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%) G2 22 (54%) Treatment response sd 20 (49%) pd 21 (51%) Ki 67 1% 2 (5%) 2% 17 (41%) 4% 2 (5%) 5% 8 (20%) 10% 12 (29%) * Mean ( s.d. ). LTB was
Department of Clinical Biochemistry, Hospital of South West Jutland, Esbjerg, Denmark
Department of Endocrinology and Internal Medicine, Aarhus University Hospital, Aarhus, Denmark
Department of Internal Medicine, Lillebaelt Hospital, Kolding, Denmark
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Department of Molecular Medicine, Aarhus University Hospital, Aarhus, Denmark
Department of Clinical Genetics, Aarhus University Hospital, Aarhus, Denmark
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Department of Clinical Biochemistry, Hospital of South West Jutland, Esbjerg, Denmark
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Department of Clinical Biochemistry, Hospital of South West Jutland, Esbjerg, Denmark
Department of Haematology, Erasmus MC, University Medical Centre Rotterdam, Rotterdam, The Netherlands
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Department of Molecular Medicine, Aarhus University Hospital, Aarhus, Denmark
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Department of Clinical Biochemistry, Hospital of South West Jutland, Esbjerg, Denmark
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Introduction The risk of venous thromboembolism (VTE) among men born with Klinefelter syndrome (KS, 47,XXY) overall is increased more than four-fold ( 1 , 2 , 3 ). In particular, the relative risk of VTE in KS compared with the background