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University Clinic of Medicine, Cantonal Hospital Baselland, Liestal, Switzerland
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that modulates this circadian cortisol rhythm. The cortisol peak in response to food intake was first described in the 1970s and has been repeatedly reproduced in normal-weight subjects ( 2 , 3 , 4 , 5 ). In lean populations, cortisol levels rise
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investigate whether incretins themselves are responsible for these post-RYGB endocrine adaptations. We cannot exclude that the HPA-axis activation was related to early dumping syndrome and its hemodynamic implications that activate the stress response, since
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vascular health. Patients with overt DM are usually associated with some degree of endothelial cell damage and stress oxidation whereas GDM usually develops in pregnant women with healthy vascular endothelial cells. It develops in response to human
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offspring ( 1 , 3 ). The pathogenesis of GDM is still unclear, but there is growing evidence that genetic variants, advanced maternal age, pre-pregnancy BMI, oxidative stress, chronic inflammation, dyslipidemia, unbalanced hormone secretion, and/or β
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ferroptosis inhibitor Fer-1 could induce PA-caused hepatocyte death ( 66 ). Heat shock factor 1 (HSF1) is a stress-responsive transcription factor that plays a dominant role in the heat shock response ( 67 ). HSF1 is induced in various cardiovascular diseases
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evaluated. Furthermore, potential dose-dependent effects of ALA supplementation on cardiometabolic risk factors in patients with type 2 diabetes have not been evaluated. Dose–response meta-analysis of differences in means is a new statistical approach that
Department of Clinical Research, University of Southern Denmark, Odense, Denmark
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Department of Sports Science and Clinical Biomechanics, University of Southern Denmark, Odense, Denmark
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Department of Clinical Research, University of Southern Denmark, Odense, Denmark
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between insulin secretion and insulin sensitivity is hyperbolic ( 17 , 18 ), and, therefore, when insulin sensitivity is improved in response to exercise training, less insulin secretion is needed. It is, therefore, preferable that insulin sensitivity and
Weihai Institute for Bionics, Jilin University, Weihai, China
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College of Biological and Agricultural Engineering, Jilin University, Changchun, China
Key Laboratory of Bionic Engineering, Ministry of Education, Jilin University, Changchun, China
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the baseline level before the next set of sampling. Table 1 Summary of the sensor array. Number The gas sensor The response characteristics S1 TGS2612 Butane, methane, propane. S2 TGS2611 Methane, natural
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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-β (TGF-β) family, has been identified as a stress-responsive cytokine and metabolic modulator whose expression is markedly elevated in response to stress conditions, aging, obesity, and diabetes ( 3 , 6 , 7 , 8 , 9 , 10 , 11 , 12 , 13 ). In
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reactions ( 11 ). NLRP3 orchestrates the cleavage and maturation of IL-1β and IL-18, setting off an intricate network of cellular responses that culminate in both local and systemic inflammation ( 12 ). In the context of obesity, NLRP3 emerges as a potential