International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Rigshospitalet, University of Copenhagen, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Rigshospitalet, University of Copenhagen, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Rigshospitalet, University of Copenhagen, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Rigshospitalet, University of Copenhagen, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Rigshospitalet, University of Copenhagen, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Rigshospitalet, University of Copenhagen, Copenhagen, Denmark
Department of Clinical Medicine, University of Copenhagen, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Rigshospitalet, University of Copenhagen, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Rigshospitalet, University of Copenhagen, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Rigshospitalet, University of Copenhagen, Copenhagen, Denmark
Department of Clinical Medicine, University of Copenhagen, Copenhagen, Denmark
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Introduction Luteinizing hormone (LH) and follicle-stimulating hormone (FSH), both produced in the gonadotropic cells of the anterior pituitary gland, have recently been described as an excellent marker of sex when combined in the LH/FSH ratio
School of Life Science and Engineering, Southwest Jiaotong University, Chengdu, Sichuan, China
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.001 LH (IU/L) 4.80 (3.30, 7.20) 10.80 (6.20, 16.00) <0.001 <0.001 FSH (IU/L) 6.56 ± 2.62 6.01 ± 2.12 <0.001 0.027 LH/FSH 0.97 (0.65, 1.46) 1.96 (1.24, 2.82) <0.001 <0.001 Metabolic profile b Fasting
Department of Pediatrics, Navarra Hospital Complex, Pamplona, Spain
Navarra Institute for Health Research (IdisNA), Pamplona, Spain
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Navarra Institute for Health Research (IdisNA), Pamplona, Spain
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Navarra Institute for Health Research (IdisNA), Pamplona, Spain
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sensitive immunochemiluminescence assays (Immulite 2500) with a sensitivity of <0.1 U/L for LH and FSH. Intra-assay coefficients of variation at 0.3 IU/L for LH were 3.5% and FSH 5%. Baseline LH/FSH ratio and LH/FSH ratio post leuprorelin administration were
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Department of Gynecology and Obstetrics, Division of Genetic Epidemiology, Vitateq Biotechnology GmbH, University of Duisburg-Essen, D-45122 Essen, Germany
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acid compositional analysis. Within-run and total coefficient of variation (CV) values were 3.3 and 6.2%, respectively, at a mean concentration of 73 mg/l (15) . Automated chemiluminescence immunoassay systems were used for the determination of LH, FSH
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(blood sampled immediately prior to the next injection) together with serum LH, FSH, and SHBG in the same sample. Individual dose titration was performed by evaluating at each visit whether the treatment had maintained symptomatic benefit according to the
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, Acne score, LH, LH/FSH, testosterone, E2 and DHEA in NG increased significantly ( P < 0.05). Compared with OC, Hirsutism score, LH/FSH and testosterone in OG increased significantly ( P < 0.05), while FSH in OG decreased significantly ( P < 0
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%) Chiasmal contact 36/65 (55%) Visual deficits 8/65 (12%) Endocrine evaluation Any pituitary deficit 29/65 (44%) LH/FSH deficit 27/65 (41%) TSH deficit 19/65 (29%) ACTH deficit 10/65 (15%) PRL elevation
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follicles with 17-OH-progesterone in aforementioned study, which was revealed in the present study ( 21 ). In conclusion, the present study was conducted to evaluate the levels of LH, FSH and androgenic hormones free testosterone, 17-OH-progesterone and
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blood specimens without anticoagulant were centrifuged at 4244 g for 10 min, and the resultant serum specimens were aliquoted and stored at –20°C. The serum LH, FSH, and estradiol (E2) levels were measured by chemiluminescence immunoassays (Mindray, CL
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Radiotherapy Related Research, The Christie NHS Foundation Trust, Manchester, UK
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Radiotherapy Related Research, The Christie NHS Foundation Trust, Manchester, UK
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deficiency in 6.9%. Luteinizing hormone and follicle-stimulating hormone (LH/FSH) deficiency was found in only 4.1% of the children following RT. In terms of dose-response, 78.8% of those who developed a growth hormone deficiency received a dose ≥ 40 Gy to