Alexander V Amram
Alexander V Amram
Department of Physiology, Cardiovascular Research Institute, University of California, San Francisco, San Francisco, California, USA
Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, San Francisco, California, USA
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Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, San Francisco, California, USA
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Stephen Cutie
Stephen Cutie
Department of Physiology, Cardiovascular Research Institute, University of California, San Francisco, San Francisco, California, USA
Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, San Francisco, California, USA
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Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, San Francisco, California, USA
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Guo N Huang
Guo N Huang
Department of Physiology, Cardiovascular Research Institute, University of California, San Francisco, San Francisco, California, USA
Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, San Francisco, California, USA
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Eli and Edythe Broad Center for Regeneration Medicine and Stem Cell Research, University of California, San Francisco, San Francisco, California, USA
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study that found that prenatal mice with CM-specific GR knockouts die prematurely due to the development of various cardiac pathologies ( 47 ), an interesting development occurred where double knockout mouse models with inactivated MR in addition to GR
Ursula M M Costa
Ursula M M Costa
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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Carla R P Oliveira
Carla R P Oliveira
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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Roberto Salvatori
Roberto Salvatori
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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José A S Barreto-Filho
José A S Barreto-Filho
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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Viviane C Campos
Viviane C Campos
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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Francielle T Oliveira
Francielle T Oliveira
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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Ivina E S Rocha
Ivina E S Rocha
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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Joselina L M Oliveira
Joselina L M Oliveira
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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Wersley A Silva
Wersley A Silva
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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Manuel H Aguiar-Oliveira
Manuel H Aguiar-Oliveira
Division of Cardiology, Division of Endocrinology, Division of Endocrinology, Federal University of Sergipe, Aracaju, SE 49060-100, Brazil
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synergistic anabolic effect on muscle mass, but antagonist effects on insulin action (GH-reducing and IGF1 increasing insulin sensitivity) and lipolysis (GH increasing and IGF1 reducing it) (2) . Adult onset GH deficiency (GHD) has been described as model of
Robert I Menzies
Robert I Menzies
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Xin Zhao
Xin Zhao
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Linda J Mullins
Linda J Mullins
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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John J Mullins
John J Mullins
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Carolynn Cairns
Carolynn Cairns
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Nicola Wrobel
Nicola Wrobel
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Donald R Dunbar
Donald R Dunbar
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Matthew A Bailey
Matthew A Bailey
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Christopher J Kenyon
Christopher J Kenyon
The University/BHF Centre for Cardiovascular Science, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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associated with apoptosis Discussion An infusion of ACTH was used to model molecular and morphological changes in the mouse adrenal gland that contribute to long-term steroidogenic control. Previously we have reported pathophysiological responses to
Xuhua Mao
Xuhua Mao
Department of Clinical Laboratory, Yixing People’s Hospital, Yixing, Wuxi, Jiangsu, China
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Hucheng Chen
Hucheng Chen
Department of Nuclear Medicine, Nanjing Hospital Affiliated to Nanjing Medical University, Nanjing, Jiangsu, China
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Junmin Tang
Junmin Tang
Department of Clinical Laboratory, Yixing People’s Hospital, Yixing, Wuxi, Jiangsu, China
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Liangliang Wang
Liangliang Wang
Department of Neurology, Yixing People’s Hospital Affiliated to Jiangsu University, Yixing, Wuxi, Jiangsu, China
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Tingting Shu
Tingting Shu
Department of Central Laboratory, Jiangsu Province Official Hospital, Nanjing, Jiangsu, China
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Fatty Acid oxidation and increased hepatic glucose production in a mouse model of hereditary hemochromatosis . Diabetes 2011 60 80 – 87 . ( doi:10.2337/db10-0593 )
Yunting Lin
Yunting Lin
Department of Surgical Medicine, Ningbo Medical Center Lihuili Hospital, Ningbo, Zhejiang, China
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Endi Song
Endi Song
Department of Internal Medicine, Ningbo Yinzhou No.2 Hospital, Ningbo, Zhejiang, China
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Han Jin
Han Jin
Department of Medicine, Ningbo University, Ningbo, Zhejiang, China
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Yong Jin
Yong Jin
Department of Internal Medicine, Ningbo Yinzhou No.2 Hospital, Ningbo, Zhejiang, China
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LH-induced endothelial dysfunction and arteriosclerosis. Additionally, in a mouse model, mice treated with GnRH/LHRH antagonists (exhibiting higher FSH levels but unchanged testosterone levels) had less fat mass and fewer atherosclerotic plaques than
Angelica Amorim Amato
Angelica Amorim Amato
Department of Pharmaceutical Sciences, University of Brasilia, Brasilia, Brazil
Department of Developmental and Cell Biology, University of California, Irvine, California, USA
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Department of Developmental and Cell Biology, University of California, Irvine, California, USA
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Hailey Brit Wheeler
Hailey Brit Wheeler
Department of Developmental and Cell Biology, University of California, Irvine, California, USA
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Bruce Blumberg
Bruce Blumberg
Department of Developmental and Cell Biology, University of California, Irvine, California, USA
Department of Pharmaceutical Sciences, University of California, Irvine, California, USA
