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hypertension in patients with this condition because of elevated progesterone levels and altered specificity of the MR receptor with progesterone and traditional MR antagonists now acting as potent MR agonists. The biochemical profile includes normal potassium
K.G. Jebsen Center for Autoimmune Disorders, University of Bergen, Bergen, Norway
Department of Medicine, Haukeland University Hospital, Bergen, Norway
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K.G. Jebsen Center for Autoimmune Disorders, University of Bergen, Bergen, Norway
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Department of Endocrinology, Metabolism and Diabetes, Karolinska University Hospital, Stockholm, Sweden
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Department of Sleep, Landspitali University Hospital Reykjavík, Reykjavik, Iceland
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Albacete Faculty of Medicine, Castilla-La Mancha University, Albacete, Spain
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The National Research Center for the Working Environment, Copenhagen, Denmark
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K.G. Jebsen Center for Autoimmune Disorders, University of Bergen, Bergen, Norway
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did not use HRT. FSH and luteinizing hormone (LH), were analyzed using ELISAs (Demeditec Diagnostics, Kiel, Germany), and the steroid hormones (17β-estradiol, estrone, progesterone, testosterone and DHEA-S) were measured using liquid chromatography
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ovulating, non-hyperandrogenic women. The women in the control group were healthy volunteers with normal ovulating cycles (28 ± 2 days, blood progesterone levels >10 ng/mL in the luteal phase of two consecutive cycles), no signs of hyperandrogenism or
School of Life Science and Engineering, Southwest Jiaotong University, Chengdu, Sichuan, China
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.1093/humrep/des191 ) 29 Yuan XH Fan YY Yang CR Gao XR Zhang LL Hu Y Wang YQ & Jun H . Progesterone amplifies oxidative stress signal and promotes NO production via H2O2 in mouse kidney arterial endothelial cells . Journal of Steroid
Unit of Endocrinology, Diabetes Mellitus and Metabolism, ARETAIEION University Hospital, School of Medicine, National and Kapodistrian University of Athens, Athens, Greece
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Unit of Endocrinology, Diabetes Mellitus and Metabolism, ARETAIEION University Hospital, School of Medicine, National and Kapodistrian University of Athens, Athens, Greece
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Unit of Endocrinology, Diabetes Mellitus and Metabolism, ARETAIEION University Hospital, School of Medicine, National and Kapodistrian University of Athens, Athens, Greece
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adrenals, leading thus to the production of cortisol and DHEAS ( Fig. 2 ). In humans and primates, at term, the placenta does not express cytochrome P450c17, an enzyme necessary for estrogen synthesis from progesterone. In placental syncytiotrophoblast
Department of Endocrinology and Metabolism, People's Hospital of Liaoning Province, Shenyang, People’s Republic of China
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Department of Laboratory Medical, The First Hospital of China Medical University, Shenyang, People’s Republic of China
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.114.118513 ) 25031358 25 Lee JH Kim TH Oh SJ Yoo JY Akira S Ku BJ Lydon JP Jeong JW . Signal transducer and activator of transcription-3 (Stat3) plays a critical role in implantation via progesterone receptor in uterus . FASEB Journal 2013 27
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therapy and not suitable for local treatment. Inclusion criteria were immunohistochemical oestrogen receptor (ER) positivity (≥1%) and/or progesterone receptor (PR) positivity (≥1%); in all cases, the tumour had to be human epidermal growth factor receptor
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postnatal months 3 and 12 in infants born to pre-eclamptic and normotensive mothers. Figure 1 Schematic flow diagram representing the steroid hormone pathways regulating cortisol availability. Progesterone-, corticosterone- and cortisol-related steroid
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postmenopausal patients or on the 7th day in the menstrual cycle for premenopausal patients. ER positivity or progesterone receptor positivity was defined as both ≥1% positive tumor cells with nuclear staining. The study was approved by the Ethics Commission of
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GDM has not been fully elucidated. Previous studies have suggested that antagonism of insulin by hormones secreted by the placenta, such as placental prolactin (HPL), estrogen, progesterone, etc., mainly contributes to pathogenesis of GDM ( 2 ). In