Filter

Refine by collections

  • Metabolic Syndrome and Diabetes (6)

Refine by date

—
  • Print
  • Save

Search Results

You are looking at 1 - 6 of 6 items for :

  • "migration" x
  • Metabolic Syndrome and Diabetes x
Clear All
Wang-shu Liu Department of Endocrinology, Affiliated Hospital 2 of Nantong University and First People’s Hospital of Nantong City, Nantong, China

Search for other papers by Wang-shu Liu in
Google Scholar
PubMed Close
,
Ling-yan Hua Department of Ophthalmology, Affiliated Hospital 2 of Nantong University and First People’s Hospital of Nantong City, Nantong, China

Search for other papers by Ling-yan Hua in
Google Scholar
PubMed Close
,
Su-xiang Zhu Department of Endocrinology, Affiliated Hospital 2 of Nantong University and First People’s Hospital of Nantong City, Nantong, China

Search for other papers by Su-xiang Zhu in
Google Scholar
PubMed Close
,
Feng Xu Department of Endocrinology, Affiliated Hospital 2 of Nantong University and First People’s Hospital of Nantong City, Nantong, China

Search for other papers by Feng Xu in
Google Scholar
PubMed Close
,
Xue-qin Wang Department of Endocrinology, Affiliated Hospital 2 of Nantong University and First People’s Hospital of Nantong City, Nantong, China

Search for other papers by Xue-qin Wang in
Google Scholar
PubMed Close
,
Chun-feng Lu Department of Endocrinology, Affiliated Hospital 2 of Nantong University and First People’s Hospital of Nantong City, Nantong, China

Search for other papers by Chun-feng Lu in
Google Scholar
PubMed Close
,
Jian-bin Su Department of Endocrinology, Affiliated Hospital 2 of Nantong University and First People’s Hospital of Nantong City, Nantong, China

Search for other papers by Jian-bin Su in
Google Scholar
PubMed Close
, and
Feng Qi Emergency Intensive Care Unit, Affiliated Hospital 2 of Nantong University and First People’s Hospital of Nantong City, Nantong, China

Search for other papers by Feng Qi in
Google Scholar
PubMed Close

levels may be closely associated with CAN in patients with T2D. However, SDF-1 also promotes the mobilization and migration of endothelial progenitor cells (EPCs), so a number of studies have reported that elevated SDF-1 levels may have a protective

Article Type:
Research Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-21-0629
Volume/Issue:
Volume 11: Issue 4
Open access
Felix Reschke Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany

Search for other papers by Felix Reschke in
Google Scholar
PubMed Close
,
Torben Biester Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany

Search for other papers by Torben Biester in
Google Scholar
PubMed Close
,
Thekla von dem Berge Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany

Search for other papers by Thekla von dem Berge in
Google Scholar
PubMed Close
,
Dagmar Jamiolkowski Auf Der Bult Children’s Hospital, Department of Paediatric Dermatology, Hannover, Germany

Search for other papers by Dagmar Jamiolkowski in
Google Scholar
PubMed Close
,
Laura Hasse Auf Der Bult Children’s Hospital, Department of Paediatric Dermatology, Hannover, Germany

Search for other papers by Laura Hasse in
Google Scholar
PubMed Close
,
Francesca Dassie Padua University Hospital, Clinica Medica 3, Department of Medicine (DIMED), Padova, Veneto, Italy

Search for other papers by Francesca Dassie in
Google Scholar
PubMed Close
,
Pietro Maffei Padua University Hospital, Clinica Medica 3, Department of Medicine (DIMED), Padova, Veneto, Italy

Search for other papers by Pietro Maffei in
Google Scholar
PubMed Close
,
Katharina Klee Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany

Search for other papers by Katharina Klee in
Google Scholar
PubMed Close
,
Olga Kordonouri Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany

Search for other papers by Olga Kordonouri in
Google Scholar
PubMed Close
,
Hagen Ott Auf Der Bult Children’s Hospital, Department of Paediatric Dermatology, Hannover, Germany

Search for other papers by Hagen Ott in
Google Scholar
PubMed Close
, and
Thomas Danne Auf Der Bult Children’s Hospital, Centre for Paediatric Endocrinology, Diabetology, and Clinical Research, Hannover, Germany

Search for other papers by Thomas Danne in
Google Scholar
PubMed Close

keratinocyte and fibroblast proliferation, differentiation and migration ( 8 ). Among other things, this results in a disturbed barrier function and delayed wound healing. In addition, a reduced synthesis of collagen 1, a functional impairment of proliferation

