Search for other papers by Louise Færch in
Google Scholar
PubMed
Department of Cardiology, Department of Growth and Reproduction, Faculty of Health Sciences, Nephrology and Endocrinology H, Hillerød University Hospital, Dyrehavevej 29, DK-3400 Hillerød, Denmark
Search for other papers by Anders Juul in
Google Scholar
PubMed
Search for other papers by Ulrik Pedersen-Bjergaard in
Google Scholar
PubMed
Department of Cardiology, Department of Growth and Reproduction, Faculty of Health Sciences, Nephrology and Endocrinology H, Hillerød University Hospital, Dyrehavevej 29, DK-3400 Hillerød, Denmark
Search for other papers by Birger Thorsteinsson in
Google Scholar
PubMed
muscle and bone turnover as well as lipid metabolism and growth (23, 24, 25) , and our study supports that a low IGF1 level in patients with type 1 diabetes may increase by lowering HbA1c and thereby diminish the negative influence of a low IGF1 on the
Search for other papers by Stavroula A Paschou in
Google Scholar
PubMed
Search for other papers by Eleni Palioura in
Google Scholar
PubMed
Search for other papers by Dimitrios Ioannidis in
Google Scholar
PubMed
Search for other papers by Panagiotis Anagnostis in
Google Scholar
PubMed
Search for other papers by Argyro Panagiotakou in
Google Scholar
PubMed
Search for other papers by Vasiliki Loi in
Google Scholar
PubMed
Search for other papers by Georgios Karageorgos in
Google Scholar
PubMed
Search for other papers by Dimitrios G Goulis in
Google Scholar
PubMed
Search for other papers by Andromachi Vryonidou in
Google Scholar
PubMed
, 13 ). The underlying pathogenic mechanisms of these findings remain unclear, but may reflect a direct effect of DHEA-S on insulin ( 24 ) and lipid metabolism ( 25 ). Dehydroepiandrosterone (DHEA) concentrations have been positively correlated to
Search for other papers by Xiao-Shan Huang in
Google Scholar
PubMed
Search for other papers by Ning Dai in
Google Scholar
PubMed
Search for other papers by Jian-Xia Xu in
Google Scholar
PubMed
Search for other papers by Jun-Yi Xiang in
Google Scholar
PubMed
Search for other papers by Xiao-Zhong Zheng in
Google Scholar
PubMed
Search for other papers by Tian-Yu Ke in
Google Scholar
PubMed
Search for other papers by Lin-Ying Ma in
Google Scholar
PubMed
Search for other papers by Qi-Hao Shi in
Google Scholar
PubMed
Search for other papers by Shu-Feng Fan in
Google Scholar
PubMed
regulator of glucose and lipid metabolism . Annual Review of Nutrition 2007 27 179 – 192 . ( https://doi.org/10.1146/annurev.nutr.27.061406.093618 ) 24 Uyeda K & Repa JJ . Carbohydrate response element binding protein, ChREBP, a transcription
Department of Nuclear Medicine, CHU de Bordeaux, Pessac, France
INRA, Nutrition et Neurobiologie Intégrée, UMR1286, Bordeaux, France
Search for other papers by J Brossaud in
Google Scholar
PubMed
INRA, Nutrition et Neurobiologie Intégrée, UMR1286, Bordeaux, France
Search for other papers by V Pallet in
Google Scholar
PubMed
Department of Nuclear Medicine, CHU de Bordeaux, Pessac, France
INRA, Nutrition et Neurobiologie Intégrée, UMR1286, Bordeaux, France
Search for other papers by J-B Corcuff in
Google Scholar
PubMed
may play a role in pancreatic function, but confirmation of these studies has to be obtained. Finally, we will not cover the very interesting topic of glucose and lipid metabolism here because many interesting studies and reviews detailed the
Search for other papers by Eric M Ndombi in
Google Scholar
PubMed
Search for other papers by Valentine Budambula in
Google Scholar
PubMed
Search for other papers by Mark K Webale in
Google Scholar
PubMed
Search for other papers by Francis O Musumba in
Google Scholar
PubMed
Search for other papers by Jesca O Wesongah in
Google Scholar
PubMed
Search for other papers by Erick Mibei in
Google Scholar
PubMed
Search for other papers by Aabid A Ahmed in
Google Scholar
PubMed
Search for other papers by Raphael Lihana in
Google Scholar
PubMed
Search for other papers by Tom Were in
Google Scholar
PubMed
visceral adiposity in HIV and HCV co-infected treatment-naive and HCV mono-infected IDUs indicates alterations in lipid metabolism. This premise is supported by previous studies in HIV and HCV co-infected patients showing increased risk of having
Search for other papers by Line K Johnson in
Google Scholar
PubMed
Morbid Obesity Centre, Department of Nutrition, Norwegian National Advisory Unit on Familial Hypercholesterolemia, Department of Gynecology, Institute of Clinical Medicine, Department of Endocrinology, Vestfold Hospital Trust, PO Box 2168, 3103 Tønsberg, Norway
Search for other papers by Kirsten B Holven in
Google Scholar
PubMed
Search for other papers by Njord Nordstrand in
Google Scholar
PubMed
