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Bushra Shahida Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden

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Tereza Planck Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden

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Tania Singh Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden

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Peter Åsman Department of Clinical Sciences Malmö, Ophthalmology, Lund University, Malmö, Sweden
Department of Ophthalmology, Skåne University Hospital, Malmö, Sweden

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Mikael Lantz Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden

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, others assume the role of regulatory T lymphocytes, capable of mitigating immune reactivity ( 2 ). The autoimmune response observed in GD originates from the presence of circulating immunoglobulin G (IgG) antibodies targeting the thyroid

Article Type:
Research Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-23-0374
Volume/Issue:
Volume 13: Issue 6
Open access
Elizabeth Yan Zhang Department of Pharmacology, Department of Biology, Toxicology and Therapeutics, School of Medicine, University of Kansas Medical Center, Room 4061 of KLSIC Building, 2146 West 39th Street, Kansas City, Kansas 66160, USA

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Bao-Ting Zhu Department of Pharmacology, Department of Biology, Toxicology and Therapeutics, School of Medicine, University of Kansas Medical Center, Room 4061 of KLSIC Building, 2146 West 39th Street, Kansas City, Kansas 66160, USA
Department of Pharmacology, Department of Biology, Toxicology and Therapeutics, School of Medicine, University of Kansas Medical Center, Room 4061 of KLSIC Building, 2146 West 39th Street, Kansas City, Kansas 66160, USA

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-induced skin hypersensitivity by E 3 (as presented in Fig. 1 B, C, D, and E) was found to be accompanied by marked reductions (≥50%) in DNCB-specific serum Abs (e.g., specific IgA, IgG, IgG1, IgG2a, and IgG2b) compared with animals treated with DNCB alone

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-14-0080
Volume/Issue:
Volume 3: Issue 4
Open access
Nancy J Olsen Division of Rheumatology, Division of Endocrinology, Diabetes, and Metabolism, College of Medicine, Milton S Hershey Medical Center, The Pennsylvania State University, Mail Code H044, 500 University Drive, Hershey, Pennsylvania 17033-0850, USA

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Ann L Benko Division of Rheumatology, Division of Endocrinology, Diabetes, and Metabolism, College of Medicine, Milton S Hershey Medical Center, The Pennsylvania State University, Mail Code H044, 500 University Drive, Hershey, Pennsylvania 17033-0850, USA

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William J Kovacs Division of Rheumatology, Division of Endocrinology, Diabetes, and Metabolism, College of Medicine, Milton S Hershey Medical Center, The Pennsylvania State University, Mail Code H044, 500 University Drive, Hershey, Pennsylvania 17033-0850, USA

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body composition (20) , in HDL levels (21) , and in response to treatment with exogenous androgens (22) . In a previous study, we found that longer AR CAG repeats were associated with more exuberant expression of IgG autoantibodies in males with

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-14-0039
Volume/Issue:
Volume 3: Issue 2
Open access
Raja Padidela Royal Manchester Children’s Hospital and Faculty of Biology, Medicine and Health, University of Manchester, Manchester, UK

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Moira S Cheung Evelina London Children’s Hospital, London, UK

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Vrinda Saraff Birmingham Women’s and Children’s Hospital, Birmingham, UK

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Poonam Dharmaraj Alder Hey Children’s NHS Foundation Trust, Liverpool, UK

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left untreated, tertiary hyperparathyroidism. Active vitamin D analogues can prevent hyperparathyroidism but increase the risk of nephrocalcinosis. Burosumab is a recombinant human IgG1 MAB that binds to and inhibits the activity of fibroblast growth

Article Type:
Research Article
Collections:
Paediatric Endocrinology Collection
DOI:
https://doi.org/10.1530/EC-20-0291
Volume/Issue:
Volume 9: Issue 10
Open access
Stavroula Stavrou Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Michael Gratz Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Eileen Tremmel Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Christina Kuhn Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Simone Hofmann Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Helene Heidegger Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Mina Peryanova Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Kerstin Hermelink Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Stefan Hutter Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Bettina Toth Department of Gynaecological Endocrinology and Reproductive Medicine, Medical University Innsbruck, Innsbruck, Austria

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Doris Mayr Department of Pathology, Hospital of the LMU, Munich, Germany

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Sven Mahner Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Udo Jeschke Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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Aurelia Vattai Department of Gynecology and Obstetrics, Hospital of the LMU, Munich, Germany

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added on third trimester placenta) (Negative Control for Super Sensitive Rabbit Antibodies, Rabbit IgG, BioGenex, Fremont, USA) were included in each experiment (Supplementary Fig. 1, see section on supplementary data given at the end of this article

