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Dario de Biase, Federica Torricelli, Moira Ragazzi, Benedetta Donati, Elisabetta Kuhn, Michela Visani, Giorgia Acquaviva, Annalisa Pession, Giovanni Tallini, Simonetta Piana and Alessia Ciarrocchi

elongation and unleashed cell proliferation capacity. Recently, increased frequency of mutations within the MED12 gene was reported in a large cohort of fatal non-anaplastic thyroid cancers ( 15 ). This protein is a component of the mediator complex

Open access

Ashley N Reeb, Andrea Ziegler and Reigh-Yi Lin

represent a gold standard for cancer research due to their superior xenografting capability ( 17 , 18 ). We have previously used the orthotopic and tail vein injection models in NSG mice to study the anaplastic thyroid cancer cell (ATC) lines THJ-11T and

Open access

Tiemo S Gerber, Arno Schad, Nils Hartmann, Erik Springer, Ulrich Zechner and Thomas J Musholt

showed a KIT mutation ( 37 ). In our study, we detected four KIT variants. This discrepancy may indicate a slowing effect on cancer growth that is lost during tumour dedifferentiation to anaplastic thyroid cancer, which may be lost in favour of a more

Open access

Nassim Ghaffari-Tabrizi-Wizsy, Christina Angelika Passegger, Laura Nebel, Fabian Krismer, Gudrun Herzer-Schneidhofer, Gert Schwach and Roswitha Pfragner

Searle K Lowerison M Ruicci K Yoo J Fung K MacNeil D Lacefield C , et al . Lestaurtinib is a potent inhibitor of anaplastic thyroid cancer cell line models . PLoS One 2018 13 e0207152 . ( ) 10

Open access

Anello Marcello Poma, Riccardo Giannini, Paolo Piaggi, Clara Ugolini, Gabriele Materazzi, Paolo Miccoli, Paolo Vitti and Fulvio Basolo

( 22 , 42 , 51 ). Moreover, the forced expression of TFF3 in transfected anaplastic thyroid cancer cells restored an epithelial-like cell morphology and the expression of differentiation markers of follicular thyroid cells ( 35 ). SDPR is a

Open access

Huy Gia Vuong, Uyen N P Duong, Ahmed M A Altibi, Hanh T T Ngo, Thong Quang Pham, Hung Minh Tran, Greta Gandolfi and Lewis Hassell

Shah RH Dogan S Ricarte-Filho JC Krishnamoorthy GP Xu B , et al . Genomic and transcriptomic hallmarks of poorly differentiated and anaplastic thyroid cancers . Journal of Clinical Investigation 2016 126 1052 – 1066 . ( doi:10

Open access

Joanna Klubo-Gwiezdzinska, John Costello Jr, Kirk Jensen, Aneeta Patel, Rok Tkavc, Douglas Van Nostrand, Kenneth D Burman, Leonard Wartofsky and Vasyl Vasko

University of the Health Sciences. Human thyroid cancer cell lines derived from follicular thyroid cancer (FTC133), papillary thyroid cancer (TPC1 and BCPAP) and anaplastic thyroid cancer (C643) cells, as well as NIH3T3 mouse embryonic fibroblast cells were

Open access

Wei Li, Qing Huang, Danyang Sun, Guizhi Zhang and Jian Tan

tissue had only one patient ( Fig. 1A ). Then, we performed a comprehensive analysis of RDM1 expression profiles in various types of thyroid cancer vs normal thyroid tissues (a total of 105 clinical samples including papillary, anaplastic thyroid cancer

Open access

Barbora Pekova, Sarka Dvorakova, Vlasta Sykorova, Gabriela Vacinova, Eliska Vaclavikova, Jitka Moravcova, Rami Katra, Petr Vlcek, Pavla Sykorova, Daniela Kodetova, Josef Vcelak and Bela Bendlova

% or more of pediatric thyroid cancer ( 4 ). Medullary (MTC) and follicular thyroid cancer (FTC) are relatively rare. MTC is mostly familial than sporadic in children and adolescents. Poorly differentiated cancer and anaplastic thyroid cancer (ATC) have