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syndrome . PNAS 2007 104 12587 – 12594 . ( https://doi.org/10.1073/pnas.0705408104 ) 28 Petersen KF Shulman GI . Pathogenesis of skeletal muscle insulin resistance in type 2 diabetes mellitus . American Journal of Cardiology 2002 90 11G – 18G
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comorbidity such as history of diabetes mellitus, gestational diabetes, past or present history of thyroid disease, vascular disease, chronic hypertension, renal disease, polycystic ovary syndrome, use of corticosteroids, β-blockers, β-agonists and other drugs
Monash Centre for Health Research and Implementation, Monash University, Clayton, Victoria, Australia
Australian Institute for Musculoskeletal Science, Victoria University, Melbourne, Victoria, Australia
Medicine-Western Health, Faculty of Medicine, Dentistry and Health Science, Melbourne University, Melbourne, Victoria, Australia
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Australian Institute for Musculoskeletal Science, Victoria University, Melbourne, Victoria, Australia
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Division of Diabetes, Endocrinology & Gastroenterology, School of Medical Sciences, Faculty of Biology, Medicine and Health, The University of Manchester, Manchester, UK
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Diabetes and Endocrine Units, Monash Health, Clayton, Victoria, Australia
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Introduction Polycystic ovary syndrome (PCOS) affects 8–13% of women of reproductive age ( 1 ) has major metabolic (increased type 2 diabetes mellitus (T2D) and cardiovascular risk factors) ( 2 , 3 , 4 ), reproductive (leading cause of
Division of Biomedical Information Analysis, Iwate Tohoku Medical Megabank Organization, Disaster Reconstruction Center, Iwate Medical University, Yahaba, Japan
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Faculty of Health Science, Bukkyo University, Kyoto, Japan
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secretion . American Journal of Physiology 1982 243 E310 – E318 . ( https://doi.org/10.1152/ajpendo.1982.243.4.E310 ) 42 Hales CN Barker DJ . Type 2 (non-insulin-dependent) diabetes mellitus: the thrifty phenotype hypothesis . Diabetologia 1992 35
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Pediatric team of the Clinical Investigation Center 9302/INSERM, Hospital of Children, Toulouse, France
Institut Toulousain des Maladies Infectieuses et Inflammatoires (Infinity), INSERM UMR1291 - CNRS UMR5051 - Université Toulouse III, Toulouse, France
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Patient-Reported Outcomes Unit (PROQOL), UMR 1123, University Paris Cité, INSERM, Paris, France
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hypothalamic pituitary axis hormonal deficiencies and/or diabetes insipidus should be on an optimized and stable treatment regimen for at least 3 months prior to inclusion. Children with cancer history, psychosocial dwarfism, diabetes mellitus, human
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EMN Van Pareren YK Mulder PGH Hokken-Koelega AC. Risk factors for diabetes mellitus type 2 and metabolic syndrome are comparable for previously growth hormone (GH)-treated young adults born small for gestational age (SGA) and untreated
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National and Kapodistrian University of Athens Medical School, ‘Aghia Sophia’ Children’s Hospital, Athens, Greece
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, bronchopulmonary dysplasia, obstructive apnea, etc.) • Metabolic (poorly controlled diabetes mellitus) • Hematological (chronic severe anemia, hemochromatosis) • Oncological (leukemias, lymphomas, tumors of the CNS, etc.) • CNS (idiopathic cerebral palsy, mental
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multiple analyses the risk of pregnancy, preeclampsia and gestational diabetes mellitus in PCOS women is at least three times higher ( 11 ). The risk of early delivery in these individuals is at least double, and their infants have had twice the risk of
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, diabetes mellitus (DM), nonalcoholic fatty liver disease (NAFLD), cardiovascular disease, endometrial lesions, and hypertension during pregnancy ( 2 , 3 , 4 , 5 ). The etiology of PCOS is unclear at present, though environmental and genetic factors are
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gestational diabetes mellitus ( n = 1) and premature delivery at 36 weeks ( n = 1). The other 2 women experienced first-trimester miscarriages after IVF-ET. Figure 3 Fertility outcomes among 106 patients with IHH. Twenty-four women who were planning a