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Introduction About 80–90% of primary hyperparathyroidism (pHPT) cases are caused by parathyroid adenoma. Long-term increased parathyroid hormone (PTH) secretion leads to a generalized disorder of calcium, phosphate and bone metabolism. The
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may influence adipose tissue differentiation and growth, impacting obesity through gene expression regulation or by modulating parathyroid hormone (PTH), calcium, and leptin ( 11 , 12 , 13 ). While several observational studies have explored the link
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/ by Alfred F Hess. A more recent definition of vitamin D deficiency has grown to include defective chondrocyte differentiation and lack of mineralization of the growth plate, but the common feature of vitamin D deficiency is insufficiently
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the endothelial surface. This induces the release of proinflammatory cytokines (IL6, IL8), growth factors such as PDGF, chemokines such as MCP1 and a lower expression of the enzyme endothelial nitric oxide synthase (eNOS) responsible for the production