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Chun-feng Lu, Wang-shu Liu, Xiao-qin Ge, Feng Xu, Jian-bin Su, Xue-qin Wang, and Yan Wang

, further understanding of the mechanism of immune system dysfunction in GD pathogenesis and exploring new therapeutic targets are of great importance for improving the prognosis of GD patients. Adenosine deaminase (ADA) is a polymorphic enzyme which is

Open access

Chun-feng Lu, Xiao-qin Ge, Yan Wang, Jian-bin Su, Xue-qin Wang, Dong-mei Zhang, Feng Xu, Wang-shu Liu, and Min Su

in patients with T2D is as high as 30% ( 7 ). Therefore, it is of great clinical significance to fully assess the risk factors for prolonged QTc interval in T2D patients. Adenosine deaminase (ADA), abundantly expressed in various cells and tissues

Open access

Chun-feng Lu, Wang-shu Liu, Xiao-qin Ge, and Feng Xu

. Adenosine deaminase (ADA), a polymorphic enzyme, is expressed in all human tissues and plays an important role in regulating adenosine concentration by catalyzing the irreversible deamination of adenosine to inosine ( 7 ). Adenosine is an important paracrine

Open access

Kunal Thakkar, Swati Ramteke-Jadhav, Rajeev Kasaliwal, Saba Samad Memon, Virendra Patil, Puja Thadani, Nilesh Lomte, Shilpa Sankhe, Atul Goel, Sridhar Epari, Naina Goel, Anurag Lila, Nalini S Shah, and Tushar Bandgar

was diagnosed based on characteristic findings on cerebrospinal fluid analysis (lymphocytic pleocytosis with high adenosine deaminase levels) and therapeutic response to anti-tubercular antibiotics; 2) case 5, patient with aneurysm of internal carotid