Search Results
Search for other papers by Rebeca Esquivel-Zuniga in
Google Scholar
PubMed
Search for other papers by Alan D Rogol in
Google Scholar
PubMed
disease, anorexia nervosa, excessive exercise, chronic kidney disease, sickle cell disease and thalassemia, severe obesity, opioids); chronic stress Female Turner syndrome Premature ovarian insufficiency Kallmann syndrome Combined pituitary
Search for other papers by Martijn J J Finken in
Google Scholar
PubMed
Search for other papers by Aleid J G Wirix in
Google Scholar
PubMed
Search for other papers by Ines A von Rosenstiel-Jadoul in
Google Scholar
PubMed
Search for other papers by Bibian van der Voorn in
Google Scholar
PubMed
Search for other papers by Mai J M Chinapaw in
Google Scholar
PubMed
Search for other papers by Michaela F Hartmann in
Google Scholar
PubMed
Search for other papers by Joana E Kist-van Holthe in
Google Scholar
PubMed
Search for other papers by Stefan A Wudy in
Google Scholar
PubMed
Search for other papers by Joost Rotteveel in
Google Scholar
PubMed
, while the reverse reaction is catalyzed by the type 2 isozyme in the kidney ( 14 ). Only few studies have investigated whether childhood obesity-associated hypertension is accompanied by alterations in 11β-HSDs. A small study (of n = 41) found that
Search for other papers by Sommayya Aftab in
Google Scholar
PubMed
Search for other papers by Diliara Gubaeva in
Google Scholar
PubMed
Search for other papers by Jayne A L Houghton in
Google Scholar
PubMed
Search for other papers by Antonia Dastamani in
Google Scholar
PubMed
Search for other papers by Ellada Sotiridou in
Google Scholar
PubMed
Search for other papers by Clare Gilbert in
Google Scholar
PubMed
Search for other papers by Sarah E Flanagan in
Google Scholar
PubMed
Search for other papers by Anatoly Tiulpakov in
Google Scholar
PubMed
Search for other papers by Maria Melikyan in
Google Scholar
PubMed
Search for other papers by Pratik Shah in
Google Scholar
PubMed
). GDH is highly expressed in pancreatic β-cells, liver, kidney and brain, where it plays an important role in the metabolism of amino acids and ammonia ( 3 ). In pancreatic β-cells, it catalyses the oxidative deamination of glutamate to alpha
Department of Child and Adolescent Medicine, Section of Pediatric Cardiology, University Hospital Jena, Am Klinikum, Jena, Germany
Search for other papers by Alexandra Kiess in
Google Scholar
PubMed
Search for other papers by Jessica Green in
Google Scholar
PubMed
Search for other papers by Anja Willenberg in
Google Scholar
PubMed
Search for other papers by Uta Ceglarek in
Google Scholar
PubMed
Search for other papers by Ingo Dähnert in
Google Scholar
PubMed
Department of Women and Child Health, Hospital for Children and Adolescents and Center for Pediatric Research (CPL), University of Leipzig, Liebigstrasse, Leipzig, Germany
Search for other papers by Wieland Kiess in
Google Scholar
PubMed
Department of Women and Child Health, Hospital for Children and Adolescents and Center for Pediatric Research (CPL), University of Leipzig, Liebigstrasse, Leipzig, Germany
Search for other papers by Mandy Vogel in
Google Scholar
PubMed
that associations would exist between the cardiac marker levels and an individual’s growth and developmental status as represented by IGF-1 and IGFBP3 levels or their metabolic status as represented by HbA1c or the kidney function marker Cystatin C
Search for other papers by Mireille N M van Poppel in
Google Scholar
PubMed
Search for other papers by Christopher J Nolan in
Google Scholar
PubMed
Search for other papers by Gernot Desoye in
Google Scholar
PubMed
Mutation in GL13 NA Absent gallbladder, thyroidal atrophy, adrenal atrophy, malrotation of intestine, atresia of anus, bilateral hypoplasia of kidney, hypoplasia of tentalia, hypospadia, polysyndactyly, polysplenia Johnson et al. ( 41 ) M
Search for other papers by Nathalia G B P Ferreira in
Google Scholar
PubMed
Search for other papers by Joao L O Madeira in
Google Scholar
PubMed
Search for other papers by Peter Gergics in
Google Scholar
PubMed
Search for other papers by Renata Kertsz in
Google Scholar
PubMed
Search for other papers by Juliana M Marques in
Google Scholar
PubMed
Search for other papers by Nicholas S S Trigueiro in
Google Scholar
PubMed
Search for other papers by Anna Flavia Figueredo Benedetti in
Google Scholar
PubMed
Search for other papers by Bruna V Azevedo in
Google Scholar
PubMed
Universidade de São Paulo, Zebrafish Facility, São Paulo, São Paulo, Brazil
Search for other papers by Bianca H V Fernandes in
Google Scholar
PubMed
Search for other papers by Debora D Bissegatto in
Google Scholar
PubMed
Search for other papers by Isabela P Biscotto in
Google Scholar
PubMed
Search for other papers by Qing Fang in
Google Scholar
PubMed
Search for other papers by Qianyi Ma in
Google Scholar
PubMed
Search for other papers by Asye B Ozel in
Google Scholar
PubMed
Search for other papers by Jun Li in
Google Scholar
PubMed
Search for other papers by Sally A Camper in
Google Scholar
PubMed
Search for other papers by Alexander A L Jorge in
Google Scholar
PubMed
Search for other papers by Berenice B Mendonça in
Google Scholar
PubMed
Search for other papers by Ivo J P Arnhold in
Google Scholar
PubMed
Search for other papers by Luciani R Carvalho in
Google Scholar
PubMed
, such as the development of the genitals, skeleton, and/or kidney ( 3 ). Genotype–phenotype correlations are not always clear, making it difficult to select candidate genes for analysis and to predict the severity of clinical presentation based on DNA