Filter

Refine by collections

  • Thyroid (4)

Refine by date

—
  • Print
  • Save

Search Results

You are looking at 1 - 4 of 4 items for :

Clear All
Caiyan Mo Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

Search for other papers by Caiyan Mo in
Google Scholar
PubMed Close
,
Tao Tong Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

Search for other papers by Tao Tong in
Google Scholar
PubMed Close
,
Ying Guo Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

Search for other papers by Ying Guo in
Google Scholar
PubMed Close
,
Zheng Li Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

Search for other papers by Zheng Li in
Google Scholar
PubMed Close
, and
Liyong Zhong Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

Search for other papers by Liyong Zhong in
Google Scholar
PubMed Close

Introduction Growth hormone-secreting pituitary adenoma (GHPA) is due to the overproduction of growth hormone (GH) by pituitary adenomas, which stimulates the liver to produce insulin-like growth factor 1 (IGF-1), and the long

Article Type:
Research Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-23-0439
Volume/Issue:
Volume 13: Issue 4
Open access
Bushra Shahida Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden

Search for other papers by Bushra Shahida in
Google Scholar
PubMed Close
,
Tereza Planck Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden

Search for other papers by Tereza Planck in
Google Scholar
PubMed Close
,
Tania Singh Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden

Search for other papers by Tania Singh in
Google Scholar
PubMed Close
,
Peter Åsman Department of Clinical Sciences Malmö, Ophthalmology, Lund University, Malmö, Sweden
Department of Ophthalmology, Skåne University Hospital, Malmö, Sweden

Search for other papers by Peter Åsman in
Google Scholar
PubMed Close
, and
Mikael Lantz Department of Clinical Sciences, Genomics, Diabetes and Endocrinology, Lund University, Malmö, Sweden
Department of Diabetes & Endocrinology, Skåne University Hospital, Malmö, Sweden

Search for other papers by Mikael Lantz in
Google Scholar
PubMed Close

TSHR induces cross-talk with insulin-like growth factor 1 receptor (IGF1R) ( 10 ), which results in elevated levels of hyaluronic acid, leading to the disruption of the extraocular muscles ( 11 ). T cells bind to CD40 on orbital fibroblasts and induce

Article Type:
Research Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-23-0374
Volume/Issue:
Volume 13: Issue 6
Open access
Brenda Anguiano Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Universidad Nacional Autónoma de México, Campus Juriquilla, Querétaro, México

Search for other papers by Brenda Anguiano in
Google Scholar
PubMed Close
,
Carlos Montes de Oca Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Universidad Nacional Autónoma de México, Campus Juriquilla, Querétaro, México

Search for other papers by Carlos Montes de Oca in
Google Scholar
PubMed Close
,
Evangelina Delgado-González Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Universidad Nacional Autónoma de México, Campus Juriquilla, Querétaro, México

Search for other papers by Evangelina Delgado-González in
Google Scholar
PubMed Close
, and
Carmen Aceves Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Universidad Nacional Autónoma de México, Campus Juriquilla, Querétaro, México

Search for other papers by Carmen Aceves in
Google Scholar
PubMed Close

-adrenergic receptor levels. (70) 0.1 LNCaP Biphasic response. Increases the specific binding to GH and GH receptor expression within the first 24 h and reduces the binding at 72 h. (63) 0.1 LNCaP Increases gene expression of IGF-1, IGF

Article Type:
Review Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-21-0581
Volume/Issue:
Volume 11: Issue 2
Open access
Jakub Supronik Department of Endocrinology, Diabetology and Internal Medicine, Medical University of Bialystok, Bialystok, Poland

Search for other papers by Jakub Supronik in
Google Scholar
PubMed Close
,
Małgorzata Szelachowska Department of Endocrinology, Diabetology and Internal Medicine, Medical University of Bialystok, Bialystok, Poland

Search for other papers by Małgorzata Szelachowska in
Google Scholar
PubMed Close
,
Adam Kretowski Department of Endocrinology, Diabetology and Internal Medicine, Medical University of Bialystok, Bialystok, Poland
Clinical Research Centre, Medical University of Bialystok, Bialystok, Poland

Search for other papers by Adam Kretowski in
Google Scholar
PubMed Close
, and
Katarzyna Siewko Department of Endocrinology, Diabetology and Internal Medicine, Medical University of Bialystok, Bialystok, Poland

Search for other papers by Katarzyna Siewko in
Google Scholar
PubMed Close

cells ( 16 ). Lately, it has been suggested that cross-talk between TSHR activated by TRAbs and insulin-like growth factor 1 receptor (IGF1R), which is overexpressed in the orbital connective tissue, plays a great role in the pathogenesis of this process

Article Type:
Review Article
Collections:
Thyroid
DOI:
https://doi.org/10.1530/EC-22-0303
Volume/Issue:
Volume 11: Issue 12
Open access