Article Type:
Review Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-22-0410
Volume/Issue:
Volume 12: Issue 7
Open access
Sigrid Bjerge Gribsholt Department of Endocrinology and Internal Medicine, Aarhus University Hospital, Aarhus, Denmark
Steno Diabetes Center Aarhus, Aarhus University Hospital, Aarhus, Denmark
Department of Clinical Epidemiology, Aarhus University Hospital and Aarhus University, Aarhus, Denmark

Search for other papers by Sigrid Bjerge Gribsholt in
Google Scholar
PubMed Close
,
Morten Schmidt Department of Clinical Epidemiology, Aarhus University Hospital and Aarhus University, Aarhus, Denmark
Department of Clinical Medicine, Aarhus University, Aarhus, Denmark
Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark

Search for other papers by Morten Schmidt in
Google Scholar
PubMed Close
,
Eskild Bendix Kristiansen Department of Clinical Epidemiology, Aarhus University Hospital and Aarhus University, Aarhus, Denmark

Search for other papers by Eskild Bendix Kristiansen in
Google Scholar
PubMed Close
,
Bjørn Richelsen Department of Endocrinology and Internal Medicine, Aarhus University Hospital, Aarhus, Denmark
Steno Diabetes Center Aarhus, Aarhus University Hospital, Aarhus, Denmark
Department of Clinical Medicine, Aarhus University, Aarhus, Denmark

Search for other papers by Bjørn Richelsen in
Google Scholar
PubMed Close
, and
Henrik Toft Sørensen Department of Clinical Epidemiology, Aarhus University Hospital and Aarhus University, Aarhus, Denmark
Department of Clinical Medicine, Aarhus University, Aarhus, Denmark

Search for other papers by Henrik Toft Sørensen in
Google Scholar
PubMed Close

for diagnosis codes), and from the CRS we obtained information on vital status and migration ( 20 ). For patients with a secondary diagnosis code of overweight/obesity, we tabulated the 25 most common primary diagnosis codes (Supplementary Table 4

Article Type:
Research Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-23-0452
Volume/Issue:
Volume 13: Issue 4
Open access
Zhandong Lei Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China
Department of Anatomy, Shanxi Medical University, Taiyuan, China

Search for other papers by Zhandong Lei in
Google Scholar
PubMed Close
,
Yunfei Chen Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China

Search for other papers by Yunfei Chen in
Google Scholar
PubMed Close
,
Jin Wang Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China

Search for other papers by Jin Wang in
Google Scholar
PubMed Close
,
Yan Zhang Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China

Search for other papers by Yan Zhang in
Google Scholar
PubMed Close
,
Wenjuan Shi Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China

Search for other papers by Wenjuan Shi in
Google Scholar
PubMed Close
,
Xuejiao Wang Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China

Search for other papers by Xuejiao Wang in
Google Scholar
PubMed Close
,
Dehai Xing Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China

Search for other papers by Dehai Xing in
Google Scholar
PubMed Close
,
Dongxue Li Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China

Search for other papers by Dongxue Li in
Google Scholar
PubMed Close
, and
Xiangying Jiao Key Laboratory of Cellular Physiology, Ministry of Education, and the Department of Physiology, Shanxi Medical University, Taiyuan, China

Search for other papers by Xiangying Jiao in
Google Scholar
PubMed Close

International 2020 44 2094 – 2106 . ( https://doi.org/10.1002/cbin.11418 ) 45 Zhang J Tian X Yin H Xiao S Yi S Zhang Y Zeng F . TXNIP induced by MondoA, rather than ChREBP, suppresses cervical cancer cell proliferation, migration and invasion

Article Type:
Research Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-21-0641
Volume/Issue:
Volume 11: Issue 4
Open access
Weiwei He School of Medicine, Xiamen University, Xiamen, China
Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, China
Fujian Provincial Key Laboratory of Translational Medicine for Diabetes, Xiamen, China

Search for other papers by Weiwei He in
Google Scholar
PubMed Close
,
Caoxin Huang Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, China
Fujian Provincial Key Laboratory of Translational Medicine for Diabetes, Xiamen, China

Search for other papers by Caoxin Huang in
Google Scholar
PubMed Close
,
Xiulin Shi Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, China
Fujian Provincial Key Laboratory of Translational Medicine for Diabetes, Xiamen, China
Department of Endocrinology and Diabetes, The First Affiliated Hospital of Xiamen University, Xiamen, China

