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HUH Diagnostic Center and Helsinki University Hospital, Helsinki, Finland
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HUH Diagnostic Center and Helsinki University Hospital, Helsinki, Finland
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HUH Diagnostic Center and Helsinki University Hospital, Helsinki, Finland
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(A and B) Native GNEN-1 cells on the growth surface. The cells grow in clusters adherent to each other and the growth surface. The cells have dendritic-like sprouts from the soma. Clear nucleoli are seen. (C) GNEN-1 cells stained immunohistochemically
Berlin Institute of Health (BIH), Berlin, Germany
Department of Nephrology, School of Medicine, Heinrich-Heine-Universität Düsseldorf, Düsseldorf, Germany
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Core Unit Bioinformatics, Berlin Institute of Health, Berlin, Germany
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Core Unit Bioinformatics, Berlin Institute of Health, Berlin, Germany
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Max Delbrück Center for Molecular Medicine in the Helmholtz Association, Berlin, Germany
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Berlin Institute of Health (BIH), Berlin, Germany
Department of Nephrology, School of Medicine, Heinrich-Heine-Universität Düsseldorf, Düsseldorf, Germany
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doublings; cPD) is indicated. During long-term treatment, cells initially died, but the population recovered after approximately 30 days. (B) IC 50 of mitotane in 12 clones derived from long-term treated HAC-15 cells vs 6 control clones measured by MTT
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triglycerides were significantly increased compared to the control group in knockdown liver cells. In addition, it has been found that the activation of farnesoid X receptor leads to upregulation of gene expression encoding fetuin-B ( 8 ). Zhu and other scholars
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Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China
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-infiltrating immune cells (TIICs) was analyzed by the Tumor Immune Estimation Resource (TIMER). TIMER is a widely used tool for analyzing gene expression and infiltration of six immune cell types (B cells, CD4+ T cells, CD8+ T cells, neutrophils, macrophages, and
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partly inhibited migration induced by OA in MDA-MB-231 cells. Figure 1 FFAR1 and FFAR4 mediate migration induced by OA in breast cancer cells. Panel (A), (B) and (D) Cultures of MDA-MB-231 and MCF-7 cells were treated with DC260126 or AH7614
Faculty of Medicine, University of Freiburg, Freiburg, Germany
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Faculty of Medicine, University of Freiburg, Freiburg, Germany
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Faculty of Medicine, University of Freiburg, Freiburg, Germany
Institute of Veterinary Medicine, Georg-August-University Goettingen, Goettingen, Germany
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Box plot diagram of mRNA expression levels of DDX1 (A), DDX5 (B), DDX17 (C) and ERα (D) in response to endoxifen and fulvestrant. mRNA expression levels of DDX1, DDX5, DDX17 and ESR- α in breast cancer cell lines are displayed under anti
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survival factor for SH-SY5Y cells, we assayed the effect of IGF-1 on cell death induced by MPP + insult. As shown in Fig. 1B , MPP + -induced cell death was partially but significantly attenuated by pretreatment of cells with 10 nM IGF-1. The LDH activity
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Guangdong Geriatric Institute, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
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Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Reproductive Medicine Center, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Sciences, Guangzhou, China
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Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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Division of Endocrinology, Department of Internal Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China
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Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai National Center for Translational Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
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+ T cells (Supplementary Table 4). The results of hierarchical cluster analyses showed distinguishable lncRNA and mRNA expression profiles between the healthy control CD4+ T cells and GD CD4+ T cells ( Fig. 1A and B ). To visualize the
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observed in the absence of the primary antibody, and chromogranin A-positive cells in the normal small intestine stained positive for PTPRM ( Fig. 2A and B ). These cells likely represent the enterochromaffin cell of origin of SI-NETs. Figure 1
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Department of General Surgery, Xiangya Hospital, Central South University, Changsha, China
Department of Burns and Plastic Surgery, The Third Xiangya Hospital, Central South University, Changsha, China
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G2 43 G3 12 Resection R0 67 R1 16 R2 6 a Overlapping samples; b FFPE, formalin-fixed paraffin-embedded tissue. pNEN cell lines BON1 and QGP1 The human metastasized adherent