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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
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Department of Urology, the 2nd affiliated hospital, Jiangxi Medical College, Nanchang University, Nanchang, Jiangxi Province, China
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Nanchang University, Nanchang, Jiangxi Province, China
Institute of Neuroscience, Nanchang University, Nanchang, Jiangxi Province, China
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illustrated that SLC7A11 was critical for the tumorigenicity of PitNET cells. Figure 3 Knockdown of SLC7A11 inhibits PitNET cell proliferation and induces apoptosis. (A–B) SLC7A11 knockdown efficiency was examined by Western blot. (C–D) Cell viability
National Center for Neurological Disorders, Shanghai, China
Shanghai Clinical Medical Center of Neurosurgery, Shanghai, China
Neurosurgical Institute of Fudan University, Shanghai, China
Shanghai Key Laboratory of Brain Function and Restoration and Neural Regeneration, Shanghai, China
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National Center for Neurological Disorders, Shanghai, China
Shanghai Clinical Medical Center of Neurosurgery, Shanghai, China
Neurosurgical Institute of Fudan University, Shanghai, China
Shanghai Key Laboratory of Brain Function and Restoration and Neural Regeneration, Shanghai, China
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National Center for Neurological Disorders, Shanghai, China
Shanghai Clinical Medical Center of Neurosurgery, Shanghai, China
Neurosurgical Institute of Fudan University, Shanghai, China
Shanghai Key Laboratory of Brain Function and Restoration and Neural Regeneration, Shanghai, China
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National Center for Neurological Disorders, Shanghai, China
Shanghai Clinical Medical Center of Neurosurgery, Shanghai, China
Neurosurgical Institute of Fudan University, Shanghai, China
Shanghai Key Laboratory of Brain Function and Restoration and Neural Regeneration, Shanghai, China
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National Center for Neurological Disorders, Shanghai, China
Shanghai Clinical Medical Center of Neurosurgery, Shanghai, China
Neurosurgical Institute of Fudan University, Shanghai, China
Shanghai Key Laboratory of Brain Function and Restoration and Neural Regeneration, Shanghai, China
Department of Pathology, Huashan Hospital, Shanghai Medical School, Shanghai, China
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National Center for Neurological Disorders, Shanghai, China
Shanghai Clinical Medical Center of Neurosurgery, Shanghai, China
Neurosurgical Institute of Fudan University, Shanghai, China
Shanghai Key Laboratory of Brain Function and Restoration and Neural Regeneration, Shanghai, China
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National Center for Neurological Disorders, Shanghai, China
Shanghai Clinical Medical Center of Neurosurgery, Shanghai, China
Neurosurgical Institute of Fudan University, Shanghai, China
Shanghai Key Laboratory of Brain Function and Restoration and Neural Regeneration, Shanghai, China
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National Center for Neurological Disorders, Shanghai, China
Shanghai Clinical Medical Center of Neurosurgery, Shanghai, China
Neurosurgical Institute of Fudan University, Shanghai, China
Shanghai Key Laboratory of Brain Function and Restoration and Neural Regeneration, Shanghai, China
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contrast imaging (B and D) was observed. A tumor was observed above the pituitary gland during the endoscopic trans-tuberculum surgery (E). Hematoxylin and eosin staining shows bipolar spindled cells arranged in a fascicular or storiform pattern (F). The
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further analyze the liver situation of congenital IGHD patients. Metabolomics has been used in the comprehensive profiling of small molecular metabolites (<1 kDa) in cells, tissues, and whole organisms ( 7 ). It has been roughly divided into targeted
Princess Máxima Center for Pediatric Oncology, AB Utrecht, The Netherlands
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Princess Máxima Center for Pediatric Oncology, AB Utrecht, The Netherlands
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Princess Máxima Center for Pediatric Oncology, AB Utrecht, The Netherlands
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. ( https://doi.org/10.1053/j.seminhematol.2013.06.007 ) 13 Bakker B Oostdijk W Bresters D Walenkamp MJ Vossen JM Wit JM . Disturbances of growth and endocrine function after busulphan-based conditioning for haematopoietic stem cell
Division of Epidemiology and Biometry, Carl von Ossietzky University, Oldenburg, Germany
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Faculty of Health Sciences and Medicine, University of Lucerne, Lucerne, Switzerland
Department of Pediatrics, McMaster Children’s Hospital and McMaster University, Hamilton, Ontario, Canada
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, anterior, and posterior hypothalamus are compressed and displaced (B: sagittal T2WI and C: sagittal T1 WI with contrast enhancement). Figure 4 Hematoxylin and eosin staining of tumor material of case 3 showing palisading of the basal cells in
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Radiotherapy Related Research, The Christie NHS Foundation Trust, Manchester, UK
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Radiotherapy Related Research, The Christie NHS Foundation Trust, Manchester, UK
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%, respectively. Tumours such as ependymomas, craniopharyngiomas, germ cell tumors, and choroid plexus tumours are much less common, and each have a prevalence below 10% ( 2 ). Although oncological therapies have evolved over time, a balance between treatment
Fuzong Clinical Medical College of Fujian Medical University, Fuzhou, China
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Department of Neurosurgery, 900th Hospital, Fuzhou, China
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-enhanced images of the change in tumor cavity height after transsphenoidal surgery in two cases. Panels A and B and panels C and D are from the same patient, respectively. (A) Before surgery, the tumor cavity height is 28.88 mm. (B) After surgery, the tumor cavity
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removing residual after chemoradiotherapy. Figure 1 Different imaging grades for patients with bifocal intracranial germ cell tumors. Each lesion was marked by white dotted lines: (A) the neuroimaging features of sellar-predominant subtype 1, (B
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’-AGGAGCAAATGCCCTTTCTT-3’ (reverse). Figure 2 Distribution of mutations in the ANOS1 gene in the study series. (A) Structure of the ANOS1 gene. Coding exons are indicated by boxes, introns are indicated by black lines. (B) Structure of ANOS1 protein and
School of Medicine, Western Sydney University, Sydney, Australia
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Westmead Clinical School, University of Sydney, Sydney, Australia
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Westmead Clinical School, University of Sydney, Sydney, Australia
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Department of Neurosurgery, Westmead Hospital, Sydney, Australia
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School of Medicine, Western Sydney University, Sydney, Australia
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272639, Abcam). Somatostatin receptor (SSTR) status was examined using anti-SSTR2 (EP149, Epitomics – an Abcam company) and anti-SSTR5 (ab109495, Abcam). Keratins were localised using cytokeratin 8/18 (B221 and B23.1, Cell Marque – a Sigma-Aldrich company