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Chenyu Jiang
Chenyu Jiang
Department of Obstetrics and Gynecology, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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Mi Zhou
Mi Zhou
Department of Obstetrics and Gynecology, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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Huai Bai
Huai Bai
Laboratory of Genetic Disease and Perinatal Medicine, Key Laboratory of Birth Defects and Related Diseases of Women and Children, Ministry of Education, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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Meng Chen
Meng Chen
Department of Obstetrics and Gynecology, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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Chunyi Yang
Chunyi Yang
Department of Obstetrics and Gynecology, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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Kaifeng Hu
Kaifeng Hu
Laboratory of Genetic Disease and Perinatal Medicine, Key Laboratory of Birth Defects and Related Diseases of Women and Children, Ministry of Education, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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Yujie Wu
Yujie Wu
Department of Obstetrics and Gynecology, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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Qingqing Liu
Qingqing Liu
Laboratory of Genetic Disease and Perinatal Medicine, Key Laboratory of Birth Defects and Related Diseases of Women and Children, Ministry of Education, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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Yangyu Zhao
Yangyu Zhao
Department of Obstetrics and Gynecology, Peking University Third Hospital, Beijing, China
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Xinghui Liu
Xinghui Liu
Department of Obstetrics and Gynecology, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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Ping Fan
Ping Fan
Laboratory of Genetic Disease and Perinatal Medicine, Key Laboratory of Birth Defects and Related Diseases of Women and Children, Ministry of Education, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China
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offspring ( 1 , 3 ). The pathogenesis of GDM is still unclear, but there is growing evidence that genetic variants, advanced maternal age, pre-pregnancy BMI, oxidative stress, chronic inflammation, dyslipidemia, unbalanced hormone secretion, and/or β
Panisa Hantrakun
Panisa Hantrakun
Department of Obstetrics and Gynecology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
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Rattanaporn Sekararithi
Rattanaporn Sekararithi
Department of Obstetrics and Gynecology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
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Thidarat Jaiwongkam
Thidarat Jaiwongkam
Cardiac Electrophysiology Research and Training Center (CERT), Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
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Sirinart Kumfu
Sirinart Kumfu
Cardiac Electrophysiology Research and Training Center (CERT), Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
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Chatree Chai-adisaksopha
Chatree Chai-adisaksopha
Division of Hematology, Department of Internal Medicine, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
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Nipon Chattipakorn
Nipon Chattipakorn
Cardiac Electrophysiology Research and Training Center (CERT), Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
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Theera Tongsong
Theera Tongsong
Department of Obstetrics and Gynecology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
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Phudit Jatavan
Phudit Jatavan
Department of Obstetrics and Gynecology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
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Highlight Metformin significantly reduces fetal weight in women with GDM, theoretically preventing macrosomia. Metformin does not improve oxidative stress and platelet dysfunction in women with GDM. Introduction
Svjatoslavs Kistkins
Svjatoslavs Kistkins
Pauls Stradiņš Clinical University Hospital, Riga, Latvia
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Othmar Moser
Othmar Moser
Division of Exercise Physiology and Metabolism, Institute of Sport Science, University of Bayreuth, Bayreuth, Germany
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Vitālijs Ankudovičs
Vitālijs Ankudovičs
Pauls Stradiņš Clinical University Hospital, Riga, Latvia
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Dmitrijs Blizņuks
Dmitrijs Blizņuks
Institute of Smart Computing Technologies, Riga Technical University, Riga, Latvia
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Timurs Mihailovs
Timurs Mihailovs
Institute of Smart Computing Technologies, Riga Technical University, Riga, Latvia
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Sergejs Lobanovs
Sergejs Lobanovs
Pauls Stradiņš Clinical University Hospital, Riga, Latvia
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Harald Sourij
Harald Sourij
Trials Unit for Interdisciplinary Metabolic Medicine, Division of Endocrinology and Diabetolgoy, Medical University of Graz, Graz, Austria
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Andreas F H Pfeiffer
Andreas F H Pfeiffer
