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childhood, followed by a sharp acceleration from the onset of puberty onward, up to early adulthood. During puberty, the growth hormone (GH)/insulin-like growth factor (IGF)-1 axis and sex steroids become the main determinants of bone development, acting on
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CUNY Graduate School of Public Health and Health Policy, New York, New York, USA
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Department of Laboratory Medicine and Pathology, University of Washington, Seattle, Washington, USA
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Introduction Estradiol (17β-estradiol) is a natural sex steroid available in several exogenous preparations. The Endocrine Society recommended high-dose exogenous estradiol treatment as one part of feminizing hormone therapy for transgender
Alfred Health, Melbourne, Victoria, Australia
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Department of General Practice, Melbourne Medical School, The University of Melbourne, Victoria, Australia
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chloroquine (3 subjects) was continued throughout the study period. AAS, anabolic androgenic steroid; CI, confidence interval; FSH, follicle-stimulating hormone; IM, intramuscular; LH, luteinising hormone; PO, oral; RR, relative risk. To aid
EndoERN, APHP Consortium Pitie Salpetriere Hospital, Necker Hospital, Paris, France
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EndoERN, APHP Consortium Pitie Salpetriere Hospital, Necker Hospital, Paris, France
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EndoERN, APHP Consortium Pitie Salpetriere Hospital, Necker Hospital, Paris, France
Sorbonne University, Paris, France
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human ovary . Journal of Clinical Endocrinology and Metabolism 1994 79 670 – 672 . ( https://doi.org/10.1210/jcem.79.2.7519196 ) 5 Doldi N Bassan M Bonzi V Ferrari A . Effects of growth hormone and growth hormone-releasing hormone on steroid
Department of Medicine (Austin Health), University of Melbourne, Heidelberg, Victoria, Australia
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Department of Medicine (Austin Health), University of Melbourne, Heidelberg, Victoria, Australia
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Department of Medicine (Austin Health), University of Melbourne, Heidelberg, Victoria, Australia
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Department of Medicine (Austin Health), University of Melbourne, Heidelberg, Victoria, Australia
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complete the entire PSQI questionnaire. Figure 1 Study subjects and flow of participants. Baseline clinical characteristics are shown in Table 1 . There was no difference in age, duration of feminising hormone therapy, sex steroid
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Introduction Luteinizing hormone/chorionic gonadotropin receptor ( LHCGR ) is a G-protein coupled receptor that contains 674 amino acids and is a common receptor for both luteinizing hormone (LH) and human chorionic gonadotropin (hCG) ( 1
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the general population, although the risk in DBA is not as high as in FA or DC/TBD ( 4 ). Anti-Müllerian hormone (AMH) is a glycoprotein in the transforming growth factor-β (TGF-β) superfamily ( 16 , 17 ). AMH is a key factor in cell proliferation
Department of Growth and Reproduction, Copenhagen University Hospital - Rigshospitalet, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Copenhagen University Hospital - Rigshospitalet, Copenhagen, Denmark
Department of Clinical Medicine, University of Copenhagen, Copenhagen, Denmark
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International Center for Research and Research Training in Endocrine Disruption of Male Reproduction and Child Health (EDMaRC), Copenhagen University Hospital - Rigshospitalet, Copenhagen, Denmark
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and for comparison across age. A limitation of the study is the small number of boys with KS included. The current rationale to leverage age- and sex-adjusted biomarker SDS scores was previously explored as a reference framework for steroid hormones
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Department of Medicine, University of Padova, Padova, Italy
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-0209 ) 40 Callewaert F Boonen S & Vanderschueren D . Sex steroids and the male skeleton: a tale of two hormones . Trends in Endocrinology and Metabolism 2010 21 89 – 95 . ( https://doi.org/10.1016/j.tem.2009.09.002 ) 41 Marcus R Leary D
K.G. Jebsen Center for Autoimmune Disorders, University of Bergen, Bergen, Norway
Department of Medicine, Haukeland University Hospital, Bergen, Norway
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Department of Gynecology and Obstetrics, Haukeland University Hospital, Bergen, Norway
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K.G. Jebsen Center for Autoimmune Disorders, University of Bergen, Bergen, Norway
Department of Medicine, Haukeland University Hospital, Bergen, Norway
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Department of Endocrinology, Metabolism and Diabetes, Karolinska University Hospital, Stockholm, Sweden
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Department of Sleep, Landspitali University Hospital Reykjavík, Reykjavik, Iceland
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Allergy and Lung Health Unit, Melbourne School of Population and Global Health, The University of Melbourne, Melbourne, Victoria, Australia
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Albacete Faculty of Medicine, Castilla-La Mancha University, Albacete, Spain
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The National Research Center for the Working Environment, Copenhagen, Denmark
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K.G. Jebsen Center for Autoimmune Disorders, University of Bergen, Bergen, Norway
Department of Medicine, Haukeland University Hospital, Bergen, Norway
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did not use HRT. FSH and luteinizing hormone (LH), were analyzed using ELISAs (Demeditec Diagnostics, Kiel, Germany), and the steroid hormones (17β-estradiol, estrone, progesterone, testosterone and DHEA-S) were measured using liquid chromatography