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J Chycki, A Zajac, M Michalczyk, A Maszczyk and J Langfort


The present study verified the effect of moderate-to-high-intensity aerobic exercise on the endocrine response profile and adipose tissue in young healthy men with different phenotype characteristics.


Eighteen men were divided into three experimental groups with defined body components and specific physical fitness: Endurance phenotype – EP (n = 6; low body mass; low fat content; aerobic endurance trained), Athletic phenotype – AP (n = 6; high body mass; low fat content, resistance trained), Obesity phenotype – OP (n = 6; high body mass; high fat content, untrained).


The participants performed an progressive exercise protocol on a treadmill (30% VO2max, 50% VO2max, 70% VO2max), separated by 45 s of passive rest for blood collection.


Plasma glucose oxidation increased in relation to exercise intensity, but to a greater extent in the AP group. The free fatty acids’ plasma level decreased with a rise in exercise intensity, but with different kinetics in particular phenotypes. Plasma growth hormone increased after the cessation of exercise and was significantly higher in all groups 45 min into recovery compared to resting values. Plasma insulin decreased during exercise in all groups, but in the OP, the decrease was blunted.


The results indicate that the rate of lipolysis, hormonal and metabolic response to aerobic exercise depends on the individuals’ phenotype. Thus, exercise type, duration and intensity have to be strictly individualized in relation to phenotype in order to reach optimal metabolic benefits.

Open access

Ermina Bach, Niels Møller, Jens Otto L Jørgensen, Mads Buhl and Holger Jon Møller

Soop M Duxbury H Agwunobi AO Gibson JM Hopkins SJ Childs C Cooper RG Maycock P Little RA Carlson GL . Euglycemic hyperinsulinemia augments the cytokine and endocrine responses to endotoxin in humans . American Journal of Physiology: Endocrinology and

Open access

Fiona Broughton Pipkin, Hiten D Mistry, Chandrima Roy, Bernhard Dick, Jason Waugh, Rebecca Chikhi, Lesia O Kurlak and Markus G Mohaupt

programming of postnatal endocrine responses by glucocorticoids . Reproduction 2002 124 459 – 467 . ( doi:10.1530/rep.0.1240459 ). 10 Benediktsson R Lindsay RS Noble J Seckl JR Edwards CR . Glucocorticoid exposure in utero: new model for

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Alexander Tacey, Lewan Parker, Bu B Yeap, John Joseph, Ee M Lim, Andrew Garnham, David L Hare, Tara Brennan-Speranza and Itamar Levinger

. ( ) 10.1210/jc.2010-2729 32 Deuster PA Petrides JS Singh A Chrousos GP Poth M. Endocrine response to high-intensity exercise: dose-dependent effects of dexamethasone . Journal of Clinical Endocrinology and

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Eva O Melin, Jonatan Dereke, Maria Thunander and Magnus Hillman

pathogenesis of DR occurs by alterations in multiple biochemical pathways, which involve several metabolic, inflammatory and endocrine responses ( 1 , 2 , 4 , 6 ). Depression is the most common psychiatric disorder worldwide with great impact not only on

Open access

Antonia Ertelt, Ann-Kristin Barton, Robert R Schmitz and Heidrun Gehlen

period . Journal of Animal Science 2002 80 2695 – 2703 . 62 Cartmill JA Thompson DL Storer WA . Endocrine responses in mares and geldings with high body condition scores grouped by high vs. low resting leptin concentrations . Journal of

Open access

Carolina Inda, Natalia G Armando, Paula A dos Santos Claro and Susana Silberstein

CRH receptor in the brain, whereas CRHR2 expression is more restricted ( 20 ). The role of CRHR2 activation mediating anxiety and depression has been less clear. Crhr2 -knockout mice showed an anxiogenic phenotype, increased endocrine response, and