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neurological impact of subclinical hypoglycaemic episodes that could have happened before the clinical presentation of the HI. Conclusion Children with HI/HA syndrome due to an activating mutation in the GLUD1 gene are prone to neurodevelopmental
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Internal Medicine, S. Maria delle Croci Hospital, AUSL Romagna, Ravenna, Italy
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Istituto Auxologico Italiano, IRCCS, Obesity Unit - Laboratory of Nutrition and Obesity Research, Department of Endocrine and Metabolic Diseases, Milan, Italy
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insulin secretory defects may play a negative role. The concept that subclinical insulin secretory defects may affect linear growth independently from hyperglycemia has been suggested by a small-sized study in CF ( 20 ), but further evidence arises from
Department of Child and Adolescent Medicine, Section of Pediatric Cardiology, University Hospital Jena, Am Klinikum, Jena, Germany
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Department of Women and Child Health, Hospital for Children and Adolescents and Center for Pediatric Research (CPL), University of Leipzig, Liebigstrasse, Leipzig, Germany
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Department of Women and Child Health, Hospital for Children and Adolescents and Center for Pediatric Research (CPL), University of Leipzig, Liebigstrasse, Leipzig, Germany
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. ( https://doi.org/10.7326/0003-4819-139-8-200310210-00007 ) 16 Eichner M Wallaschofski H Schminke U Völzke H Dörr M Felix SB Nauck M & Friedrich N . Relation of IGF-I with subclinical cardiovascular markers including intima