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-17-0272
Volume/Issue:
Volume 7: Issue 2
Open access
Michael J Gratz Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Stavroula Stavrou Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Christina Kuhn Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Simone Hofmann Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Kerstin Hermelink Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Helene Heidegger Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Stefan Hutter Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Doris Mayr Department of Pathology, University Hospital, LMU Munich, Munich, Germany

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Sven Mahner Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Udo Jeschke Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Aurelia Vattai Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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on the third trimester placenta) (Negative Control for Super Sensitive Rabbit Antibodies, Rabbit IgG; Biogenics, Fremont, CA, USA) were included in each experiment (Supplementary Fig. 1, see section on supplementary data given at the end of this

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0126
Volume/Issue:
Volume 7: Issue 5
Open access
Chenmin Wei Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
Department of Endocrinology, The Affiliated Jiangning Hospital of Nanjing Medical University, Nanjing, Jiangsu, China

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Zichen Zhang Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China

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Qi Fu Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China

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Yunqiang He Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China

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Tao Yang Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China

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Min Sun Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China

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-rabbit IgG (H+L), Cat# 34206ES60) and Alexa Fluor 594 (donkey anti-mouse IgG (H+L), Cat# 34112ES60) were purchased from Yeasen Biotech Co (Shanghai, China). Cell culture, FFA preparation, and cell treatment MIN6 cells (passage 20–45) were routinely

Article Type:
Research Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-22-0221
Volume/Issue:
Volume 12: Issue 1
Open access
Yao Ye Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Aurelia Vattai Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Nina Ditsch Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Christina Kuhn Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Martina Rahmeh Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Sven Mahner Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Myriam Ripphahn Walter Brendel Centre of Experimental Medicine, Department of Cardiovascular Physiology and Pathophysiology, Biomedical Center, Ludwig-Maximilians University of Munich, Munich, Germany

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Roland Immler Walter Brendel Centre of Experimental Medicine, Department of Cardiovascular Physiology and Pathophysiology, Biomedical Center, Ludwig-Maximilians University of Munich, Munich, Germany

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Markus Sperandio Walter Brendel Centre of Experimental Medicine, Department of Cardiovascular Physiology and Pathophysiology, Biomedical Center, Ludwig-Maximilians University of Munich, Munich, Germany

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Udo Jeschke Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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Viktoria von Schönfeldt Department of Obstetrics and Gynecology, University Hospital, LMU Munich, Munich, Germany

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(Reagent 1, Zytochem-Plus HRP-Polymer-Kit (mouse/rabbit)) for 20 min to avoid non-specific binding of the primary antibodies. The primary antibodies used for the experiment were anti-COX rabbit IgG polyclonal antibody (Sigma, SAB4502491, 1:400 dilution

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0106
Volume/Issue:
Volume 7: Issue 5
Open access
Kazunori Morohoshi Department of Life Sciences, Laboratory of Functional Anatomy, Faculty of Agriculture, Meiji University, Kawasaki, Japan

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Ryo Mochinaga Department of Life Sciences, Laboratory of Functional Anatomy, Faculty of Agriculture, Meiji University, Kawasaki, Japan

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Tsukasa Watanabe Department of Life Sciences, Laboratory of Functional Anatomy, Faculty of Agriculture, Meiji University, Kawasaki, Japan

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Ryojun Nakajima Department of Life Sciences, Laboratory of Functional Anatomy, Faculty of Agriculture, Meiji University, Kawasaki, Japan

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Toshio Harigaya Department of Life Sciences, Laboratory of Functional Anatomy, Faculty of Agriculture, Meiji University, Kawasaki, Japan

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added to proteinG agarose, and the beads–antibody mixture was rotated overnight at 4°C. Mouse IgG was added to proteinG agarose instead of integrin alpha5 beta1 monoclonal antibody as negative control. The antibody coupled beads were blocked by the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/EC-18-0116
Volume/Issue:
Volume 7: Issue 5
Open access
Jaafar Jaafar Division of Endocrinology and Diabetology, Geneva University Hospitals and Faculty of Medicine, Geneva, Switzerland

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Eugenio Fernandez Department of Oncology, Geneva University Hospitals and Faculty of Medicine, Geneva, Switzerland

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Heba Alwan Division of Endocrinology and Diabetology, Geneva University Hospitals and Faculty of Medicine, Geneva, Switzerland

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Jacques Philippe Division of Endocrinology and Diabetology, Geneva University Hospitals and Faculty of Medicine, Geneva, Switzerland

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differentiation ( 3 ). The same team, based on the development of a characteristic lupus-like proliferative arthritis and glomerulonephritis with predominant IgG3 deposition in PD-1-deficient (PD-1−/−) mice, suggested that PD-1 is involved in the maintenance of

Article Type:
Review Article
DOI:
https://doi.org/10.1530/EC-18-0079
Volume/Issue:
Volume 7: Issue 5
Open access