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Shuqi Li Department of Endocrinology, Zhongshan Hospital, Fudan University, Shanghai, China

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Chenye Shi Department of General Surgery, Zhongshan Hospital, Fudan University, Shanghai, China

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Haifu Wu Department of General Surgery, Zhongshan Hospital, Fudan University, Shanghai, China

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Hongmei Yan Department of Endocrinology, Zhongshan Hospital, Fudan University, Shanghai, China

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Mingfeng Xia Department of Endocrinology, Zhongshan Hospital, Fudan University, Shanghai, China

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Heng Jiao Department of General Surgery, Zhongshan Hospital, Fudan University, Shanghai, China

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Yang He Department of Endocrinology, Zhongshan Hospital, Fudan University, Shanghai, China

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Ming Zhong Department of Critical Care Medicine, Zhongshan Hospital, Fudan University, Shanghai, China

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Wenhui Lou Department of General Surgery, Zhongshan Hospital, Fudan University, Shanghai, China

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Xin Gao Department of Endocrinology, Zhongshan Hospital, Fudan University, Shanghai, China

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Hua Bian Department of Endocrinology, Zhongshan Hospital, Fudan University, Shanghai, China

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Xinxia Chang Department of Endocrinology, Zhongshan Hospital, Fudan University, Shanghai, China

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, the enrichment of glycerolipid metabolism, fructose and mannose degradation, and gluconeogenesis in the late ascending type (cluster 3) suggested that LSG altered carbohydrate and lipid metabolism. We observed an increase in glyceraldehyde (a key

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M Guftar Shaikh Department of Paediatric Endocrinology, Royal Hospital for Children, Glasgow, UK
Developmental Endocrinology Research Group, University of Glasgow, Glasgow, UK

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Timothy G Barrett Department of Endocrinology, Birmingham Womens and Children’s Hospital, Birmingham, UK
Institute of Cancer and Genomic Sciences, University of Birmingham, Birmingham, UK

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Nicola Bridges Department of Paediatric Endocrinology, Chelsea and Westminster Hospital, London, UK

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Robin Chung Research Working Group, Prader-Willi Syndrome Association, Northampton, UK

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Evelien F Gevers Department of Paediatric Endocrinology, Barts Health NHS Trust, Royal London Hospital, London, UK
Centre for Endocrinology, William Harvey Research Institute, Barts and The London Medical School, Queen Mary University of London, London, UK

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Anthony P Goldstone PsychoNeuroEndocrinologyResearch Group, Division of Psychiatry, Department of Brain Sciences, Faculty of Medicine, Imperial College London, London, UK
Department of Endocrinology, Imperial College Healthcare NHS Trust, Hammersmith Hospital, London, UK

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Anthony Holland Department of Psychiatry, University of Cambridge, Cambridge, UK

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Shankar Kanumakala Royal Alexandra Children’s Hospital, Brighton, UK

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Ruth Krone Department of Endocrinology, Birmingham Womens and Children’s Hospital, Birmingham, UK

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Andreas Kyriakou Department of Paediatric Endocrinology, Royal Hospital for Children, Glasgow, UK
Department of Paediatric Endocrinology, Makarios Children's Hospital, Nicosia, Cyprus

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E Anne Livesey Royal Alexandra Children’s Hospital, Brighton, UK
Sussex Community NHS Trust, Brighton, UK

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Angela K Lucas-Herald Department of Paediatric Endocrinology, Royal Hospital for Children, Glasgow, UK
Developmental Endocrinology Research Group, University of Glasgow, Glasgow, UK

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Christina Meade CHI at Tallaght University Hospital, Dublin, Republic of Ireland

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Susan Passmore Prader-Willi Syndrome Association, Northampton, UK

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Edna Roche CHI at Tallaght University Hospital, Dublin, Republic of Ireland
The University of Dublin, Trinity College Dublin, Dublin, Republic of Ireland

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Chris Smith Royal Alexandra Children’s Hospital, Brighton, UK

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Sarita Soni Learning Disability Psychiatry, NHS Greater Glasgow and Clyde, Glasgow, UK

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excess food intake, especially during adolescence, remains vital. Control of the food environment to reduce access to, and hence intake of, excess food, especially carbohydrates, is also vital in the management of T2DM. Targeting unrecognised food intake

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