Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain
University Center of Defense of Madrid (CUD-ACD), Madrid, Spain
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Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain
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Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain
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Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain
University Center of Defense of Madrid (CUD-ACD), Madrid, Spain
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Networking Biomedical Research Center on Bioengineering, Biomaterials and Nanomedicine (CIBER-BBN), Madrid, Spain
Ramón y Cajal Institute of Sanitary Research (IRYCIS), Madrid, Spain
University Center of Defense of Madrid (CUD-ACD), Madrid, Spain
Immune System Diseases-Rheumatology and Oncology Service, University Hospital Príncipe de Asturias, Alcalá de Henares, Madrid, Spain
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.1155/2014/562625 ) 25587545 39 Eming SA Werner S Bugnon P Wickenhauser C Siewe L Utermöhlen O Davidson JM Krieg T Roers A. Accelerated wound closure in mice deficient for interleukin-10 . American Journal of Pathology 2007 170 188 – 202 . ( https://doi.org/10
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.5 * Values are presented as means ± s.d. * P < 0.05, ** P < 0.01; water vs land trials. CVRR, coefficient of variation of R–R intervals; HR, heart rate; IL-6, interleukin-6; RER, respiratory exchange ratio; VO 2 , oxygen uptake
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, and TNF-α and interleukin-6 (IL-6) concentrations were measured using an ELISA kit (R&D Systems) according to the manufacturer’s instructions. For the in vivo experiment, serum TSH, FT3, and FT4 levels of mice were determined using an ELISA kit
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were related to the JAK-STAT signalling pathway. IL6ST is a member of the interleukin-6 cytokine family, which is capable of forming a complex with IL6 receptor (IL6R) and assists in IL6R activation, thereby initiating signal transduction cascades in
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interleukin (IL)-1α and IL-1β show concentration-dependent negative regulation of NIS ( 16 , 17 ). IL1 is secreted mainly by neutrophils ( 15 ). Previous studies have found that peripheral blood IL-1β decreases in patients with hyperthyroidism. We speculated
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Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar São João, Porto, Portugal
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Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar São João, Porto, Portugal
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der Meer MJ Ross HA Smals AG Benraad TJ Kloppenborg PW . Continuous infusion of interleukin-1 beta induces a nonthyroidal illness syndrome in the rat . Endocrinology 1992 131 2139 – 2146 . ( https://doi.org/10.1210/endo.131.5.1425414 ) 10
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reactive oxygen species and myeloperoxidase levels was reported, particularly in patients with insulin resistance. Moreover, it was shown that inflammation in PCOS induces leukocyte–endothelium interactions and a simultaneous increase in interleukin-6
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over-treatment. Inflammatory cytokines may also have a role in this phenomenon. Interleukin (IL)-1β and IL-6 are both pro-inflammatory cytokines whose levels have been shown to increase within hours of infection in crticially ill patients and are
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Hayakawa Y. Hypothalamic interleukin-1 beta and tumor necrosis factor-alpha, but not interleukin-6, mediate the endotoxin-induced suppression of the reproductive axis in rats . Endocrinology 2003 144 4868 – 4875 . ( https://doi.org/10.1210/en.2003
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Synlab Academy, Synlab Holding Germany GmbH, Heidelberg, Germany
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levels of interleukin-6 (IL-6). Data analyses Continuous data following a normal distribution are reported as means with standard deviations ( s.d. ), while variables with a skewed distribution are shown as medians with interquartile ranges and