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was an observational study with consecutive enrollment. Females with newly diagnosed overt autoimmune hypo- or hyperthyroidism (Hashimoto’s disease or Graves’ disease, respectively) were included. Inclusion criteria for hypothyroid group consisted of
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inflammation can lead to an increase in extracellular fluid levels and water retention ( 3 ). This effect is also observed in patients with Hashimoto’s disease, where thyroid inflammation results in an increase in thyroid tissue water content ( 4 ). Research
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genes for certain enzymes of pyrimidine and purine salvage pathway in peripheral blood leukocytes collected from patients with Graves’ or Hashimoto’s disease . Journal of Cellular Biochemistry 2003 89 550 – 555 . ( https://doi.org/10.1002/jcb.10533
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(TRABs >1.0 IU/L). We excluded patients with neonatal hyperthyroidism as well as patients with negative TRABs as seen in Hashimoto’s disease with hyperthyroidism. The results as well as the outcomes in patient numbers such as primary treatment
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Department of Regional Health Research, University of Southern Denmark, Odense M, Denmark
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patients with Graves’ and Hashimoto’s diseases differ from that in the general population . European Journal of Endocrinology 2007 156 631 – 636 . ( https://doi.org/10.1530/EJE-07-0015 ) 10.1530/EJE-07-0015 17535862 19 Hamilton A Newby PR Carr
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patients with thyrotoxicosis, only three had a complete suppression of TSH (below 0.1 IU/mL), which again raises suspicion that the main disorder is NTIS/drugs. The most common disease in the thyroid gland that causes dysfunction is Hashimoto’s disease
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. ( https://doi.org/10.1007/BF03343925 ) 10 Torimoto K Okada Y Nakayamada S Kubo S Tanaka Y . Anti-PD-1 antibody therapy induces Hashimoto’s disease with an increase in peripheral blood follicular helper T cells . Thyroid 2017 27 1335 – 1336
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) found that PAs had a stiffer structure than parathyroid hyperplasia lesions and reactive lymph nodes. Moreover, the study by Altinbas & Yagci ( 4 ) showed that parathyroid lesions, lymph nodes of patients with Hashimoto’s disease and jugular lymph nodes
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Hashimoto’s disease and in the proportion of Th17 cells in intractable Graves’ disease . Thyroid 2009 19 495 – 501 . ( doi:10.1089/thy.2008.0423 ) 19415997 10.1089/thy.2008.0423 38 Arribas J Esselens C. ADAM17 as a therapeutic target in
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, and was treated with levothyroxine (50–125 µg daily). Two other patients were also on levothyroxine treatment, one because of Hashimoto’s disease and one because of hypothyroidism of unknown cause. The median nodule volume at baseline was 12.0 mL