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Chiara Mele, Maria Teresa Samà, Alessandro Angelo Bisoffi, Marina Caputo, Valentina Bullara, Stefania Mai, Gillian Elisabeth Walker, Flavia Prodam, Paolo Marzullo, Gianluca Aimaretti and Loredana Pagano

). Although the pathways linking obesity and IR to thyroid cancer remain largely unknown, a potential role for insulin, growth hormone (GH) secretagogues and adipokines has been postulated ( 10 ). Insulin not only regulates cell metabolism but also stimulates

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Ningning Gong, Cuixia Gao, Xuedi Chen, Yu Wang and Limin Tian

atherosclerosis (AS) was reported in a large population-based study ( 2 , 3 ). Multiple lines of evidence have shown that adipokines ( 4 ), endothelial dysfunction ( 5 ) and dyslipidemia ( 6 ) play a central role in the development of AS, but the relationship

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Katarzyna Wyskida, Grzegorz Franik, Tomasz Wikarek, Aleksander Owczarek, Alham Delroba, Jerzy Chudek, Jerzy Sikora and Magdalena Olszanecka-Glinianowicz

Introduction It has been shown that the levels of different adipokines, such as leptin, adiponectin, resistin, vaspin, omentin-1 and RBP4 are higher in women than those in men ( 1 , 2 , 3 , 4 , 5 , 6 ), and that estrogens stimulate

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Shilpa Lingaiah, Laure Morin-Papunen, Terhi Piltonen, Inger Sundström-Poromaa, Elisabet Stener-Victorin and Juha S Tapanainen

pathogenesis of PCOS ( 5 , 6 , 7 ), it is not clear whether they have a direct association with PCOS. Adipose tissue acts as an endocrine organ secreting various adipokines including retinol-binding protein 4 (RBP4), which is mainly synthesised by hepatocytes

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Huguette S Brink, Aart Jan van der Lely and Joke van der Linden

( 12 , 13 ). Hence, there is need to predict and detect GD earlier in pregnancy in order to limit the exposure to impaired glucose metabolism. Investigating the role of adipokines associated with the pathophysiology of GD has gained interest ( 14 , 15

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Jay Toulany, Sebastian D Parlee, Christopher J Sinal, Kathryn Slayter, Shelly McNeil and Kerry B Goralski

Introduction After characterisation as an adipokine by our group in 2007, research concerning chemerin and its relationship to obesity has risen steadily. Chemerin is secreted as an inactive precursor prochemerin, which undergoes processing at

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Ningning Gong, Cuixia Gao, Xuedi Chen, Yu Wang and Limin Tian

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Lena-Maria Levin, Henry Völzke, Markus M Lerch, Jens-Peter Kühn, Matthias Nauck, Nele Friedrich and Stephanie Zylla

on the role of adipose tissue as an endocrine organ that produces a multitude of metabolically active hormones, so-called adipokines, which are known to be involved in the regulation of glucose and lipid metabolism, food intake and inflammation ( 4

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Fabian Eichelmann, Cornelia Weikert, Romina di Giuseppe, Ronald Biemann, Berend Isermann, Matthias B Schulze, Heiner Boeing and Krasimira Aleksandrova

). Ten years later (in 2007) chemerin was re-discovered as a novel adipokine regulating adipogenesis and genes critical in glucose and lipid metabolism of adipocytes ( 3 ). Subsequent research characterized additional roles of chemerin in diverse

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Nese Cinar and Alper Gurlek

visfatin is still unclear. Changes in adipokine secretion with thyroid dysfunction may represent adaptive mechanisms to the decrease or increase in basal energy expenditure and in energy substrate requirements in thyroid dysfunction. Cytokine network