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disorders of lipid metabolism ( 1 ), mainly with increased total cholesterol (TC) and LDL-C ( 2 ) in blood. Elevated LDL-C can lead to progressive lipid accumulation, plaque formation in the arteries and increase the risk of cardiovascular disease (CVD), the
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MetS ( 4 ). Thyroid hormones (THs) are vital hormones that regulate the metabolism of substances ( 5 ) and therefore affect MetS and its components by regulating energy homeostasis, lipid metabolism, and gluconeogenesis. However, the association
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. Previous studies have found that SCH affects the terms of lipid metabolism in pregnant women. However, current studies are limited, and the correlation between lipid metabolism disorders and the pathogenesis of SCH pregnancy women remains unclear. Li et al
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regulator of glucose and lipid metabolism . Annual Review of Nutrition 2007 27 179 – 192 . ( https://doi.org/10.1146/annurev.nutr.27.061406.093618 ) 24 Uyeda K & Repa JJ . Carbohydrate response element binding protein, ChREBP, a transcription
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patients on levothyroxine monotherapy have a low serum fT3 level, and those with a normal serum TSH level were not necessarily euthyroid in terms of energy expenditure, lipid metabolism, dissatisfaction, or quality of life, including fatigue ( 36 , 37
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, and the mechanism through which TSH disrupts insulin signal transduction appears to involve the ER stress–NK pathway. Elevated TSH in subclinical hypothyroidism leads to ER stress in adipose tissue, possibly via impaired lipid metabolism, expanded
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production via lipid metabolism regulation ( 4 ). Hence, any disorder affecting the liver or thyroid can potentially impact the function of the other ( 5 ). Management of hyperthyroidism co-occurring with liver failure presents unique challenges. In such