Department of Biomedical Engineering, University of California, Irvine, California, USA
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Department of Pharmaceutical Sciences, University of California, Irvine, California, USA
Department of Biomedical Engineering, University of California, Irvine, California, USA
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. ( https://doi.org/10.1016/j.celrep.2017.05.060 ) 124 Nagata M Suzuki W Iizuka S Tabuchi M Maruyama H Takeda S Aburada M Miyamoto K Type 2 diabetes mellitus in obese mouse model induced by monosodium glutamate . Experimental Animals
Ling Shan
Ling Shan
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Hospital of China Medical University, Shenyang, People’s Republic of China
Department of Endocrinology and Metabolism, People's Hospital of Liaoning Province, Shenyang, People’s Republic of China
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Department of Endocrinology and Metabolism, People's Hospital of Liaoning Province, Shenyang, People’s Republic of China
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Yingying Zhou
Yingying Zhou
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Hospital of China Medical University, Shenyang, People’s Republic of China
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Shiqiao Peng
Shiqiao Peng
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Hospital of China Medical University, Shenyang, People’s Republic of China
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Xinyi Wang
Xinyi Wang
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Hospital of China Medical University, Shenyang, People’s Republic of China
Department of Laboratory Medical, The First Hospital of China Medical University, Shenyang, People’s Republic of China
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Department of Laboratory Medical, The First Hospital of China Medical University, Shenyang, People’s Republic of China
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Zhongyan Shan
Zhongyan Shan
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Hospital of China Medical University, Shenyang, People’s Republic of China
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Weiping Teng
Weiping Teng
Department of Endocrinology and Metabolism, Institute of Endocrinology, Liaoning Provincial Key Laboratory of Endocrine Diseases, The First Hospital of China Medical University, Shenyang, People’s Republic of China
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implantation . Endocrine Reviews 2004 25 341 – 373 . ( https://doi.org/10.1210/er.2003-0020 ) 15180948 10.1210/er.2003-0020 16 Wang H Dey SK . Roadmap to embryo implantation: clues from mouse models . Nature Reviews: Genetics 2006 7 185 – 199
Stefan Schulz
Stefan Schulz
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Anika Mann
Anika Mann
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Benjamin Novakhov
Benjamin Novakhov
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Hugh D Piggins
Hugh D Piggins
Institute of Pharmacology and Toxicology, Faculty of Life Sciences, Jena University Hospital, Friedrich Schiller University Jena, Drackendorfer Straße 1, D-07747 Jena, Germany
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Amelie Lupp
Amelie Lupp
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amino acids 419–428 of the human VPAC2 receptor. Given the high degree of sequence homology in the carboxyl-terminal domain of human, mouse and rat VPAC2 receptors, cross-reactivity of SP235 between human and rodent VPAC2 receptors is expected and
Chunliang Yang
Chunliang Yang
Department of Integrated Traditional Chinese and Western Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
The Center for Biomedical Research, Tongji Hospital Research Building, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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The Center for Biomedical Research, Tongji Hospital Research Building, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Junyi Li
Junyi Li
Department of Integrated Traditional Chinese and Western Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Fei Sun
Fei Sun
The Center for Biomedical Research, Tongji Hospital Research Building, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Haifeng Zhou
Haifeng Zhou
Department of Integrated Traditional Chinese and Western Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Jia Yang
Jia Yang
Department of Integrated Traditional Chinese and Western Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Chao Yang
Chao Yang
Department of Gerontology, Hubei Provincial Hospital of Integrated Chinese and Western Medicine, Wuhan, China
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protein at the steady state ( 20 ). Notably, the expression level of SGK1 sharply increases in the presence of glucocorticoids ( 4 ). Abnormally elevated SGK1 fuels blockage of insulin secretion in the mouse model. The damaging consequence of SGK1 on
Manon Engels
Manon Engels
Department of Paediatrics, Radboud Amalia Children’s Hospital, Radboud university medical center, Nijmegen, The Netherlands
Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Paul N Span
Paul N Span
Radiation Oncology, Radiotherapy and OncoImmunology Laboratory, RIMLS, Radboud university medical center, Nijmegen, The Netherlands
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Rod T Mitchell
Rod T Mitchell
MRC Centre for Reproductive Health, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Joop J T M Heuvel
Joop J T M Heuvel
Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Monica A Marijnissen-van Zanten
Monica A Marijnissen-van Zanten
Department of Pathology, Radboud university medical center, Nijmegen, The Netherlands
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Antonius E van Herwaarden
Antonius E van Herwaarden
Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Christina A Hulsbergen-van de Kaa
Christina A Hulsbergen-van de Kaa
Department of Pathology, Radboud university medical center, Nijmegen, The Netherlands
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Egbert Oosterwijk
Egbert Oosterwijk
Department of Urology, Radboud university medical center, Nijmegen, The Netherlands
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Nike M Stikkelbroeck
Nike M Stikkelbroeck
Department of Internal Medicine, Radboud university medical center, Nijmegen, The Netherlands
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Lee B Smith
Lee B Smith
MRC Centre for Reproductive Health, University of Edinburgh, The Queen’s Medical Research Institute, Edinburgh, UK
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Fred C G J Sweep
Fred C G J Sweep
Laboratory Medicine, Radboud Institute for Molecular Life Sciences (RIMLS), Radboud university medical center, Nijmegen, The Netherlands
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Hedi L Claahsen-van der Grinten
Hedi L Claahsen-van der Grinten
Department of Paediatrics, Radboud Amalia Children’s Hospital, Radboud university medical center, Nijmegen, The Netherlands
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coworkers ( 20 ) and Pihlajoki ( 21 ). Padua and coworkers ( 19 ) developed a mouse model lacking both GATA4 and GATA6 expressions in steroidogenic cells. These mice suffer from adrenal aplasia, and female mice die within days after birth. However, male mice