Search for other papers by Jan R Mellembakken in
Google Scholar
PubMed
Morbid Obesity Centre, Department of Nutrition, Norwegian National Advisory Unit on Familial Hypercholesterolemia, Department of Gynecology, Institute of Clinical Medicine, Department of Endocrinology, Vestfold Hospital Trust, PO Box 2168, 3103 Tønsberg, Norway
Search for other papers by Tom Tanbo in
Google Scholar
PubMed
Morbid Obesity Centre, Department of Nutrition, Norwegian National Advisory Unit on Familial Hypercholesterolemia, Department of Gynecology, Institute of Clinical Medicine, Department of Endocrinology, Vestfold Hospital Trust, PO Box 2168, 3103 Tønsberg, Norway
Search for other papers by Jøran Hjelmesæth in
Google Scholar
PubMed
Glucose (mmol/l) 5.3 (1.0) 5.3 (1.0) 0.991 HbA1c (%) 5.6 (0.4) 5.6 (0.6) 0.758 HOMA-IR 5.5 (4.6) 5.7 (3.2) 0.852 Insulin (pmol/l) a 129 (76) 142 (70) 0.537 C-peptide (nmol/l) a 1.3 (0.5) 1.4 (0.5) 0.478 Lipid metabolism and blood pressure Total
Search for other papers by Maria Angela D'amico in
Google Scholar
PubMed
Search for other papers by Barbara Ghinassi in
Google Scholar
PubMed
Search for other papers by Pascal Izzicupo in
Google Scholar
PubMed
Search for other papers by Lamberto Manzoli in
Google Scholar
PubMed
Search for other papers by A Di Baldassarre in
Google Scholar
PubMed
peptide's potency to inhibit cellular glucose uptake. These observations suggest that hereditary alterations in pancreastatin's primary structure may give rise to interindividual differences in glucose and lipid metabolism. Pancreastatin also inhibits
Search for other papers by Tatsuya Kondo in
Google Scholar
PubMed
Search for other papers by Nobukazu Miyakawa in
Google Scholar
PubMed
Search for other papers by Sayaka Kitano in
Google Scholar
PubMed
Search for other papers by Takuro Watanabe in
Google Scholar
PubMed
Search for other papers by Rieko Goto in
Google Scholar
PubMed
Search for other papers by Mary Ann Suico in
Google Scholar
PubMed
Search for other papers by Miki Sato in
Google Scholar
PubMed
Search for other papers by Yuki Takaki in
Google Scholar
PubMed
Search for other papers by Masaji Sakaguchi in
Google Scholar
PubMed
Search for other papers by Motoyuki Igata in
Google Scholar
PubMed
Search for other papers by Junji Kawashima in
Google Scholar
PubMed
Search for other papers by Hiroyuki Motoshima in
Google Scholar
PubMed
Search for other papers by Takeshi Matsumura in
Google Scholar
PubMed
Search for other papers by Hirofumi Kai in
Google Scholar
PubMed
Search for other papers by Eiichi Araki in
Google Scholar
PubMed
. ( https://doi.org/10.1194/jlr.M043299 ) 5 Zhou H Liu R ER stress and hepatic lipid metabolism . Frontiers in Genetics 2014 5 112. ( https://doi.org/10.3389/fgene.2014.00112 ) 6 Adachi H Kondo T Ogawa R Sasaki K Morino-Koga S
Search for other papers by Jana Ernst in
Google Scholar
PubMed
Search for other papers by Katharina Gert in
Google Scholar
PubMed
Search for other papers by Frank Bernhard Kraus in
Google Scholar
PubMed
Search for other papers by Ulrike Elisabeth Rolle-Kampczyk in
Google Scholar
PubMed
Search for other papers by Martin Wabitsch in
Google Scholar
PubMed
Search for other papers by Faramarz Dehghani in
Google Scholar
PubMed
Search for other papers by Kristina Schaedlich in
Google Scholar
PubMed
expressed adipocyte-specific protein on the surface of lipid vesicles associated with the stored triglyceride content. Thereby, it contributes to homeostasis in lipid metabolism by blocking basal and mediating hormone-stimulated lipolysis ( 49 , 50 , 51
Search for other papers by Lena-Maria Levin in
Google Scholar
PubMed
DZHK (German Centre for Cardiovascular Research), Greifswald, Germany
DZD (German Center for Diabetes Research), Greifswald, Germany
Search for other papers by Henry Völzke in
Google Scholar
PubMed
Search for other papers by Markus M Lerch in
Google Scholar
PubMed
Institute and Policlinic for Radiology and Interventional Radiology, University Hospital, Carl-Gustav-Carus University Dresden, Dresden, Germany
Search for other papers by Jens-Peter Kühn in
Google Scholar
PubMed
DZHK (German Centre for Cardiovascular Research), Greifswald, Germany
Search for other papers by Matthias Nauck in
Google Scholar
PubMed
DZHK (German Centre for Cardiovascular Research), Greifswald, Germany
Search for other papers by Nele Friedrich in
Google Scholar
PubMed
DZHK (German Centre for Cardiovascular Research), Greifswald, Germany
Search for other papers by Stephanie Zylla in
Google Scholar
PubMed
on the role of adipose tissue as an endocrine organ that produces a multitude of metabolically active hormones, so-called adipokines, which are known to be involved in the regulation of glucose and lipid metabolism, food intake and inflammation ( 4