Department of Endocrinology and Metabolic Medicine, Campus Benjamin Franklin, Charité University Medicine, Hindenburgdamm, Berlin, Germany
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Valdis Pīrāgs
Valdis Pīrāgs
Pauls Stradiņš Clinical University Hospital, Riga, Latvia
Faculty of Medicine, University of Latvia, Riga, Latvia
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Faculty of Medicine, University of Latvia, Riga, Latvia
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changing lifestyles, which include unhealthy dietary habits, sedentary behaviour, and increasing stress levels, interplaying with genetic and epigenetic factors ( 5 ). The intertwining of these two conditions amplifies the risk of various health
Patricia Arroyo Tardio
Patricia Arroyo Tardio
University Hospital Basel, Basel, Switzerland
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Gabriela Baldini
Gabriela Baldini
University Clinic of Medicine, Cantonal Hospital Baselland, Liestal, Switzerland
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Eleonora Seelig
Eleonora Seelig
University Hospital Basel, Basel, Switzerland
University Clinic of Medicine, Cantonal Hospital Baselland, Liestal, Switzerland
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University Clinic of Medicine, Cantonal Hospital Baselland, Liestal, Switzerland
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were admitted to the Clinical Trials Unit, University Hospital Basel, at 08:00 h. After arrival, stress levels were assessed with the perceived stress questionnaire ( 11 ). Basal metabolic rate was measured with indirect calorimetry (Quark RMR, COSMED
Qian Deng
Qian Deng
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Yue Zhu
Yue Zhu
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Mengmeng Zhang
Mengmeng Zhang
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Aihua Fei
Aihua Fei
Department of Endocrinology, The Second Affiliated Hospital of Anhui University of Chinese Medicine, Hefei, China
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Jiaqi Liang
Jiaqi Liang
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Jinjin Zheng
Jinjin Zheng
Graduate College of Anhui University of Chinese Medicine, Hefei, China
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Qingping Zhang
Qingping Zhang
College of Acupuncture-moxibustion and Tuina, Anhui University of Chinese Medicine, Hefei, China
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Tong Cheng
Tong Cheng
Department of Geriatrics, Zhongshan Hospital, Fudan University, Shanghai, China
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Xia Ge
Xia Ge
Department of Endocrinology, The Second Affiliated Hospital of Anhui University of Chinese Medicine, Hefei, China
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ferroptosis ( 10 , 11 , 12 ). Hyperglycemia leads to the overproduction of reactive oxygen species (ROS) and subsequently enhances oxidative stress. The increased HG and lipid ROS production, which were closely associated with pancreatic islet beta cell
Tsung-Hui Wu
Tsung-Hui Wu
Section of Endocrinology and Metabolism, Department of Medicine, Taipei Veterans General Hospital, Taipei, Taiwan
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Guan-Yu Su
Guan-Yu Su
Section of Endocrinology and Metabolism, Department of Medicine, Taipei Veterans General Hospital, Taipei, Taiwan
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Tsung-Yun Liu
Tsung-Yun Liu
Institute of Food Safety and Health Risk Assessment, College of Pharmaceutical Sciences, National Yang Ming Chiao Tung University, Taipei, Taiwan
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Hsiang-Tsui Wang
Hsiang-Tsui Wang
Institute of Food Safety and Health Risk Assessment, College of Pharmaceutical Sciences, National Yang Ming Chiao Tung University, Taipei, Taiwan
Institute of Pharmacology, College of Medicine, National Yang Ming Chiao Tung University, Taipei, Taiwan
PhD Program in Toxicology, Kaohsiung Medical University, Kaohsiung, Taiwan
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Institute of Pharmacology, College of Medicine, National Yang Ming Chiao Tung University, Taipei, Taiwan
PhD Program in Toxicology, Kaohsiung Medical University, Kaohsiung, Taiwan
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Chii-Min Hwu
Chii-Min Hwu
Section of Endocrinology and Metabolism, Department of Medicine, Taipei Veterans General Hospital, Taipei, Taiwan
Faculty of Medicine, National Yang Ming Chiao Tung University School of Medicine, Taipei, Taiwan
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Faculty of Medicine, National Yang Ming Chiao Tung University School of Medicine, Taipei, Taiwan
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effects through various mechanisms, including DNA and protein adduction, disruption of mitochondria and endoplasmic reticulum, cell membrane damage, oxidative stress, and inflammation and immune dysfunction ( 8 ). Due to an abundance of polyunsaturated
Chenmin Wei
Chenmin Wei
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
Department of Endocrinology, The Affiliated Jiangning Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Department of Endocrinology, The Affiliated Jiangning Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Zichen Zhang
Zichen Zhang
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Qi Fu
Qi Fu
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Yunqiang He
Yunqiang He
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Tao Yang
Tao Yang
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Min Sun
Min Sun
Department of Endocrinology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China
