peak in response to food intake was first described in the 1970s and has been repeatedly reproduced in normal-weight subjects ( 2 , 3 , 4 , 5 ). In lean populations, cortisol levels rise
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Department of Internal Medicine III, University Hospital Carl Gustav Carus Dresden, Dresden, Germany
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Comprehensive Heart Failure Center, Würzburg, Germany
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, the success of RYGB lies in changes of gut hormone levels relevant for food intake behavior. Elevated levels of glucagon-like peptide 1 (GLP-1), peptide tyrosine tyrosine (PYY), glucose-dependent insulinotropic polypeptide (GIP) and oxyntomodulin were
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Laboratório de Fisiologia Endócrina Doris Rosenthal, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro, Brazil
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found in the literature that is able to ameliorate some cardiovascular parameters, protect against mitochondria damage, decrease insulin levels, food intake, body weight gain, serum triglycerides, increase glucose tolerance and insulin sensitivity ( 18
Pritzker School of Medicine, Chicago, Illinois, USA
University of Chicago, Chicago, Illinois, USA
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Committee on Molecular Pathogenesis and Molecular Medicine, Chicago, Illinois, USA
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University of Chicago, Chicago, Illinois, USA
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< 0.05. In prior studies using a standard laboratory chow diet, TF altered fuel utilization preference and metabolic flexibility while disrupting diurnal rhythms of energy expenditure and food intake ( 21 ). To expand upon these findings
Division of Biomedical Information Analysis, Iwate Tohoku Medical Megabank Organization, Disaster Reconstruction Center, Iwate Medical University, Yahaba, Japan
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Faculty of Health Science, Bukkyo University, Kyoto, Japan
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KNDy system contributes to the reproductive disorder in prenatally undernourished rats. Additionally, body weight (BW), food intake, accumulation of visceral adiposity, and plasma triglyceride levels as an indicator of metabolic status were assessed
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amount of diet and ate freely. The diets of OD and ODE mice were controlled to 70% of the food intake of OC mice ( 24 ). This type of diet control program without affecting the normal nutritional status of the mice prevents mice from fighting manically