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expression of FGF21 reportedly increased in a dose-dependent manner in mice treated with exogenous TH ( 72 , 73 ). ChREBP also regulates plasma TG levels by regulating FGF21 ( 74 , 75 ). In turn, peripheral administration of FGF21 could decrease TH levels
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morbidity and adult cardiovascular metabolic syndrome ( 2 , 3 , 4 ). In contrast, large for gestational age (LGA) is associated with increased risk for obstructed labor, postpartum hemorrhage, newborn hypoglycemia, obesity, and diabetes mellitus in