Search for other papers by Xiulin Shi in
Google Scholar
PubMed Close
,
Menghua Wu School of Medicine, Xiamen University, Xiamen, China
Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, China
Fujian Provincial Key Laboratory of Translational Medicine for Diabetes, Xiamen, China

Search for other papers by Menghua Wu in
Google Scholar
PubMed Close
,
Han Li School of Medicine, Xiamen University, Xiamen, China
Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, China
Fujian Provincial Key Laboratory of Translational Medicine for Diabetes, Xiamen, China

Search for other papers by Han Li in
Google Scholar
PubMed Close
,
Qiuhong Liu School of Medicine, Xiamen University, Xiamen, China
Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, China
Fujian Provincial Key Laboratory of Translational Medicine for Diabetes, Xiamen, China

Search for other papers by Qiuhong Liu in
Google Scholar
PubMed Close
,
Xiaofang Zhang Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, China
Fujian Provincial Key Laboratory of Translational Medicine for Diabetes, Xiamen, China

Search for other papers by Xiaofang Zhang in
Google Scholar
PubMed Close
,
Yan Zhao Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, China
Fujian Provincial Key Laboratory of Translational Medicine for Diabetes, Xiamen, China

Search for other papers by Yan Zhao in
Google Scholar
PubMed Close
, and
Xuejun Li Xiamen Diabetes Institute, The First Affiliated Hospital of Xiamen University, Xiamen, China
Fujian Provincial Key Laboratory of Translational Medicine for Diabetes, Xiamen, China
Department of Endocrinology and Diabetes, The First Affiliated Hospital of Xiamen University, Xiamen, China
Xiamen Clinical Medical Center for Endocrine and Metabolic Diseases, Xiamen Diabetes Prevention and Treatment Center, Xiamen, China

Search for other papers by Xuejun Li in
Google Scholar
PubMed Close

, cell migration, inflammation, and wound healing ( 54 ). Increasingly, JunB has also been found to play an important role in fibrogenesis, and JunB activates the TGF-β pathway and promotes COL1A2 deposition ( 55 , 56 , 57 ). In this study, we found

Article Type:
Research Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-22-0502
Volume/Issue:
Volume 12: Issue 2
Open access
Qian Deng Graduate College of Anhui University of Chinese Medicine, Hefei, China

Search for other papers by Qian Deng in
Google Scholar
PubMed Close
,
Yue Zhu Graduate College of Anhui University of Chinese Medicine, Hefei, China

Search for other papers by Yue Zhu in
Google Scholar
PubMed Close
,
Mengmeng Zhang Graduate College of Anhui University of Chinese Medicine, Hefei, China

Search for other papers by Mengmeng Zhang in
Google Scholar
PubMed Close
,
Aihua Fei Department of Endocrinology, The Second Affiliated Hospital of Anhui University of Chinese Medicine, Hefei, China

Search for other papers by Aihua Fei in
Google Scholar
PubMed Close
,
Jiaqi Liang Graduate College of Anhui University of Chinese Medicine, Hefei, China

Search for other papers by Jiaqi Liang in
Google Scholar
PubMed Close
,
Jinjin Zheng Graduate College of Anhui University of Chinese Medicine, Hefei, China

Search for other papers by Jinjin Zheng in
Google Scholar
PubMed Close
,
Qingping Zhang College of Acupuncture-moxibustion and Tuina, Anhui University of Chinese Medicine, Hefei, China

Search for other papers by Qingping Zhang in
Google Scholar
PubMed Close
,
Tong Cheng Department of Geriatrics, Zhongshan Hospital, Fudan University, Shanghai, China

Search for other papers by Tong Cheng in
Google Scholar
PubMed Close
, and
Xia Ge Department of Endocrinology, The Second Affiliated Hospital of Anhui University of Chinese Medicine, Hefei, China

Search for other papers by Xia Ge in
Google Scholar
PubMed Close

mouse models and also has protective effects in models of diabetes. Additionally, through activating the PI3K-AKT signaling pathway, Fer-1-mediated inhibition of ferroptosis could significantly reduce inflammation, promote proliferation and migration of

Article Type:
Review Article
Collections:
Metabolic Syndrome and Diabetes
DOI:
https://doi.org/10.1530/EC-22-0419
Volume/Issue:
Volume 12: Issue 3
Open access