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Introduction Various factors may be responsible for β cell dysfunction, including reduced β cell mass (due to de- or transdifferentiation into other cell type) and β cell exhaustion (related to endoplasmic reticulum (ER) and oxidative stress
Mohamed Asrih
Mohamed Asrih
Service of Endocrinology, Diabetes, Nutrition and Patient Therapeutic Education, Geneva University Hospitals, Geneva, Switzerland
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Flore Sinturel
Flore Sinturel
Thoracic and Endocrine Surgery Division, Department of Surgery, Geneva University Hospitals (HUG), Geneva, Switzerland
Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Richard Dubos
Richard Dubos
Department and Division of Primary Care Medicine, Geneva University Hospitals (HUG), Geneva, Switzerland
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Idris Guessous
Idris Guessous
Department and Division of Primary Care Medicine, Geneva University Hospitals (HUG), Geneva, Switzerland
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Zoltan Pataky
Zoltan Pataky
Service of Endocrinology, Diabetes, Nutrition and Patient Therapeutic Education, Geneva University Hospitals, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Charna Dibner
Charna Dibner
Thoracic and Endocrine Surgery Division, Department of Surgery, Geneva University Hospitals (HUG), Geneva, Switzerland
Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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François R Jornayvaz
François R Jornayvaz
Service of Endocrinology, Diabetes, Nutrition and Patient Therapeutic Education, Geneva University Hospitals, Geneva, Switzerland
Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Department of Cell Physiology and Metabolism, Faculty of Medicine, University of Geneva, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Karim Gariani
Karim Gariani
Service of Endocrinology, Diabetes, Nutrition and Patient Therapeutic Education, Geneva University Hospitals, Geneva, Switzerland
Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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Diabetes Center, Faculty of Medicine, University of Geneva, Geneva, Switzerland
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-β (TGF-β) family, has been identified as a stress-responsive cytokine and metabolic modulator whose expression is markedly elevated in response to stress conditions, aging, obesity, and diabetes ( 3 , 6 , 7 , 8 , 9 , 10 , 11 , 12 , 13 ). In
Xue-Lian Zhang
Xue-Lian Zhang
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Xinyi Zhao
Xinyi Zhao
Department of Physiology, School of Medicine, Jinan University, Guangzhou, China
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Yong Wu
Yong Wu
Department of Physiology, School of Medicine, Jinan University, Guangzhou, China
Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Wen-qing Huang
Wen-qing Huang
Department of Transfusion Medicine, Shenzhen Hospital, Southern Medical University, Shenzhen, Guangdong, China
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Jun-jiang Chen
Jun-jiang Chen
Department of Physiology, School of Medicine, Jinan University, Guangzhou, China
Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Peijie Hu
Peijie Hu
Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Wei Liu
Wei Liu
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Yi-Wen Chen
Yi-Wen Chen
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Jin Hao
Jin Hao
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Rong-Rong Xie
Rong-Rong Xie
Department of Endocrinology, Beijing Tongren Hospital, Capital Medical University, Beijing, China
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Hsiao Chang Chan
Hsiao Chang Chan
Epithelial Cell Biology Research Center, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong SAR, China
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Ye Chun Ruan
Ye Chun Ruan
Department of Biomedical Engineering, Faculty of Engineering, The Hong Kong Polytechnic University, Hong Kong, China
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Hui Chen
Hui Chen
Cell-Gene Therapy Translational Medicine Research Center, The Third Affiliated Hospital of Sun Yat-Sen University, Guangzhou, China
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Jinghui Guo
Jinghui Guo
Department of Physiology, School of Medicine, Jinan University, Guangzhou, China
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dysfunction in type 2 diabetic mouse model via reducing oxidative stress, inflammation and pathological remodeling ( 7 ). However, despite the suggested therapeutic potential of Ang(1–7) for diabetes, whether the expression of endogenous Ang(1–7) and MAS-1 is
Aliyu Tijani Jibril
Aliyu Tijani Jibril
Department of Community Nutrition, School of Nutritional Sciences and Dietetics, Tehran University of Medical Sciences, Tehran, Iran
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Ahmad Jayedi
Ahmad Jayedi
Social Determinants of Health Research Center, Semnan University of Medical Sciences, Semnan, Iran
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Sakineh Shab-Bidar
Sakineh Shab-Bidar
Department of Community Nutrition, School of Nutritional Sciences and Dietetics, Tehran University of Medical Sciences, Tehran, Iran
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. The oxidative stress state in diabetes mellitus type 2 patients with different medications types . Journal of Chemical Health Risks 2022 12 523– 525 . ( https://doi.org/10.22034/JCHR.2022.690774 ) 4 Adnan Khalaf M Ghassan Zainal I
